Gerd Walz

22.8k total citations · 4 hit papers
222 papers, 14.1k citations indexed

About

Gerd Walz is a scholar working on Molecular Biology, Genetics and Nephrology. According to data from OpenAlex, Gerd Walz has authored 222 papers receiving a total of 14.1k indexed citations (citations by other indexed papers that have themselves been cited), including 127 papers in Molecular Biology, 97 papers in Genetics and 40 papers in Nephrology. Recurrent topics in Gerd Walz's work include Genetic and Kidney Cyst Diseases (88 papers), Renal and related cancers (71 papers) and Renal Diseases and Glomerulopathies (35 papers). Gerd Walz is often cited by papers focused on Genetic and Kidney Cyst Diseases (88 papers), Renal and related cancers (71 papers) and Renal Diseases and Glomerulopathies (35 papers). Gerd Walz collaborates with scholars based in Germany, United States and United Kingdom. Gerd Walz's co-authors include Thomas Benzing, Tobias B. Huber, Brian Seed, Alejandro Aruffo, Waldemar Kolanus, Emily Kim, Thierry Arnould, Lorenz Sellin, E. Wolfgang Kuehn and Leonidas Tsiokas and has published in prestigious journals such as Science, New England Journal of Medicine and Cell.

In The Last Decade

Gerd Walz

217 papers receiving 13.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Recognition by Elam-1 of the Sialyl-Le ^x Determinant on Myeloid and Tumor Cells

1990 846 citations Gerd Walz et al. profile →
The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease

2006 612 citations Jonathan M. Shillingford, Noel Murcia et al. Proceedings of the National Academy of Sciences profile →
Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

2005 588 citations Matias Simons, Bernhard Schermer et al. profile →
Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice

2010 586 citations Björn Hartleben, Markus Gödel et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Gerd Walz Germany	60	8.6k	5.7k	2.6k	1.9k	1.8k	222	14.1k
Peter Mündel United States	74	8.3k 1.0×	3.2k 0.6×	12.8k 4.9×	1.8k 0.9×	1.6k 0.9×	158	20.0k
Katsuya Okawa Japan	52	10.3k 1.2×	1.8k 0.3×	1.6k 0.6×	650 0.3×	3.9k 2.1×	118	15.9k
Allen M. Spiegel United States	79	10.0k 1.2×	2.7k 0.5×	3.2k 1.2×	763 0.4×	2.1k 1.1×	294	19.0k
David P. Witte United States	65	5.9k 0.7×	1.6k 0.3×	802 0.3×	752 0.4×	1.3k 0.7×	240	13.3k
Aristidis Moustakas Sweden	68	13.1k 1.5×	1.3k 0.2×	380 0.1×	1.4k 0.7×	1.5k 0.8×	161	17.8k
Herbert Y. Lin United States	55	6.5k 0.8×	846 0.1×	1.1k 0.4×	567 0.3×	797 0.4×	110	13.7k
Amparo Cano Spain	59	15.7k 1.8×	2.0k 0.4×	280 0.1×	1.5k 0.8×	2.4k 1.3×	134	22.1k
Magnus Nordenskjöld Sweden	57	4.8k 0.6×	3.7k 0.6×	265 0.1×	1.4k 0.7×	713 0.4×	241	12.9k
Diego H. Castrillón United States	50	7.5k 0.9×	1.8k 0.3×	404 0.2×	606 0.3×	925 0.5×	111	12.4k
Masahide Takahashi Japan	65	8.9k 1.0×	1.8k 0.3×	237 0.1×	746 0.4×	1.8k 1.0×	367	16.0k

Countries citing papers authored by Gerd Walz

Since Specialization

Citations

This map shows the geographic impact of Gerd Walz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gerd Walz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gerd Walz more than expected).

Fields of papers citing papers by Gerd Walz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gerd Walz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gerd Walz. The network helps show where Gerd Walz may publish in the future.

Co-authorship network of co-authors of Gerd Walz

This figure shows the co-authorship network connecting the top 25 collaborators of Gerd Walz. A scholar is included among the top collaborators of Gerd Walz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gerd Walz. Gerd Walz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Franz, Henriette, et al.. (2025). Targeting GLP-1 Signaling Ameliorates Cystogenesis in a Zebrafish Model of Nephronophthisis. International Journal of Molecular Sciences. 26(15). 7366–7366.

Laggner, Christian, et al.. (2024). Discovery of a Small Molecule with an Inhibitory Role for RAB11. International Journal of Molecular Sciences. 25(23). 13224–13224.

Schneider, Johanna, et al.. (2023). Nirmatrelvir/ritonavir treatment in SARS-CoV-2 positive kidney transplant recipients – a case series with four patients. BMC Nephrology. 24(1). 99–99. 3 indexed citations

Lang, Konrad, Claire Leroy, Mengmeng Chen, et al.. (2022). Nephrotic Syndrome Gene TBC1D8B Is Required for Endosomal Maturation and Nephrin Endocytosis in Drosophila. Journal of the American Society of Nephrology. 33(12). 2174–2193. 12 indexed citations

Lang, Konrad, Mengmeng Chen, Lea Gerstner, et al.. (2022). Selective endocytosis controls slit diaphragm maintenance and dynamics in Drosophila nephrocytes. eLife. 11. 19 indexed citations

Yasunaga, Takayuki, Martin Helmstädter, Daniel Epting, et al.. (2022). Microridge-like structures anchor motile cilia. Nature Communications. 13(1). 2056–2056. 16 indexed citations

Rieg, Siegbert, Elke Neumann‐Haefelin, Paul Biever, et al.. (2020). Comparison of different anticoagulation strategies for renal replacement therapy in critically ill patients with COVID-19: a cohort study. BMC Nephrology. 21(1). 20 indexed citations

Yuan, Jia, Shizu Aikawa, Wenbo Deng, et al.. (2019). Primary decidual zone formation requires Scribble for pregnancy success in mice. Nature Communications. 10(1). 5425–5425. 50 indexed citations

Hoff, Sylvia, Daniel Epting, Nathalie Falk, et al.. (2018). The nucleoside-diphosphate kinase NME3 associates with nephronophthisis proteins and is required for ciliary function during renal development. Journal of Biological Chemistry. 293(39). 15243–15255. 11 indexed citations

10.

Shamseldin, Hanan E., Toma A. Yakulov, Amal Hashem, Gerd Walz, & Fowzan S. Alkuraya. (2016). ANKS3 is mutated in a family with autosomal recessive laterality defect. Human Genetics. 135(11). 1233–1239. 15 indexed citations

11.

Yakulov, Toma A. & Gerd Walz. (2015). Zebrafish Database: Customizable, Free, and Open-Source Solution for Facility Management. Zebrafish. 12(6). 462–469. 5 indexed citations

12.

Prentzell, Mirja Tamara, Birgit Holzwarth, Kathrin Kläsener, et al.. (2015). TSC1 Activates TGF-β-Smad2/3 Signaling in Growth Arrest and Epithelial-to-Mesenchymal Transition. Developmental Cell. 32(5). 617–630. 52 indexed citations

13.

Yakulov, Toma A., Takayuki Yasunaga, Haribaskar Ramachandran, et al.. (2015). Anks3 interacts with nephronophthisis proteins and is required for normal renal development. Kidney International. 87(6). 1191–1200. 30 indexed citations

14.

Grahammer, Florian, Malte Roerden, Nicola Wanner, et al.. (2014). mTORC1 maintains renal tubular homeostasis and is essential in response to ischemic stress. Proceedings of the National Academy of Sciences. 111(27). E2817–26. 83 indexed citations

15.

Wanner, Nicola, Björn Hartleben, Nadja Herbach, et al.. (2014). Unraveling the Role of Podocyte Turnover in Glomerular Aging and Injury. Journal of the American Society of Nephrology. 25(4). 707–716. 143 indexed citations

16.

Hartleben, Björn, Markus Gödel, Catherine Meyer‐Schwesinger, et al.. (2010). Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice. Journal of Clinical Investigation. 120(4). 1084–1096. 586 indexed citations breakdown →

17.

Skouloudaki, Kassiani, Matias Simons, Christopher Boehlke, et al.. (2009). Scribble participates in Hippo signaling and is required for normal zebrafish pronephros development. Proceedings of the National Academy of Sciences. 106(21). 8579–8584. 116 indexed citations

18.

Schermer, Bernhard, Cristina Ghenoiu, Malte P. Bartram, et al.. (2006). The von Hippel-Lindau tumor suppressor protein controls ciliogenesis by orienting microtubule growth. The Journal of Cell Biology. 175(4). 547–554. 145 indexed citations

19.

Shillingford, Jonathan M., Noel Murcia, Seng Hui Low, et al.. (2006). The mTOR pathway is regulated by polycystin-1, and its inhibition reverses renal cystogenesis in polycystic kidney disease. Proceedings of the National Academy of Sciences. 103(14). 5466–5471. 612 indexed citations breakdown →

20.

Huber, Tobias B., Bernhard Schermer, Roman Ulrich Müller, et al.. (2006). Podocin and MEC-2 bind cholesterol to regulate the activity of associated ion channels. Proceedings of the National Academy of Sciences. 103(46). 17079–17086. 236 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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