Thomas Ott

3.8k total citations
61 papers, 2.7k citations indexed

About

Thomas Ott is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Thomas Ott has authored 61 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Molecular Biology, 16 papers in Cellular and Molecular Neuroscience and 11 papers in Neurology. Recurrent topics in Thomas Ott's work include Connexins and lens biology (24 papers), Heat shock proteins research (15 papers) and Neurological disorders and treatments (9 papers). Thomas Ott is often cited by papers focused on Connexins and lens biology (24 papers), Heat shock proteins research (15 papers) and Neurological disorders and treatments (9 papers). Thomas Ott collaborates with scholars based in Germany, United States and United Kingdom. Thomas Ott's co-authors include Klaus Willecke, Dominik Eckardt, Robert P. Millar, Robin Sellar, Sonja Hombach, Vincent Michel, Martine Cohen‐Salmon, Daniel C. Marcus, Jean-Pierre Hardelin and A. Paula Monaghan and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Thomas Ott

60 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Ott Germany 29 1.9k 543 394 318 307 61 2.7k
J. Fielding Hejtmancik United States 32 1.9k 1.0× 279 0.5× 312 0.8× 162 0.5× 403 1.3× 63 2.8k
Ingo Kurth Germany 31 1.6k 0.9× 730 1.3× 106 0.3× 139 0.4× 399 1.3× 110 3.5k
Emerald Perlas Italy 27 1.4k 0.7× 401 0.7× 63 0.2× 625 2.0× 345 1.1× 39 2.8k
Stéphane Schurmans Belgium 32 2.3k 1.3× 983 1.8× 541 1.4× 94 0.3× 912 3.0× 94 4.1k
Elena I. Rugarli Germany 39 4.4k 2.4× 1.1k 2.0× 80 0.2× 329 1.0× 566 1.8× 80 5.8k
Kimia Kahrizi Iran 30 1.8k 1.0× 203 0.4× 826 2.1× 271 0.9× 835 2.7× 141 3.0k
Vivian R. Albert United States 26 1.9k 1.0× 567 1.0× 94 0.2× 153 0.5× 968 3.2× 42 3.5k
Mario Vallejo Spain 32 2.0k 1.1× 844 1.6× 48 0.1× 413 1.3× 819 2.7× 81 3.8k
Otto Traub Germany 49 7.2k 3.8× 878 1.6× 632 1.6× 201 0.6× 887 2.9× 104 8.2k
Yoshinobu Sugitani Japan 18 1.9k 1.0× 259 0.5× 350 0.9× 179 0.6× 382 1.2× 26 3.1k

Countries citing papers authored by Thomas Ott

Since Specialization
Citations

This map shows the geographic impact of Thomas Ott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Ott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Ott more than expected).

Fields of papers citing papers by Thomas Ott

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Ott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Ott. The network helps show where Thomas Ott may publish in the future.

Co-authorship network of co-authors of Thomas Ott

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Ott. A scholar is included among the top collaborators of Thomas Ott based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Ott. Thomas Ott is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ott, Thomas, Hong T. Nguyen, Nuran Ay, et al.. (2025). Efficient synthesis of well-defined ordered mesoporous aluminosilicates with tailorable acidity. Microporous and Mesoporous Materials. 402. 113991–113991.
2.
Knörr, Susanne, Muthuraman Muthuraman, Thomas Ott, et al.. (2024). Disturbed brain energy metabolism in a rodent model of DYT-TOR1A dystonia. Neurobiology of Disease. 194. 106462–106462. 4 indexed citations
3.
Cheng, Fubo, Chang Liu, Peter A. Barbuti, et al.. (2022). Intronic enhancers of the human SNCA gene predominantly regulate its expression in brain in vivo. Science Advances. 8(47). eabq6324–eabq6324. 6 indexed citations
4.
Cheng, Fubo, Peter A. Barbuti, Paola Bonsi, et al.. (2022). DYT6 mutated THAP1 is a cell type dependent regulator of the SP1 family. Brain. 145(11). 3968–3984. 6 indexed citations
5.
Cheng, Fubo, Michael Walter, Zinah Wassouf, et al.. (2020). Unraveling Molecular Mechanisms of THAP1 Missense Mutations in DYT6 Dystonia. Journal of Molecular Neuroscience. 70(7). 999–1008. 14 indexed citations
6.
Yu-Taeger, Libo, Thomas Ott, Paola Bonsi, et al.. (2019). Impaired dopamine- and adenosine-mediated signaling and plasticity in a novel rodent model for DYT25 dystonia. Neurobiology of Disease. 134. 104634–104634. 21 indexed citations
7.
Yu-Taeger, Libo, Nicolas Casadei, Michael Walter, et al.. (2018). Dynamic nuclear envelope phenotype in rats overexpressing mutated human torsinA protein. Biology Open. 7(7). 2 indexed citations
8.
Levada, Kateryna, Nurdan Güldiken, Xiaoji Zhang, et al.. (2018). Hsp72 protects against liver injury via attenuation of hepatocellular death, oxidative stress, and JNK signaling. Journal of Hepatology. 68(5). 996–1005. 52 indexed citations
9.
Thunemann, Martin, Lai Wen, Matthias Hillenbrand, et al.. (2013). Transgenic Mice for cGMP Imaging. Circulation Research. 113(4). 365–371. 61 indexed citations
10.
Wei, Tao, Timm Schubert, François Paquet‐Durand, et al.. (2012). Light-Driven Calcium Signals in Mouse Cone Photoreceptors. Journal of Neuroscience. 32(20). 6981–6994. 28 indexed citations
11.
Willecke, Klaus, et al.. (2007). Biological Functions of Connexin Genes Revealed by Human Genetic Defects, Dominant Negative Approaches and Targeted Deletions in the Mouse. Novartis Foundation symposium. 219. 76–96. 13 indexed citations
12.
Ott, Thomas, et al.. (2006). The N-terminal domain of CCL21 reconstitutes high affinity binding, G protein activation, and chemotactic activity, to the C-terminal domain of CCL19. Biochemical and Biophysical Research Communications. 348(3). 1089–1093. 18 indexed citations
13.
Ott, Thomas, Anil Pahuja, Sarah A. Nickolls, David G. Alleva, & R. Scott Struthers. (2004). Identification of CC Chemokine Receptor 7 Residues Important for Receptor Activation. Journal of Biological Chemistry. 279(41). 42383–42392. 21 indexed citations
14.
Yoon, Byung–Il, Yoko Hirabayashi, Yasushi Kawasaki, et al.. (2003). Exacerbation of benzene pneumotoxicity in connexin 32 knockout mice: enhanced proliferation of CYP2E1-immunoreactive alveolar epithelial cells. Toxicology. 195(1). 19–29. 14 indexed citations
15.
Rabionet, Raquel, et al.. (2002). Human connexin26 (GJB2) deafness mutations affect the function of gap junction channels at different levels of protein expression.. Human Genetics. 111(2). 190–197. 84 indexed citations
16.
Ott, Thomas, Brigitte E. Troskie, Roger W. Roeske, et al.. (2002). Two Mutations in Extracellular Loop 2 of the Human GnRH Receptor Convert an Antagonist to an Agonist. Molecular Endocrinology. 16(5). 1079–1088. 32 indexed citations
17.
Temme, Achim, Frank Stümpel, Goran Söhl, et al.. (2001). Dilated bile canaliculi and attenuated decrease of nerve-dependent bile secretion in connexin32-deficient mouse liver. Pflügers Archiv - European Journal of Physiology. 442(6). 961–966. 39 indexed citations
18.
Willecke, Klaus, Achim Temme, Barbara Teubner, & Thomas Ott. (1999). Characterization of Targeted Connexin32‐Deficient Mice: A Model for the Human Charcot‐Marie‐Tooth (X‐Type) Inherited Disease. Annals of the New York Academy of Sciences. 883(1). 302–309. 11 indexed citations
19.
Stümpel, Frank, Thomas Ott, K. Willecke, & Kurt Jungermann. (1998). Connexin 32 Gap Junctions Enhance Stimulation of Glucose Output by Glucagon and Noradrenaline in Mouse Liver. Hepatology. 28(6). 1616–1620. 64 indexed citations
20.
Ott, Thomas, Klaus H. Kaestner, A. Paula Monaghan, & Günther Schütz. (1996). The mouse homolog of the region specific homeotic gene spalt of Drosophila is expressed in the developing nervous system and in mesoderm-derived structures. Mechanisms of Development. 56(1-2). 117–128. 64 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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