A. Luis

847 total citations
22 papers, 652 citations indexed

About

A. Luis is a scholar working on Molecular Biology, Physiology and Rehabilitation. According to data from OpenAlex, A. Luis has authored 22 papers receiving a total of 652 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 13 papers in Physiology and 3 papers in Rehabilitation. Recurrent topics in A. Luis's work include Connexins and lens biology (14 papers), Muscle Physiology and Disorders (11 papers) and Adipose Tissue and Metabolism (9 papers). A. Luis is often cited by papers focused on Connexins and lens biology (14 papers), Muscle Physiology and Disorders (11 papers) and Adipose Tissue and Metabolism (9 papers). A. Luis collaborates with scholars based in Chile, United States and Germany. A. Luis's co-authors include Juan C. Sáez, Carlos Puebla, Bruno A. Cisterna, Aníbal A. Vargas, Marina Frank, Klaus Willecke, Rosalba Escamilla, Manuel A. Riquelme, Christopher Cardozo and José Luis Vega and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and The Journal of Physiology.

In The Last Decade

A. Luis

20 papers receiving 648 citations

Peers

A. Luis

PHYSI

CMN

REHAB

EAS

Marion Pauly Canada

Aníbal A. Vargas Chile

Giulia Favaro Italy

Maitea Guridi United States

Heather N. Carter Canada

Hairui Yuan China

Sylviane Lagarrigue Switzerland

Matthew Triolo Canada

Rojina Ranjit United States

Gesa Santos Switzerland

Marion Pauly Canada

A. Luis

457 ×0.8

331 ×1.3

PHYSI

53 ×0.5

CMN

67 ×0.9

REHAB

23 ×0.4

EAS

Citations per year, relative to A. Luis A. Luis (= 1×) peers Marion Pauly

Countries citing papers authored by A. Luis

Since Specialization

Citations

This map shows the geographic impact of A. Luis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Luis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Luis more than expected).

Fields of papers citing papers by A. Luis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Luis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Luis. The network helps show where A. Luis may publish in the future.

Co-authorship network of co-authors of A. Luis

This figure shows the co-authorship network connecting the top 25 collaborators of A. Luis. A scholar is included among the top collaborators of A. Luis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. Luis. A. Luis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Balboa, Elisa, et al.. (2025). Connexin hemichannels and early atrophic signaling in muscle during sepsis. Frontiers in Physiology. 16. 1514769–1514769.

Toro, Carlos A., et al.. (2024). Pathophysiological role of connexin and pannexin hemichannels in neuromuscular disorders. The Journal of Physiology. 603(15). 4213–4235.

Luis, A., et al.. (2023). Skeletal Muscle Atrophy Induced by Diabetes Is Mediated by Non-Selective Channels and Prevented by Boldine. Biomolecules. 13(4). 708–708. 10 indexed citations

Luis, A., et al.. (2020). Blockade of Hemichannels Normalizes the Differentiation Fate of Myoblasts and Features of Skeletal Muscles from Dysferlin-Deficient Mice. International Journal of Molecular Sciences. 21(17). 6025–6025. 14 indexed citations

Bevilacqua, Jorge A., et al.. (2020). Myofibers deficient in connexins 43 and 45 expression protect mice from skeletal muscle and systemic dysfunction promoted by a dysferlin mutation. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1866(8). 165800–165800. 12 indexed citations

Cisterna, Bruno A., Aníbal A. Vargas, Carlos Puebla, et al.. (2020). Active acetylcholine receptors prevent the atrophy of skeletal muscles and favor reinnervation. Nature Communications. 11(1). 1073–1073. 82 indexed citations

Jara‐Gutiérrez, Carlos, et al.. (2020). N-Acetylcysteine Reduces Skeletal Muscles Oxidative Stress and Improves Grip Strength in Dysferlin-Deficient Bla/J Mice. International Journal of Molecular Sciences. 21(12). 4293–4293. 12 indexed citations

Luis, A., Elisa Balboa, Aníbal A. Vargas, et al.. (2019). De novo expression of functional connexins 43 and 45 hemichannels increases sarcolemmal permeability of skeletal myofibers during endotoxemia. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(10). 2765–2773. 17 indexed citations

Vargas, Aníbal A., Bruno A. Cisterna, A. Luis, et al.. (2017). On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells. Frontiers in Physiology. 8. 38–38. 22 indexed citations

10.

Luis, A., Elisa Balboa, Carlos Puebla, et al.. (2016). Dexamethasone-induced muscular atrophy is mediated by functional expression of connexin-based hemichannels. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1862(10). 1891–1899. 44 indexed citations

11.

Cárdenas, Ana M., Arlek M. González‐Jamett, A. Luis, Jorge A. Bevilacqua, & Pablo Caviedes. (2016). Dysferlin function in skeletal muscle: Possible pathological mechanisms and therapeutical targets in dysferlinopathies. Experimental Neurology. 283(Pt A). 246–254. 51 indexed citations

12.

Luis, A., Jorge A. Bevilacqua, Ana M. Cárdenas, et al.. (2016). The absence of dysferlin induces the expression of functional connexin-based hemichannels in human myotubes. BMC Cell Biology. 17(S1). 15–15. 19 indexed citations

13.

Luis, A., Carlos Puebla, Bruno A. Cisterna, et al.. (2016). Fast skeletal myofibers of mdx mouse, model of Duchenne muscular dystrophy, express connexin hemichannels that lead to apoptosis. Cellular and Molecular Life Sciences. 73(13). 2583–2599. 35 indexed citations

14.

Riquelme, Manuel A., A. Luis, José Luis Vega, et al.. (2015). Pannexin channels mediate the acquisition of myogenic commitment in C2C12 reserve cells promoted by P2 receptor activation. Frontiers in Cell and Developmental Biology. 3. 25–25. 12 indexed citations

15.

Figueroa, Vania, et al.. (2014). Extracellular gentamicin reduces the activity of connexin hemichannels and interferes with purinergic Ca2+ signaling in HeLa cells. Frontiers in Cellular Neuroscience. 8. 265–265. 23 indexed citations

16.

Luis, A., et al.. (2014). Pannexin 1 channels in skeletal muscles. Frontiers in Physiology. 5. 139–139. 21 indexed citations

17.

Luis, A., Bruno A. Cisterna, Carlos Puebla, et al.. (2013). De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy. Proceedings of the National Academy of Sciences. 110(40). 16229–16234. 96 indexed citations

18.

Riquelme, Manuel A., A. Luis, José Luis Vega, et al.. (2013). The ATP required for potentiation of skeletal muscle contraction is released via pannexin hemichannels. Neuropharmacology. 75. 594–603. 89 indexed citations

19.

Luis, A., Manuel A. Riquelme, Bruno A. Cisterna, et al.. (2012). Connexin- and Pannexin-Based Channels in Normal Skeletal Muscles and Their Possible Role in Muscle Atrophy. The Journal of Membrane Biology. 245(8). 423–436. 35 indexed citations

20.

Luis, A.. (2006). Thyroid hormone calorigenesis and mitochondrial redox signaling: upregulation of gene expression. Frontiers in bioscience. 12(1). 1220–1220. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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