Robert Feil

14.6k total citations · 3 hit papers
160 papers, 10.6k citations indexed

About

Robert Feil is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Robert Feil has authored 160 papers receiving a total of 10.6k indexed citations (citations by other indexed papers that have themselves been cited), including 100 papers in Molecular Biology, 66 papers in Physiology and 36 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Robert Feil's work include Nitric Oxide and Endothelin Effects (54 papers), Receptor Mechanisms and Signaling (36 papers) and Phosphodiesterase function and regulation (35 papers). Robert Feil is often cited by papers focused on Nitric Oxide and Endothelin Effects (54 papers), Receptor Mechanisms and Signaling (36 papers) and Phosphodiesterase function and regulation (35 papers). Robert Feil collaborates with scholars based in Germany, United States and France. Robert Feil's co-authors include Franz Hofmann, Susanne Feil, Daniel Metzger, Thomas Kleppisch, Pierre Chambon, Jens Schlossmann, Bénédicte Mascrez, Pierre Chambon, Marianne LeMeur and Jacques Brocard and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Robert Feil

154 papers receiving 10.5k citations

Hit Papers

Regulation of Cre Recombinase Activity by Mutated Estroge... 1996 2026 2006 2016 1997 1996 2014 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert Feil Germany 57 6.0k 2.5k 2.1k 1.7k 1.2k 160 10.6k
Thierry Pedrazzini Switzerland 52 5.4k 0.9× 1.8k 0.7× 2.2k 1.0× 3.4k 2.0× 1.5k 1.3× 137 12.8k
Michael I. Kotlikoff United States 56 5.6k 0.9× 2.1k 0.8× 1.8k 0.9× 2.1k 1.2× 615 0.5× 146 9.0k
T. Kendall Harden United States 61 6.6k 1.1× 1.2k 0.5× 880 0.4× 1.7k 1.0× 1.3k 1.1× 214 11.5k
Kevin R. Lynch United States 71 10.7k 1.8× 2.5k 1.0× 1.6k 0.8× 2.2k 1.3× 2.1k 1.7× 202 16.0k
Luc Leybaert Belgium 60 7.8k 1.3× 1.6k 0.6× 631 0.3× 1.7k 1.0× 697 0.6× 212 11.5k
Yoh Takuwa Japan 57 7.6k 1.3× 2.8k 1.1× 1.3k 0.6× 1.1k 0.6× 1.3k 1.1× 182 11.7k
Marco Conti United States 85 14.5k 2.4× 2.0k 0.8× 2.0k 1.0× 2.0k 1.2× 2.3k 1.9× 238 21.0k
Claes Wahlestedt United States 65 12.6k 2.1× 2.0k 0.8× 704 0.3× 2.8k 1.6× 699 0.6× 238 18.1k
Anders Oldfors Sweden 55 10.4k 1.7× 1.9k 0.7× 1.8k 0.9× 1.6k 0.9× 424 0.4× 286 13.7k
Ricardo E. Dolmetsch United States 42 9.4k 1.6× 1.1k 0.4× 1.0k 0.5× 5.0k 2.9× 1.5k 1.3× 69 14.6k

Countries citing papers authored by Robert Feil

Since Specialization
Citations

This map shows the geographic impact of Robert Feil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Feil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Feil more than expected).

Fields of papers citing papers by Robert Feil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Feil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Feil. The network helps show where Robert Feil may publish in the future.

Co-authorship network of co-authors of Robert Feil

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Feil. A scholar is included among the top collaborators of Robert Feil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Feil. Robert Feil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schmidt, Hannes, Maria T. K. Zaldivia, Michael Krämer, et al.. (2025). Single-cell analysis identifies the CNP/GC-B/cGMP axis as marker and regulator of modulated VSMCs in atherosclerosis. Nature Communications. 16(1). 429–429. 2 indexed citations
2.
Proikas‐Cezanne, Tassula, et al.. (2025). International symposium “The Different Facets of Guanine Nucleotide Signaling”. Naunyn-Schmiedeberg s Archives of Pharmacology. 398(11). 16279–16287.
3.
Hernández‐Olmos, Víctor, Wiebke Kallenborn-Gerhardt, Ruirui Lu, et al.. (2024). Inhibition of Phosphodiesterase 10A Alleviates Pain-like Behavior in Mice. Anesthesiology. 142(2). 332–348. 1 indexed citations
4.
Görlitz, Frederik, et al.. (2024). Real-time imaging of cGMP signaling shows pronounced differences between glomerular endothelial cells and podocytes. Scientific Reports. 14(1). 26099–26099. 1 indexed citations
5.
Feil, Susanne, Walter Ehrlichmann, Gerald Reischl, et al.. (2023). Noninvasive Detection of Smooth Muscle Cell-Derived Hot Spots to Study Atherosclerosis by PET/MRI in Mice. Circulation Research. 132(6). 747–750. 5 indexed citations
6.
Zhang, Yujing, Shang Fa Yang, Susanne Feil, et al.. (2022). Optogenetic manipulation of cyclic guanosine monophosphate to probe phosphodiesterase activities in megakaryocytes. Open Biology. 12(8). 220058–220058. 2 indexed citations
7.
Tikoo, Shweta, Rohit Jain, Florence Tomasetig, et al.. (2021). Amelanotic B16-F10 Melanoma Compatible with Advanced Three-Dimensional Imaging Modalities. Journal of Investigative Dermatology. 141(8). 2090–2094.e6. 5 indexed citations
8.
Schmidt, Hannes, Deborah M. Dickey, Jerid W. Robinson, et al.. (2018). Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is Essential for Bifurcation of Sensory Axons. Journal of Neuroscience. 38(45). 9768–9780. 16 indexed citations
9.
Wen, Lai, Susanne Feil, Martin Thunemann, et al.. (2018). A shear-dependent NO-cGMP-cGKI cascade in platelets acts as an auto-regulatory brake of thrombosis. Nature Communications. 9(1). 4301–4301. 34 indexed citations
11.
Thunemann, Martin, Lai Wen, Matthias Hillenbrand, et al.. (2013). Transgenic Mice for cGMP Imaging. Circulation Research. 113(4). 365–371. 61 indexed citations
12.
Franz, Christoph, Ulrike Zimmermann, Sze Chim Lee, et al.. (2013). Autonomous functions of murine thyroid hormone receptor TRα and TRβ in cochlear hair cells. Molecular and Cellular Endocrinology. 382(1). 26–37. 19 indexed citations
13.
Labat, Carlos, Mathias Mericskay, Karima Ait‐Aissa, et al.. (2013). Inactivation of Serum Response Factor Contributes To Decrease Vascular Muscular Tone and Arterial Stiffness in Mice. Circulation Research. 112(7). 1035–1045. 34 indexed citations
14.
Dankworth, Beatrice, Martin Kruse, Michael Hartmann, et al.. (2011). A cardiac pathway of cyclic GMP-independent signaling of guanylyl cyclase A, the receptor for atrial natriuretic peptide. Proceedings of the National Academy of Sciences. 108(45). 18500–18505. 35 indexed citations
15.
Fenske, Stefanie, Robert M. Mader, Stylianos Michalakis, et al.. (2011). HCN3 Contributes to the Ventricular Action Potential Waveform in the Murine Heart. Circulation Research. 109(9). 1015–1023. 53 indexed citations
16.
Weinmeister, Pascal, Robert Łukowski, Stefan Linder, et al.. (2008). Cyclic Guanosine Monophosphate-dependent Protein Kinase I Promotes Adhesion of Primary Vascular Smooth Muscle Cells. Molecular Biology of the Cell. 19(10). 4434–4441. 21 indexed citations
17.
Döring, Britta, Oksana Shynlova, Prudence Tsui, et al.. (2006). Ablation of connexin43 in uterine smooth muscle cells of the mouse causes delayed parturition. Journal of Cell Science. 119(9). 1715–1722. 88 indexed citations
18.
Feil, Robert, Susanne Feil, & Franz Hofmann. (2005). A heretical view on the role of NO and cGMP in vascular proliferative diseases. Trends in Molecular Medicine. 11(2). 71–75. 40 indexed citations
19.
Feil, Robert, et al.. (2003). Involvement of L-type calcium channels (Cav1.2) in the carbachol-induced contraction of murine urinary bladder.. mediaTUM (Technical University of Munich). 2 indexed citations
20.
Wit, Cor de, et al.. (2003). NO-, but not ACh-induced arteriolar dilations are impaired in cGMP-dependent protein kinase deficient mice. mediaTUM (Technical University of Munich). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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