Lawrence D. Schweitzer
- Molecular Biology top 10%
- Cell Biology top 5%
- Epidemiology
- Immunology
- Cancer Research top 10%
- Co-authors
- David M. SabatiniLiron Bar‐PeledRoberto ZoncuNir HacohenTenzin KunchokElizaveta FreinkmanMonther Abu-RemailehSze Ham Chan
- Topics
- PI3K/AKT/mTOR signaling in cancer (4 papers)Protein Kinase Regulation and GTPase Signaling (2 papers)Immunotherapy and Immune Responses (2 papers)
- Cited by
- PhysiologyCell BiologyAging
- Journals
- NatureCellScientific Reports
- Partner nations
- United StatesItaly
In The Last Decade
Lawrence D. Schweitzer
10 papers receiving 1.3k citations
Hit Papers
Peers
Comparison fields: 5 of 96
- Molecular Biology 920
- Cell Biology 309
- Epidemiology 200
- Immunology 190
- Cancer Research 160
Countries citing papers authored by Lawrence D. Schweitzer
This map shows the geographic impact of Lawrence D. Schweitzer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lawrence D. Schweitzer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lawrence D. Schweitzer more than expected).
Fields of papers citing papers by Lawrence D. Schweitzer
This network shows the impact of papers produced by Lawrence D. Schweitzer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lawrence D. Schweitzer. The network helps show where Lawrence D. Schweitzer may publish in the future.
Co-authorship network of co-authors of Lawrence D. Schweitzer
This figure shows the co-authorship network connecting the top 25 collaborators of Lawrence D. Schweitzer. A scholar is included among the top collaborators of Lawrence D. Schweitzer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lawrence D. Schweitzer. Lawrence D. Schweitzer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 1 | |
| 3 | 71 | |
| 4 | 226 | |
| 5 | 171 | |
| 6 | 26 | |
| 7 | 12 | |
| 8 | 71 | |
| 9 | Ragulator Is a GEF for the Rag GTPases that Signal Amino Acid Levels to mTORC1breakdown → | 714 |
| 10 | Glutaric acidemia type II and flavin-dependent enzymes in morphogenesis. | 10 |
About Lawrence D. Schweitzer
Lawrence D. Schweitzer is a scholar working on Immunology and Allergy, Clinical Biochemistry and Immunology, having authored 10 papers that have together received 1.3k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (4 papers), Protein Kinase Regulation and GTPase Signaling (2 papers) and Immunotherapy and Immune Responses (2 papers). The work is most often cited by research in Physiology (120 citations), Cell Biology (309 citations) and Aging (29 citations). Lawrence D. Schweitzer has collaborated with scholars based in United States and Italy. Frequent co-authors include David M. Sabatini, Liron Bar‐Peled, Roberto Zoncu, Nir Hacohen, Tenzin Kunchok, Elizaveta Freinkman, Monther Abu-Remaileh, Sze Ham Chan, Naama Kanarek and Caroline A. Lewis. Their work appears in journals such as Nature, Cell and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.