Hermann Pavenstädt

16.2k total citations · 3 hit papers
122 papers, 7.6k citations indexed

About

Hermann Pavenstädt is a scholar working on Nephrology, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Hermann Pavenstädt has authored 122 papers receiving a total of 7.6k indexed citations (citations by other indexed papers that have themselves been cited), including 65 papers in Nephrology, 45 papers in Molecular Biology and 15 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Hermann Pavenstädt's work include Renal Diseases and Glomerulopathies (54 papers), Chronic Kidney Disease and Diabetes (22 papers) and Ion Transport and Channel Regulation (14 papers). Hermann Pavenstädt is often cited by papers focused on Renal Diseases and Glomerulopathies (54 papers), Chronic Kidney Disease and Diabetes (22 papers) and Ion Transport and Channel Regulation (14 papers). Hermann Pavenstädt collaborates with scholars based in Germany, United States and Switzerland. Hermann Pavenstädt's co-authors include Wilhelm Kriz, Matthias Kretzler, Peter Mündel, Aimée Zúñiga, Rolf Zeller, Jochen Reiser, Gary Davidson, Tobias B. Huber, Hans Oberleithner and Gerd Walz and has published in prestigious journals such as Journal of Clinical Investigation, Physiological Reviews and PLoS ONE.

In The Last Decade

Hermann Pavenstädt

120 papers receiving 7.5k citations

Hit Papers

Cell Biology of the Glomerular Podocyte 1997 2026 2006 2016 2003 1997 2010 250 500 750 1000
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Cell Biology of the Glomerular Podocyte
    2003 1.2k citations Hermann Pavenstädt et al. profile →
  2. Rearrangements of the Cytoskeleton and Cell Contacts Induce Process Formation during Differentiation of Conditionally Immortalized Mouse Podocyte Cell Lines
    1997 790 citations Peter Mündel, Hermann Pavenstädt et al. profile →
  3. Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice
    2010 586 citations Björn Hartleben, Markus Gödel et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hermann Pavenstädt Germany 41 4.1k 2.8k 979 903 773 122 7.6k
Clemens D. Cohen Germany 57 3.7k 0.9× 3.7k 1.3× 965 1.0× 1.1k 1.3× 878 1.1× 146 9.8k
Hermann Pavenstädt Germany 38 1.9k 0.5× 2.2k 0.8× 793 0.8× 812 0.9× 631 0.8× 167 6.7k
Emile de Heer Netherlands 47 2.6k 0.6× 2.9k 1.0× 1.7k 1.7× 1.1k 1.2× 478 0.6× 204 8.5k
Christian Faul United States 38 4.9k 1.2× 2.9k 1.0× 1.3k 1.3× 634 0.7× 260 0.3× 89 7.3k
Gavin I. Welsh United Kingdom 47 2.0k 0.5× 3.5k 1.3× 606 0.6× 970 1.1× 387 0.5× 141 6.6k
Maria Pia Rastaldi Italy 43 4.1k 1.0× 2.6k 0.9× 709 0.7× 759 0.8× 392 0.5× 120 7.3k
Taiji Matsusaka Japan 43 1.9k 0.5× 2.9k 1.0× 502 0.5× 643 0.7× 1.1k 1.4× 124 7.4k
Lawrence B. Holzman United States 58 5.7k 1.4× 5.1k 1.8× 1.7k 1.7× 913 1.0× 298 0.4× 115 9.9k
Liliane J. Striker United States 48 2.1k 0.5× 2.2k 0.8× 933 1.0× 828 0.9× 232 0.3× 115 6.9k
Kenichi Shikata Japan 41 1.6k 0.4× 1.8k 0.6× 387 0.4× 725 0.8× 898 1.2× 177 6.1k

Countries citing papers authored by Hermann Pavenstädt

Since Specialization
Citations

This map shows the geographic impact of Hermann Pavenstädt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hermann Pavenstädt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hermann Pavenstädt more than expected).

Fields of papers citing papers by Hermann Pavenstädt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hermann Pavenstädt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hermann Pavenstädt. The network helps show where Hermann Pavenstädt may publish in the future.

Co-authorship network of co-authors of Hermann Pavenstädt

This figure shows the co-authorship network connecting the top 25 collaborators of Hermann Pavenstädt. A scholar is included among the top collaborators of Hermann Pavenstädt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hermann Pavenstädt. Hermann Pavenstädt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Beul, Katrin, et al.. (2023). The Endothelial Glycocalyx as a Target of Excess Soluble Fms-like Tyrosine Kinase-1. International Journal of Molecular Sciences. 24(6). 5380–5380. 4 indexed citations
2.
Jehn, U., Katharina Schütte‐Nütgen, Gerold Thölking, et al.. (2022). The Influence of Parathyroidectomy on Osteoporotic Fractures in Kidney Transplant Recipients: Results from a Retrospective Single-Center Trial. Journal of Clinical Medicine. 11(3). 654–654. 1 indexed citations
3.
Breljak, Davorka, et al.. (2022). Impact of Pals1 on Expression and Localization of Transporters Belonging to the Solute Carrier Family. Frontiers in Molecular Biosciences. 9. 792829–792829. 2 indexed citations
4.
Janning, Annette, Jan Halbritter, Michael P. Krahn, et al.. (2020). A Deregulated Stress Response Underlies Distinct INF2-Associated Disease Profiles. Journal of the American Society of Nephrology. 31(6). 1296–1313. 22 indexed citations
5.
Jehn, U., Katharina Schütte‐Nütgen, Jan Kunert, et al.. (2020). Antihypertensive Treatment in Kidney Transplant Recipients—A Current Single Center Experience. Journal of Clinical Medicine. 9(12). 3969–3969. 3 indexed citations
6.
Skryabin, Boris V., Laura Katharina Sievers, Barbara Heitplatz, et al.. (2020). TrkC Is Essential for Nephron Function and Trans-Activates Igf1R Signaling. Journal of the American Society of Nephrology. 32(2). 357–374. 4 indexed citations
7.
Rovas, Alexandros, Kristina Kusche‐Vihrog, Paul Van Slyke, et al.. (2019). Tie2 Activation Promotes Protection and Reconstitution of the Endothelial Glycocalyx in Human Sepsis. Thrombosis and Haemostasis. 119(11). 1827–1838. 39 indexed citations
8.
Rovas, Alexandros, Konrad Buscher, Kristina Kusche‐Vihrog, et al.. (2019). Symmetric dimethylarginine in dysfunctional high-density lipoprotein mediates endothelial glycocalyx breakdown in chronic kidney disease. Kidney International. 97(3). 502–515. 18 indexed citations
9.
Lenders, Malte, Boris Schmitz, Bernd Kasprzak, et al.. (2015). Differential response to endothelial epithelial sodium channel inhibition ex vivo correlates with arterial stiffness in humans. Journal of Hypertension. 33(12). 2455–2462. 15 indexed citations
10.
Kliche, Katrin, et al.. (2015). Recharging Red Blood Cell Surface by Hemodialysis. Cellular Physiology and Biochemistry. 35(3). 1107–1115. 6 indexed citations
11.
Wennmann, Dirk Oliver, Beate Vollenbröker, Frank Erdmann, et al.. (2014). The Hippo pathway is controlled by Angiotensin II signaling and its reactivation induces apoptosis in podocytes. Cell Death and Disease. 5(11). e1519–e1519. 82 indexed citations
12.
Hartleben, Björn, Markus Gödel, Catherine Meyer‐Schwesinger, et al.. (2010). Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice. Journal of Clinical Investigation. 120(4). 1084–1096. 586 indexed citations breakdown →
13.
Ren, Shuyu, Andrea Bábelová, Kristin Moreth, et al.. (2009). Transforming growth factor-β2 upregulates sphingosine kinase-1 activity, which in turn attenuates the fibrotic response to TGF-β2 by impeding CTGF expression. Kidney International. 76(8). 857–867. 70 indexed citations
14.
Geyer, Marcel, et al.. (2008). α/β hydrolase 1 is upregulated in D5 dopamine receptor knockout mice and reduces O2- production of NADPH oxidase. Biochemical and Biophysical Research Communications. 379(1). 81–85. 13 indexed citations
15.
Pache, Gregor, Sebastian Wiesemann, Erik Springer, et al.. (2006). Upregulation of Id-1 via BMP-2 receptors induces reactive oxygen species in podocytes. American Journal of Physiology-Renal Physiology. 291(3). F654–F662. 19 indexed citations
16.
Huwiler, Andrea, Shuyu Ren, Harry Holthöfer, Hermann Pavenstädt, & Josef Pfeilschifter. (2003). Inflammatory cytokines upregulate nephrin expression in human embryonic kidney epithelial cells and podocytes. Biochemical and Biophysical Research Communications. 305(1). 136–142. 31 indexed citations
17.
Fischer, Karl-Georg, Tobias B. Huber, Anna Henger, et al.. (2002). Eluate Derived by Extracorporal Antibody-Based Immunoadsorption Elevates the Cytosolic Ca<sup>2+</sup> Concentration in Podocytes via B<sub>2</sub> Kinin Receptors. Kidney & Blood Pressure Research. 25(6). 384–393. 3 indexed citations
18.
Gloy, Joachim, Anna Henger, K.-G. Fischer, et al.. (1997). Angiotensin II depolarizes podocytes in the intact glomerulus of the Rat.. Journal of Clinical Investigation. 99(11). 2772–2781. 73 indexed citations
19.
Krämer‐Guth, Annette, Hermann Pavenstädt, Thomas Quaschning, et al.. (1996). Interaction of native and oxidized lipoprotein(a) with human mesangial cells and matrix. Kidney International. 49(5). 1250–1261. 19 indexed citations
20.
Pavenstädt, Hermann, Anna Henger, Verena Briner, R. Greger, & P. Schollmeyer. (1996). Extracellular ATP regulates glomerular endothelial cell function. Journal of Autonomic Pharmacology. 16(6). 389–392. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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