Maja T. Lindenmeyer
- Molecular Biology top 5%
- Nephrology top 0.2%
- Immunology top 5%
- Epidemiology top 5%
- Surgery top 10%
- Co-authors
- Clemens D. CohenMatthias KretzlerStephan SegererMatthias A. NeusserIrmgard MerfortMaria Pia RastaldiDetlef SchlöndorffAlfonso Garcı́a-Piñeres
- Topics
- Renal Diseases and Glomerulopathies (36 papers)Chronic Kidney Disease and Diabetes (20 papers)Renal and related cancers (13 papers)
- Cited by
- NephrologyImmunologyTransplantation
- Partner nations
- GermanySwitzerlandUnited States
In The Last Decade
Maja T. Lindenmeyer
96 papers receiving 4.3k citations
Hit Papers
Peers
Comparison fields: 5 of 126
- Molecular Biology 1.6k
- Nephrology 1.6k
- Immunology 812
- Epidemiology 578
- Surgery 482
Countries citing papers authored by Maja T. Lindenmeyer
This map shows the geographic impact of Maja T. Lindenmeyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maja T. Lindenmeyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maja T. Lindenmeyer more than expected).
Fields of papers citing papers by Maja T. Lindenmeyer
This network shows the impact of papers produced by Maja T. Lindenmeyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maja T. Lindenmeyer. The network helps show where Maja T. Lindenmeyer may publish in the future.
Co-authorship network of co-authors of Maja T. Lindenmeyer
This figure shows the co-authorship network connecting the top 25 collaborators of Maja T. Lindenmeyer. A scholar is included among the top collaborators of Maja T. Lindenmeyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maja T. Lindenmeyer. Maja T. Lindenmeyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 4 | |
| 3 | 9 | |
| 4 | 2 | |
| 5 | 1 | |
| 6 | 4 | |
| 7 | 7 | |
| 8 | 44 | |
| 9 | 27 | |
| 10 | 14 | |
| 11 | 29 | |
| 12 | 11 | |
| 13 | 7 | |
| 14 | 26 | |
| 15 | 26 | |
| 16 | 175 | |
| 17 | Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging micebreakdown → | 586 |
| 18 | 31 | |
| 19 | 17 | |
| 20 | 20 |
About Maja T. Lindenmeyer
Maja T. Lindenmeyer is a scholar working on Nephrology, Transplantation and Immunology, having authored 96 papers that have together received 4.3k indexed citations. Recurring topics across this work include Renal Diseases and Glomerulopathies (36 papers), Chronic Kidney Disease and Diabetes (20 papers) and Renal and related cancers (13 papers). The work is most often cited by research in Nephrology (1.6k citations), Immunology (812 citations) and Transplantation (103 citations). Maja T. Lindenmeyer has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Clemens D. Cohen, Matthias Kretzler, Stephan Segerer, Matthias A. Neusser, Irmgard Merfort, Maria Pia Rastaldi, Detlef Schlöndorff, Alfonso Garcı́a-Piñeres, Tobias B. Huber and Felix Eichinger. Their work appears in journals such as Nucleic Acids Research, Journal of Clinical Investigation and The Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.