Klaus Willecke
- Sensory Systems top 0.1%
- Cellular and Molecular Neuroscience top 0.1%
- Neuroscience and Neuropharmacology Research 37
- Molecular Biology top 0.05%
- Connexins and lens biology 266
- Heat shock proteins research 83
- Nicotinic Acetylcholine Receptors Study 80
- Ion channel regulation and function 24
- Endocrine and Autonomic Systems top 0.2%
- Neuroscience of respiration and sleep 19
- Neurology top 0.2%
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- Biochemical effects in animals 25
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- Yersinia bacterium, plague, ectoparasites research 22
Klaus Willecke
352 papers receiving 28.1k citations
Hit Papers
Peers
Comparison fields: 5 of 153
- Sensory Systems 1.8k
- Cellular and Molecular Neuroscience 6.3k
- Molecular Biology 23.3k
- Endocrine and Autonomic Systems 1.9k
- Neurology 1.9k
Countries citing papers authored by Klaus Willecke
This map shows the geographic impact of Klaus Willecke's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus Willecke with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus Willecke more than expected).
Fields of papers citing papers by Klaus Willecke
This network shows the impact of papers produced by Klaus Willecke. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus Willecke. The network helps show where Klaus Willecke may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Klaus Willecke, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 12 | |
| 2 | 2013 | 96 | |
| 3 | 2013 | 89 | |
| 4 | 2012 | 112 | |
| 5 | 2011 | 241 | |
| 6 | 2011 | 31 | |
| 7 | 2010 | 58 | |
| 8 | 2008 | 51 | |
| 9 | 2007 | 13 | |
| 10 | 2006 | 88 | |
| 11 | 2006 | 91 | |
| 12 | 2004 | 37 | |
| 13 | 2004 | 128 | |
| 14 | 2004 | 106 | |
| 15 | 2003 | 59 | |
| 16 | Cellular Expression of Neural Connexin36 in the Outer Retina of the Mouse | 2003 | 2 |
| 17 | 2002 | 84 | |
| 18 | 2001 | 16 | |
| 19 | 2001 | 30 | |
| 20 | 1998 | 239 |
About Klaus Willecke
Klaus Willecke is a scholar working on Molecular Biology, Sensory Systems and Endocrine and Autonomic Systems, having authored 353 papers that have together received 28.5k indexed citations. Recurring topics across this work include Connexins and lens biology (266 papers), Heat shock proteins research (83 papers), Nicotinic Acetylcholine Receptors Study (80 papers), Neuroscience and Neuropharmacology Research (37 papers), Biochemical effects in animals (25 papers), Ion channel regulation and function (24 papers), Yersinia bacterium, plague, ectoparasites research (22 papers) and Neuroscience of respiration and sleep (19 papers). The work is most often cited by research in Sensory Systems (1.8k citations), Cellular and Molecular Neuroscience (6.3k citations) and Molecular Biology (23.3k citations). Klaus Willecke has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Otto Traub, Goran Söhl, Martin Theis, Joachim Degen, Rolf Dermietzel, Stephan Maxeiner, C. Elfgang, Feliksas F. Bukauskas, Martin Güldenagel and Dominik Eckardt. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.