Bruno A. Cisterna

1.2k total citations
20 papers, 890 citations indexed

About

Bruno A. Cisterna is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Bruno A. Cisterna has authored 20 papers receiving a total of 890 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 6 papers in Physiology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Bruno A. Cisterna's work include Connexins and lens biology (10 papers), Muscle Physiology and Disorders (9 papers) and Adipose Tissue and Metabolism (4 papers). Bruno A. Cisterna is often cited by papers focused on Connexins and lens biology (10 papers), Muscle Physiology and Disorders (9 papers) and Adipose Tissue and Metabolism (4 papers). Bruno A. Cisterna collaborates with scholars based in Chile, United States and Germany. Bruno A. Cisterna's co-authors include Juan C. Sáez, Carlos Puebla, Christopher Cardozo, Cristián Vilos, A. Luis, Aníbal A. Vargas, Nazila Kamaly, Ali Tavakkoli, Won Il Choi and Omid C. Farokhzad and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The Journal of Cell Biology.

In The Last Decade

Bruno A. Cisterna

20 papers receiving 885 citations

Peers

Bruno A. Cisterna

PHYSI

CMN

BIOMA

Debora Lo Furno Italy

Yiyang Li China

Lin Teng China

Roberta Sessa Stilhano Brazil

Mahmood Barati Iran

Dong Hoon Hwang South Korea

Dongze Zhang United States

Massimiliano De Paola Italy

Kevin M. Tharp United States

Debora Lo Furno Italy

Bruno A. Cisterna

479 ×0.9

156 ×0.9

PHYSI

146 ×0.9

CMN

148 ×1.0

BIOMA

220 ×1.6

Citations per year, relative to Bruno A. Cisterna Bruno A. Cisterna (= 1×) peers Debora Lo Furno

Countries citing papers authored by Bruno A. Cisterna

Since Specialization

Citations

This map shows the geographic impact of Bruno A. Cisterna's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruno A. Cisterna with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruno A. Cisterna more than expected).

Fields of papers citing papers by Bruno A. Cisterna

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruno A. Cisterna. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruno A. Cisterna. The network helps show where Bruno A. Cisterna may publish in the future.

Co-authorship network of co-authors of Bruno A. Cisterna

This figure shows the co-authorship network connecting the top 25 collaborators of Bruno A. Cisterna. A scholar is included among the top collaborators of Bruno A. Cisterna based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bruno A. Cisterna. Bruno A. Cisterna is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cisterna, Bruno A., Kristen Skruber, Mitchell T. Butler, et al.. (2024). Prolonged depletion of profilin 1 or F-actin causes an adaptive response in microtubules. The Journal of Cell Biology. 223(7). 2 indexed citations

Read, Tracy‐Ann, Bruno A. Cisterna, Kristen Skruber, et al.. (2024). The actin binding protein profilin 1 localizes inside mitochondria and is critical for their function. EMBO Reports. 25(8). 3240–3262. 4 indexed citations

Vitriol, Eric A., et al.. (2023). Nonmuscle myosin 2 filaments are processive in cells. Biophysical Journal. 122(18). 3678–3689. 4 indexed citations

Cisterna, Bruno A., et al.. (2022). Advancements in the Use of Hydrogels for Regenerative Medicine: Properties and Biomedical Applications. International Journal of Biomaterials. 2022. 1–16. 122 indexed citations

Cisterna, Bruno A., Aníbal A. Vargas, Carlos Puebla, et al.. (2020). Active acetylcholine receptors prevent the atrophy of skeletal muscles and favor reinnervation. Nature Communications. 11(1). 1073–1073. 82 indexed citations

Cisterna, Bruno A., Pablo Arroyo, & Carlos Puebla. (2019). Role of Connexin-Based Gap Junction Channels in Communication of Myelin Sheath in Schwann Cells. Frontiers in Cellular Neuroscience. 13. 69–69. 19 indexed citations

Vargas, Aníbal A., Bruno A. Cisterna, A. Luis, et al.. (2017). On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells. Frontiers in Physiology. 8. 38–38. 22 indexed citations

Plotkin, Lilian I., Hannah M. Davis, Bruno A. Cisterna, & Juan C. Sáez. (2017). Connexins and Pannexins in Bone and Skeletal Muscle. Current Osteoporosis Reports. 15(4). 326–334. 35 indexed citations

Matus, María Francisca, Cristián Vilos, Bruno A. Cisterna, Eduardo Fuentes, & Iván Palomo. (2017). Nanotechnology and primary hemostasis: Differential effects of nanoparticles on platelet responses. Vascular Pharmacology. 101. 1–8. 32 indexed citations

10.

Luis, A., Elisa Balboa, Carlos Puebla, et al.. (2016). Dexamethasone-induced muscular atrophy is mediated by functional expression of connexin-based hemichannels. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1862(10). 1891–1899. 44 indexed citations

11.

Puebla, Carlos, et al.. (2016). Linoleic acid permeabilizes gastric epithelial cells by increasing connexin 43 levels in the cell membrane via a GPR40- and Akt-dependent mechanism. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1861(5). 439–448. 24 indexed citations

12.

Cisterna, Bruno A., Aníbal A. Vargas, Carlos Puebla, & Juan C. Sáez. (2016). Connexin hemichannels explain the ionic imbalance and lead to atrophy in denervated skeletal muscles. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1862(11). 2168–2176. 19 indexed citations

13.

Luis, A., Carlos Puebla, Bruno A. Cisterna, et al.. (2016). Fast skeletal myofibers of mdx mouse, model of Duchenne muscular dystrophy, express connexin hemichannels that lead to apoptosis. Cellular and Molecular Life Sciences. 73(13). 2583–2599. 35 indexed citations

14.

Johnson, Ross G., Haiying Grunenwald, Timothy J. Robinson, et al.. (2016). Connexin Hemichannels: Methods for Dye Uptake and Leakage. The Journal of Membrane Biology. 249(6). 713–741. 40 indexed citations

15.

Cisterna, Bruno A., Nazila Kamaly, Won Il Choi, et al.. (2016). Targeted Nanoparticles for Colorectal Cancer. Nanomedicine. 11(18). 2443–2456. 127 indexed citations

16.

Sáez, Juan C., Bruno A. Cisterna, Aníbal A. Vargas, & Christopher Cardozo. (2015). Regulation of pannexin and connexin channels and their functional role in skeletal muscles. Cellular and Molecular Life Sciences. 72(15). 2929–2935. 16 indexed citations

17.

Cisterna, Bruno A., et al.. (2014). Neuronal involvement in muscular atrophy. Frontiers in Cellular Neuroscience. 8. 405–405. 39 indexed citations

18.

Luis, A., Bruno A. Cisterna, Carlos Puebla, et al.. (2013). De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy. Proceedings of the National Academy of Sciences. 110(40). 16229–16234. 96 indexed citations

19.

Luis, A., Manuel A. Riquelme, Bruno A. Cisterna, et al.. (2012). Connexin- and Pannexin-Based Channels in Normal Skeletal Muscles and Their Possible Role in Muscle Atrophy. The Journal of Membrane Biology. 245(8). 423–436. 35 indexed citations

20.

Court, Felipe A., Rajiv Midha, Bruno A. Cisterna, et al.. (2011). Morphological evidence for a transport of ribosomes from Schwann cells to regenerating axons. Glia. 59(10). 1529–1539. 93 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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