Sybille Koehler

529 total citations
19 papers, 226 citations indexed

About

Sybille Koehler is a scholar working on Nephrology, Molecular Biology and Genetics. According to data from OpenAlex, Sybille Koehler has authored 19 papers receiving a total of 226 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Nephrology, 10 papers in Molecular Biology and 8 papers in Genetics. Recurrent topics in Sybille Koehler's work include Renal Diseases and Glomerulopathies (14 papers), Genetic and Kidney Cyst Diseases (8 papers) and Renal and related cancers (3 papers). Sybille Koehler is often cited by papers focused on Renal Diseases and Glomerulopathies (14 papers), Genetic and Kidney Cyst Diseases (8 papers) and Renal and related cancers (3 papers). Sybille Koehler collaborates with scholars based in Germany, United States and United Kingdom. Sybille Koehler's co-authors include Thomas Benzing, Bernhard Schermer, Paul T. Brinkkoetter, Markus M. Rinschen, Martin Kann, Tobias B. Huber, Christina Ising, Henning Hagmann, Sebastian Brähler and Christina B. Schroeter and has published in prestigious journals such as Nature Communications, Scientific Reports and The FASEB Journal.

In The Last Decade

Sybille Koehler

19 papers receiving 225 citations

Peers

Sybille Koehler
Beate Geist Germany
Patricia Schröder United States
Priyanka Rashmi United States
Lynne Staggs United States
Jenny Hurcombe United Kingdom
Enriko Klootwijk United Kingdom
Clare Thakker United Kingdom
Georgina Gyarmati United States
Keith A. Choate United States
Beate Geist Germany
Sybille Koehler
Citations per year, relative to Sybille Koehler Sybille Koehler (= 1×) peers Beate Geist

Countries citing papers authored by Sybille Koehler

Since Specialization
Citations

This map shows the geographic impact of Sybille Koehler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sybille Koehler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sybille Koehler more than expected).

Fields of papers citing papers by Sybille Koehler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sybille Koehler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sybille Koehler. The network helps show where Sybille Koehler may publish in the future.

Co-authorship network of co-authors of Sybille Koehler

This figure shows the co-authorship network connecting the top 25 collaborators of Sybille Koehler. A scholar is included among the top collaborators of Sybille Koehler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sybille Koehler. Sybille Koehler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Edenhofer, Ilka, Maja T. Lindenmeyer, Christian Lohr, et al.. (2024). Piezo activity levels need to be tightly regulated to maintain normal morphology and function in pericardial nephrocytes. Scientific Reports. 14(1). 28254–28254. 4 indexed citations
2.
Loreth, Desirée, Sybille Koehler, Marlies Sachs, et al.. (2024). The proteasome modulates endocytosis specifically in glomerular cells to promote kidney filtration. Nature Communications. 15(1). 1897–1897. 4 indexed citations
3.
Koehler, Sybille, Felicitas E. Hengel, Bernhard Dumoulin, et al.. (2024). The 14th International Podocyte Conference 2023: from podocyte biology to glomerular medicine. Kidney International. 105(5). 935–952. 2 indexed citations
4.
Koehler, Sybille & Tobias B. Huber. (2023). Insights into human kidney function from the study of Drosophila. Pediatric Nephrology. 38(12). 3875–3887. 8 indexed citations
5.
Koehler, Sybille, Tobias B. Huber, & Barry Denholm. (2022). A protective role forDrosophilaFilamin in nephrocytes via Yorkie mediated hypertrophy. Life Science Alliance. 5(12). e202101281–e202101281. 7 indexed citations
6.
Höhne, Martin, Maximilian Hohenadel, Heike Göbel, et al.. (2022). Modeling of ACTN4-Based Podocytopathy Using Drosophila Nephrocytes. Kidney International Reports. 8(2). 317–329. 6 indexed citations
7.
Koehler, Sybille & Barry Denholm. (2021). The Mechanosensitive Ion Channel Piezo Activates Rho1 in Drosophila Nephrocytes. Journal of the American Society of Nephrology. 32(10S). 525–525. 1 indexed citations
8.
Pippin, Jeffrey W., Sybille Koehler, Stuart J. Shankland, et al.. (2021). The Atypical Cyclin-Dependent Kinase 5 (Cdk5) Guards Podocytes from Apoptosis in Glomerular Disease While Being Dispensable for Podocyte Development. Cells. 10(9). 2464–2464. 9 indexed citations
9.
Jüngst, Christian, Markus M. Rinschen, Sybille Koehler, et al.. (2020). Injured Podocytes Are Sensitized to Angiotensin II–Induced Calcium Signaling. Journal of the American Society of Nephrology. 31(3). 532–542. 26 indexed citations
10.
Koehler, Sybille, Christian Jüngst, Martin Hoehne, et al.. (2020). Proteome Analysis of Isolated Podocytes Reveals Stress Responses in Glomerular Sclerosis. Journal of the American Society of Nephrology. 31(3). 544–559. 21 indexed citations
11.
Koehler, Sybille, Paul T. Brinkkoetter, & Markus M. Rinschen. (2020). Podocyte proteome analysis reveals stress responses in glomerular sclerosis. The FASEB Journal. 34(S1). 1–1. 2 indexed citations
12.
Schroeter, Christina B., Sybille Koehler, Martin Kann, et al.. (2018). Protein half‐life determines expression of proteostatic networks in podocyte differentiation. The FASEB Journal. 32(9). 4696–4713. 13 indexed citations
13.
Koehler, Sybille, Sebastian Brähler, Fabian Braun, et al.. (2017). Construction of a viral T2A-peptide based knock-in mouse model for enhanced Cre recombinase activity and fluorescent labeling of podocytes. Kidney International. 91(6). 1510–1517. 11 indexed citations
14.
Koehler, Sybille, Sebastian Brähler, Matthias J. Hackl, et al.. (2016). Single and Transient Ca2+ Peaks in Podocytes do not induce Changes in Glomerular Filtration and Perfusion. Scientific Reports. 6(1). 35400–35400. 12 indexed citations
15.
Ising, Christina, Puneet Bharill, Sebastian Brähler, et al.. (2016). Prohibitin-2 Depletion Unravels Extra-Mitochondrial Functions at the Kidney Filtration Barrier. American Journal Of Pathology. 186(5). 1128–1139. 12 indexed citations
16.
Koehler, Sybille, Frederik Tellkamp, Carien M. Niessen, et al.. (2016). Par3A is dispensable for the function of the glomerular filtration barrier of the kidney. American Journal of Physiology-Renal Physiology. 311(1). F112–F119. 8 indexed citations
17.
Rinschen, Markus M., Christina B. Schroeter, Sybille Koehler, et al.. (2016). Quantitative deep mapping of the cultured podocyte proteome uncovers shifts in proteostatic mechanisms during differentiation. American Journal of Physiology-Cell Physiology. 311(3). C404–C417. 25 indexed citations
18.
Ising, Christina, Sybille Koehler, Sebastian Brähler, et al.. (2015). Inhibition of insulin/ IGF ‐1 receptor signaling protects from mitochondria‐mediated kidney failure. EMBO Molecular Medicine. 7(3). 275–287. 54 indexed citations
19.
Braendle, W., et al.. (1989). GnRH-agonists induce modified endocrine response to gonadotropins. European Journal of Endocrinology. 120(3_Suppl). S68–S69. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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