Robert N. Correll

4.0k total citations · 2 hit papers
36 papers, 3.1k citations indexed

About

Robert N. Correll is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert N. Correll has authored 36 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 23 papers in Cardiology and Cardiovascular Medicine and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in Robert N. Correll's work include Ion channel regulation and function (17 papers), Cardiac electrophysiology and arrhythmias (12 papers) and Neuroscience and Neuropharmacology Research (6 papers). Robert N. Correll is often cited by papers focused on Ion channel regulation and function (17 papers), Cardiac electrophysiology and arrhythmias (12 papers) and Neuroscience and Neuropharmacology Research (6 papers). Robert N. Correll collaborates with scholars based in United States, Germany and Ireland. Robert N. Correll's co-authors include Jeffery D. Molkentin, Hadi Khalil, Onur Kanisicak, Jason Karch, Ronald J. Vagnozzi, Matthew J. Brody, Allen J. York, Vikram Prasad, Michelle A. Sargent and Tobias G. Schips and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Robert N. Correll

33 papers receiving 3.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Fibroblast-specific TGF-β–Smad2/3 signaling underlies cardiac fibrosis

2017 673 citations Hadi Khalil, Onur Kanisicak et al. Journal of Clinical Investigation profile →
Genetic lineage tracing defines myofibroblast origin and function in the injured heart

2016 640 citations Onur Kanisicak, Hadi Khalil et al. Nature Communications profile →

Peers

Countries citing papers authored by Robert N. Correll

Since Specialization

Citations

This map shows the geographic impact of Robert N. Correll's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert N. Correll with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert N. Correll more than expected).

Fields of papers citing papers by Robert N. Correll

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert N. Correll. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert N. Correll. The network helps show where Robert N. Correll may publish in the future.

Co-authorship network of co-authors of Robert N. Correll

This figure shows the co-authorship network connecting the top 25 collaborators of Robert N. Correll. A scholar is included among the top collaborators of Robert N. Correll based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert N. Correll. Robert N. Correll is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Regulation of cardiac fibroblast cell death by unfolded protein response signaling 2024 ·Frontiers in Physiology ·(unknown), (unknown), Robert N. Correll	3
2	Assessing wound closure in mice using skin-punch biopsy 2023 ·STAR Protocols ·(unknown), (unknown), Chuong Bui, Robert N. Correll	7
3	Disruption of valosin-containing protein activity causes cardiomyopathy and reveals pleiotropic functions in cardiac homeostasis 2019 ·Journal of Biological Chemistry ·Matthew J. Brody, Davy Vanhoutte, (unknown), Ruijie Liu, Robert N. Correll, Michelle A. Sargent, Jeffery D. Molkentin	23
4	Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability 2019 ·Nature Communications ·Tobias G. Schips, Davy Vanhoutte, (unknown), Robert N. Correll, Matthew J. Brody, Hadi Khalil, Jason Karch, (unknown), Michelle A. Sargent, Marjorie Maillet, Robert S. Ross, Jeffery D. Molkentin	42
5	Fibroblast-specific TGF-β–Smad2/3 signaling underlies cardiac fibrosis breakdown → 2017 ·Journal of Clinical Investigation ·Hadi Khalil, Onur Kanisicak, Vikram Prasad, Robert N. Correll, Xing Fu, Tobias G. Schips, Ronald J. Vagnozzi, Ruijie Liu, Thanh V. Huynh, Se‐Jin Lee, Jason Karch, Jeffery D. Molkentin	673
6	Genetic lineage tracing defines myofibroblast origin and function in the injured heart breakdown → 2016 ·Nature Communications ·Onur Kanisicak, Hadi Khalil, Malina J. Ivey, Jason Karch, Bryan D. Maliken, Robert N. Correll, Matthew J. Brody, Suh‐Chin J. Lin, Bruce J. Aronow, Michelle D. Tallquist, Jeffery D. Molkentin	640
7	A Tension-Based Model Distinguishes Hypertrophic versus Dilated Cardiomyopathy 2016 ·Cell ·Jennifer Davis, (unknown), Robert N. Correll, Catherine A. Makarewich, Jennifer A. Schwanekamp, Farid Moussavi‐Harami, Dan Wang, Allen J. York, Haodi Wu, Steven R. Houser, Christine E. Seidman, Jonathan G. Seidman, Michael Regnier, Joseph M. Metzger, Joseph C. Wu, Jeffery D. Molkentin	165
8	The Mitochondrial Calcium Uniporter Selectively Matches Metabolic Output to Acute Contractile Stress in the Heart 2015 ·Cell Reports ·Jennifer Q. Kwong, Xiyuan Lu, Robert N. Correll, Jennifer A. Schwanekamp, Ronald J. Vagnozzi, Michelle A. Sargent, Allen J. York, Jianyi Zhang, Donald M. Bers, Jeffery D. Molkentin	276
9	STIM1 elevation in the heart results in aberrant Ca2+ handling and cardiomyopathy 2015 ·Journal of Molecular and Cellular Cardiology ·Robert N. Correll, Sanjeewa A. Goonasekera, Jop H. van Berlo, (unknown), Federica Accornero, (unknown), Catherine A. Makarewich, Allen J. York, Michelle A. Sargent, Xiongwen Chen, Steven R. Houser, Jeffery D. Molkentin	89
10	Transient Receptor Potential Channels Contribute to Pathological Structural and Functional Remodeling After Myocardial Infarction 2014 ·Circulation Research ·Catherine A. Makarewich, Hongyu Zhang, Jennifer Davis, Robert N. Correll, Danielle M. Trappanese, Nicholas E. Hoffman, Constantine D. Troupes, Remus M. Berretta, Hajime Kubo, Muniswamy Madesh, Xiongwen Chen, Erhe Gao, Jeffery D. Molkentin, Steven R. Houser	97
11	A Caveolae-Targeted L-Type Ca ²⁺ Channel Antagonist Inhibits Hypertrophic Signaling Without Reducing Cardiac Contractility 2012 ·Circulation Research ·Catherine A. Makarewich, Robert N. Correll, Hui Gao, Hongyu Zhang, Baohua Yang, Remus M. Berretta, Victor Rizzo, Jeffery D. Molkentin, Steven R. Houser	86
12	Tropomyosin Dephosphorylation Results in Compensated Cardiac Hypertrophy 2012 ·Journal of Biological Chemistry ·Emily Schulz, Robert N. Correll, (unknown), Marco Stephan Lofrano‐Alves, Patti Engel, Gilbert Newman, Jo El J. Schultz, Jeffery D. Molkentin, Beata M. Wolska, R. John Solaro, David F. Wieczorek	19
13	Decreased cardiac L-type Ca2+ channel activity induces hypertrophy and heart failure in mice 2011 ·Journal of Clinical Investigation ·Sanjeewa A. Goonasekera, Karin Hammer, Mannix Auger‐Messier, Ilona Bódi, Xiongwen Chen, Hongyu Zhang, Steven Reiken, John W. Elrod, Robert N. Correll, Allen J. York, Michelle A. Sargent, Franz Hofmann, Sven Moosmang, Andrew R. Marks, Steven R. Houser, Donald M. Bers, Jeffery D. Molkentin	132
14	CIB1 is a regulator of pathological cardiac hypertrophy 2010 ·Nature Medicine ·Joerg Heineke, Mannix Auger‐Messier, Robert N. Correll, Jian Xu, (unknown), Weiping Yuan, Helmut Drexler, Leslie V. Parise, Jeffery D. Molkentin	77
15	Rem GTPase interacts with the proximal Ca_V1.2 C-terminus and modulates calcium-dependent channel inactivation 2010 ·Channels ·(unknown), Shawn M. Crump, (unknown), Robert N. Correll, Brian S. Finlin, Jonathan Satin, Douglas Andres	21
16	α1G-dependent T-type Ca2+ current antagonizes cardiac hypertrophy through a NOS3-dependent mechanism in mice 2009 ·Journal of Clinical Investigation ·Hiroyuki Nakayama, Ilona Bódi, Robert N. Correll, Xiongwen Chen, John N. Lorenz, Steven R. Houser, Jeffrey Robbins, Arnold Schwartz, Jeffery D. Molkentin	69
17	Calmodulin binding is dispensable for Rem-mediated Ca2+ channel inhibition 2007 ·Molecular and Cellular Biochemistry ·Robert N. Correll, (unknown), Dana M. Niedowicz, Jonathan Satin, Douglas Andres	4
18	Plasma Membrane Targeting Is Essential for Rem-mediated Ca2+ Channel Inhibition 2007 ·Journal of Biological Chemistry ·Robert N. Correll, (unknown), Brian S. Finlin, (unknown), Jonathan Satin, Douglas Andres	36
19	Analyses of Rem/RGK Signaling and Biological Activity 2006 ·Methods in enzymology on CD-ROM/Methods in enzymology ·Douglas Andres, Shawn M. Crump, Robert N. Correll, Jonathan Satin, Brian S. Finlin	6
20	Regulation of L-type Ca2+ Channel Activity and Insulin Secretion by the Rem2 GTPase 2005 ·Journal of Biological Chemistry ·Brian S. Finlin, Amber L. Mosley, Shawn M. Crump, Robert N. Correll, Sabire Özcan, Jonathan Satin, Douglas Andres	75

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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