Taiji Matsusaka

9.5k total citations · 3 hit papers
124 papers, 7.4k citations indexed

About

Taiji Matsusaka is a scholar working on Nephrology, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Taiji Matsusaka has authored 124 papers receiving a total of 7.4k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Nephrology, 56 papers in Molecular Biology and 32 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Taiji Matsusaka's work include Renal Diseases and Glomerulopathies (55 papers), Renin-Angiotensin System Studies (30 papers) and Chronic Kidney Disease and Diabetes (28 papers). Taiji Matsusaka is often cited by papers focused on Renal Diseases and Glomerulopathies (55 papers), Renin-Angiotensin System Studies (30 papers) and Chronic Kidney Disease and Diabetes (28 papers). Taiji Matsusaka collaborates with scholars based in Japan, United States and China. Taiji Matsusaka's co-authors include Iekuni Ichikawa, Fumio Niimura, Shizuo Akira, Yukihiro Nishio, Agnes B. Fogo, Ira Pastan, T Kishimoto, Kazuo Fujikawa, Naofumi Mukaida and Kouji Matsushima and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

Taiji Matsusaka

116 papers receiving 7.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8.

1993 875 citations Taiji Matsusaka et al. profile →
Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway

1994 868 citations Taiji Matsusaka et al. profile →
Autophagy Protects the Proximal Tubule from Degeneration and Acute Ischemic Injury

2011 380 citations Tomonori Kimura, Yoshitsugu Takabatake et al. Journal of the American Society of Nephrology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Taiji Matsusaka Japan	43	2.9k	1.9k	1.3k	1.3k	1.1k	124	7.4k
Yoshio Terada Japan	46	3.3k 1.1×	1.2k 0.6×	915 0.7×	568 0.4×	503 0.4×	253	6.6k
Rolf A.K. Stahl Germany	59	2.9k 1.0×	4.8k 2.6×	1.7k 1.3×	3.4k 2.7×	1.3k 1.1×	257	12.2k
János G. Filep Canada	59	2.9k 1.0×	729 0.4×	1.4k 1.1×	2.8k 2.2×	435 0.4×	196	9.0k
Kenichi Shikata Japan	41	1.8k 0.6×	1.6k 0.9×	775 0.6×	811 0.6×	345 0.3×	177	6.1k
Elsa Sánchez‐López Spain	38	4.1k 1.4×	653 0.4×	728 0.6×	2.2k 1.7×	979 0.9×	53	7.7k
Carla Zoja Italy	69	3.4k 1.2×	4.8k 2.6×	2.4k 1.8×	2.4k 1.9×	584 0.5×	192	13.3k
Emile de Heer Netherlands	47	2.9k 1.0×	2.6k 1.4×	451 0.3×	1.3k 1.0×	432 0.4×	204	8.5k
Katherine Gordon United States	41	2.7k 0.9×	3.7k 2.0×	774 0.6×	698 0.5×	350 0.3×	57	7.8k
Pravin C. Singhal United States	45	2.2k 0.8×	2.1k 1.1×	600 0.5×	857 0.7×	251 0.2×	301	7.4k
J Brett United States	35	3.2k 1.1×	764 0.4×	817 0.6×	2.0k 1.5×	407 0.4×	45	11.8k

Countries citing papers authored by Taiji Matsusaka

Since Specialization

Citations

This map shows the geographic impact of Taiji Matsusaka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Taiji Matsusaka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Taiji Matsusaka more than expected).

Fields of papers citing papers by Taiji Matsusaka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Taiji Matsusaka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Taiji Matsusaka. The network helps show where Taiji Matsusaka may publish in the future.

Co-authorship network of co-authors of Taiji Matsusaka

This figure shows the co-authorship network connecting the top 25 collaborators of Taiji Matsusaka. A scholar is included among the top collaborators of Taiji Matsusaka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Taiji Matsusaka. Taiji Matsusaka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kawanishi, Kunio, Jun‐Dal Kim, Masahiro Koizumi, et al.. (2025). A novel glomerulopathy model demonstrates renal counterbalance via local angiotensin II regulation. Proceedings of the Japan Academy Series B. 101(7). 431–444. 1 indexed citations

Azushima, Kengo, Kento Kitada, Kazuya Nakagawa, et al.. (2025). Keratinocyte-specific angiotensin II receptor-associated protein deficiency exacerbates angiotensin II-dependent hypertension via activation of the skin renin-angiotensin system. Nature Communications. 16(1). 4789–4789.

Kaseda, Ryohei, Yusuke Nakagawa, Hirofumi Watanabe, et al.. (2024). Nephrotic syndrome induces the upregulation of cell proliferation-related genes in tubular cells in mice. Clinical and Experimental Nephrology. 29(4). 393–404.

Zhong, Jianyong, Haichun Yang, Elaine L. Shelton, et al.. (2022). Dicarbonyl-modified lipoproteins contribute to proteinuric kidney injury. JCI Insight. 7(21). 7 indexed citations

Miyazaki, Yoichi, Taiji Matsusaka, Naoki Sugano, et al.. (2017). Forced expression of vascular endothelial growth factor-A in podocytes decreases mesangial cell numbers and attenuates endothelial cell differentiation in the mouse glomerulus. Clinical and Experimental Nephrology. 22(2). 266–274. 11 indexed citations

Lim, Beom Jin, Jae Won Yang, Jun Zou, et al.. (2017). Tubulointerstitial fibrosis can sensitize the kidney to subsequent glomerular injury. Kidney International. 92(6). 1395–1403. 42 indexed citations

Motojima, Masaru, et al.. (2016). Characterization of Kidney and Skeleton Phenotypes of Mice Double Heterozygous for Foxc1 and Foxc2. Cells Tissues Organs. 201(5). 380–389. 13 indexed citations

Matsusaka, Taiji, Masato Ohtsuka, Hiromi Miura, et al.. (2016). Establishment of Nephrin Reporter Mice and Use for Chemical Screening. PLoS ONE. 11(6). e0157497–e0157497. 6 indexed citations

Namba‐Hamano, Tomoko, Yoshitsugu Takabatake, Tomonori Kimura, et al.. (2014). Autophagic Clearance of Mitochondria in the Kidney Copes with Metabolic Acidosis. Journal of the American Society of Nephrology. 25(10). 2254–2266. 49 indexed citations

10.

Ueno, Toshiharu, Namiko Kobayashi, Makiko Nakayama, et al.. (2013). Aberrant Notch1-dependent effects on glomerular parietal epithelial cells promotes collapsing focal segmental glomerulosclerosis with progressive podocyte loss. Kidney International. 83(6). 1065–1075. 48 indexed citations

11.

Matsusaka, Taiji, Fumio Niimura, Akihiro Shimizu, et al.. (2012). Liver Angiotensinogen Is the Primary Source of Renal Angiotensin II. Journal of the American Society of Nephrology. 23(7). 1181–1189. 210 indexed citations

12.

Shimizu, Akihiro, Jianyong Zhong, Yoichi Miyazaki, et al.. (2012). ARB protects podocytes from HIV-1 nephropathy independently of podocyte AT1. Nephrology Dialysis Transplantation. 27(8). 3169–3175. 10 indexed citations

13.

Matsusaka, Taiji, Eric P. Sandgren, Ayumi Shintani, et al.. (2011). Podocyte Injury Damages Other Podocytes. Journal of the American Society of Nephrology. 22(7). 1275–1285. 88 indexed citations

14.

Shao, Jing, Masaomi Nangaku, Reiko Inagi, et al.. (2007). Receptor-independent intracellular radical scavenging activity of an angiotensin II receptor blocker. Journal of Hypertension. 25(8). 1643–1649. 32 indexed citations

15.

Zuo, Yiqin, Taiji Matsusaka, Jianyong Zhong, et al.. (2006). HIV-1 Genes vpr and nef Synergistically Damage Podocytes, Leading to Glomerulosclerosis. Journal of the American Society of Nephrology. 17(10). 2832–2843. 83 indexed citations

16.

Ichikawa, Iekuni, Ji Ma, Masaru Motojima, & Taiji Matsusaka. (2005). Podocyte damage damages podocytes: autonomous vicious cycle that drives local spread of glomerular sclerosis. Current Opinion in Nephrology & Hypertension. 14(3). 205–210. 65 indexed citations

17.

Matsusaka, Taiji, Xin Jing, Kazuto Kobayashi, et al.. (2005). Genetic Engineering of Glomerular Sclerosis in the Mouse via Control of Onset and Severity of Podocyte-Specific Injury. Journal of the American Society of Nephrology. 16(4). 1013–1023. 199 indexed citations

18.

Ma, Ji, Taiji Matsusaka, Hiroshi Kawachi, et al.. (2004). Local Actions of Endogenous Angiotensin II in Injured Glomeruli. Journal of the American Society of Nephrology. 15(5). 1268–1276. 13 indexed citations

19.

Nishida, Masashi, Hidehiko Fujinaka, Taiji Matsusaka, et al.. (2002). Absence of angiotensin II type 1 receptor in bone marrow–derived cells is detrimental in the evolution of renal fibrosis. Journal of Clinical Investigation. 110(12). 1859–1868. 91 indexed citations

20.

Tsuchida, S., Taiji Matsusaka, Soichiro Okubo, et al.. (1998). Murine double nullizygotes of the angiotensin type 1A and 1B receptor genes duplicate severe abnormal phenotypes of angiotensinogen nullizygotes.. Journal of Clinical Investigation. 101(4). 755–760. 278 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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