John D. Lambris

62.4k total citations · 15 hit papers
570 papers, 47.8k citations indexed

About

John D. Lambris is a scholar working on Immunology, Hematology and Molecular Biology. According to data from OpenAlex, John D. Lambris has authored 570 papers receiving a total of 47.8k indexed citations (citations by other indexed papers that have themselves been cited), including 417 papers in Immunology, 130 papers in Hematology and 118 papers in Molecular Biology. Recurrent topics in John D. Lambris's work include Complement system in diseases (317 papers), Monoclonal and Polyclonal Antibodies Research (79 papers) and Blood groups and transfusion (51 papers). John D. Lambris is often cited by papers focused on Complement system in diseases (317 papers), Monoclonal and Polyclonal Antibodies Research (79 papers) and Blood groups and transfusion (51 papers). John D. Lambris collaborates with scholars based in United States, Germany and Greece. John D. Lambris's co-authors include Daniel Ricklin, George Hajishengallis, Dimitrios C. Mastellos, Maciej M. Markiewski, Kun Yang, Edimara S. Reis, Arvind Sahu, Bo Nilsson, Markus Huber‐Lang and Tom Eirik Mollnes and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

John D. Lambris

562 papers receiving 46.9k citations

Hit Papers

Complement: a key system ... 2002 2026 2010 2018 2010 2007 2011 2006 2008 500 1000 1.5k 2.0k 2.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Complement: a key system for immune surveillance and homeostasis
    2010 2.8k citations Daniel Ricklin, George Hajishengallis et al. profile →
  2. The Classical Complement Cascade Mediates CNS Synapse Elimination
    2007 2.4k citations John D. Lambris et al. profile →
  3. Low-Abundance Biofilm Species Orchestrates Inflammatory Periodontal Disease through the Commensal Microbiota and Complement
    2011 903 citations George Hajishengallis, John D. Lambris et al. profile →
  4. Generation of C5a in the absence of C3: a new complement activation pathway
    2006 776 citations Markus Huber‐Lang, J. Vidya Sarma et al. profile →
  5. Modulation of the antitumor immune response by complement
    2008 598 citations Maciej M. Markiewski, John D. Lambris et al. profile →
  6. Complement evasion by human pathogens
    2008 573 citations John D. Lambris, Daniel Ricklin et al. profile →
  7. Molecular Intercommunication between the Complement and Coagulation Systems
    2010 571 citations Bo Nilsson, John D. Lambris et al. The Journal of Immunology profile →
  8. Complement and tissue factor–enriched neutrophil extracellular traps are key drivers in COVID-19 immunothrombosis
    2020 545 citations Dimitrios C. Mastellos, John D. Lambris et al. Journal of Clinical Investigation profile →
  9. The complement system in teleosts
    2002 538 citations John D. Lambris et al. profile →
  10. Drusen complement components C3a and C5a promote choroidal neovascularization
    2006 531 citations J. Vidya Sarma, John D. Lambris et al. Proceedings of the National Academy of Sciences profile →
  11. The Role of Complement in Inflammatory Diseases From Behind the Scenes into the Spotlight
    2007 525 citations Maciej M. Markiewski, John D. Lambris American Journal Of Pathology profile →
  12. Complement in disease: a defence system turning offensive
    2016 410 citations Daniel Ricklin, Edimara S. Reis et al. profile →
  13. Innate immune responses to trauma
    2018 391 citations Markus Huber‐Lang, John D. Lambris et al. profile →
  14. Current understanding of periodontal disease pathogenesis and targets for host‐modulation therapy
    2020 299 citations George Hajishengallis, John D. Lambris et al. profile →
  15. A guide to complement biology, pathology and therapeutic opportunity
    2023 135 citations Dimitrios C. Mastellos, George Hajishengallis et al. profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
John D. Lambris 27.3k 9.8k 7.2k 5.5k 3.9k 570 47.8k
Vijay K. Kuchroo 59.0k 2.2× 13.2k 1.3× 2.7k 0.4× 5.2k 0.9× 2.6k 0.7× 426 79.1k
Arlene H. Sharpe 47.7k 1.7× 16.3k 1.7× 3.5k 0.5× 5.9k 1.1× 3.4k 0.9× 408 75.8k
Leo A. B. Joosten 27.5k 1.0× 17.0k 1.7× 2.4k 0.3× 8.5k 1.6× 8.2k 2.1× 701 52.3k
Paul Kubes 26.2k 1.0× 12.7k 1.3× 3.3k 0.5× 6.9k 1.3× 2.6k 0.7× 423 52.5k
Kouji Matsushima 25.6k 0.9× 11.8k 1.2× 2.1k 0.3× 4.2k 0.8× 1.6k 0.4× 562 49.2k
Tadamitsu Kishimoto 34.0k 1.2× 22.3k 2.3× 8.8k 1.2× 6.2k 1.1× 3.1k 0.8× 643 77.6k
Jürg Tschopp 43.3k 1.6× 52.0k 5.3× 4.1k 0.6× 11.9k 2.2× 4.1k 1.1× 283 87.3k
Klaus Ley 26.9k 1.0× 16.3k 1.7× 5.3k 0.7× 5.8k 1.1× 1.1k 0.3× 457 55.9k
Nico van Rooijen 38.1k 1.4× 21.4k 2.2× 3.6k 0.5× 13.1k 2.4× 4.9k 1.3× 838 80.5k
Bruce Beutler 38.1k 1.4× 17.2k 1.8× 2.5k 0.4× 10.8k 2.0× 3.8k 1.0× 406 64.5k

Countries citing papers authored by John D. Lambris

Since Specialization
Citations

This map shows the geographic impact of John D. Lambris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John D. Lambris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John D. Lambris more than expected).

Fields of papers citing papers by John D. Lambris

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John D. Lambris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John D. Lambris. The network helps show where John D. Lambris may publish in the future.

Co-authorship network of co-authors of John D. Lambris

This figure shows the co-authorship network connecting the top 25 collaborators of John D. Lambris. A scholar is included among the top collaborators of John D. Lambris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John D. Lambris. John D. Lambris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Husebye, Harald, Terje Espevik, Liv Ryan, et al.. (2025). The role of intracellular and cell-autonomous C3 in NF-κB priming in human macrophages. Immunobiology. 230(4). 153060–153060.
2.
Senent, Yaiza, Ana Remírez, Diana Llópiz, et al.. (2024). The C5a/C5aR1 Axis Promotes Migration of Tolerogenic Dendritic Cells to Lymph Nodes, Impairing the Anticancer Immune Response. Cancer Immunology Research. 13(3). 384–399. 6 indexed citations
3.
Gerogianni, Alexandra, Camilla Mohlin, Trent M. Woodruff, et al.. (2023). In vitro evaluation of iron oxide nanoparticle-induced thromboinflammatory response using a combined human whole blood and endothelial cell model. Frontiers in Immunology. 14. 1101387–1101387. 12 indexed citations
4.
Lamers, Christina, Daniel Ricklin, & John D. Lambris. (2023). Complement‐targeted therapeutics: An emerging field enabled by academic drug discovery. American Journal of Hematology. 98(S4). S82–S89. 18 indexed citations
5.
Wang, Hui, et al.. (2022). Complement Is Required for Microbe-Driven Induction of Th17 and Periodontitis. The Journal of Immunology. 209(7). 1370–1378. 13 indexed citations
6.
Lam, Lian, John P. Reilly, Ann H. Rux, et al.. (2021). Erythrocytes identify complement activation in patients with COVID-19. American Journal of Physiology-Lung Cellular and Molecular Physiology. 321(2). L485–L489. 43 indexed citations
7.
Christiansen, Dorte, Hilde Fure, Judith Krey Ludviksen, et al.. (2021). Air Bubbles Activate Complement and Trigger Hemostasis and C3-Dependent Cytokine Release Ex Vivo in Human Whole Blood. The Journal of Immunology. 207(11). 2828–2840. 10 indexed citations
8.
Khandelwal, Sanjay, Lubica Rauova, Amrita Sarkar, et al.. (2021). Complement mediates binding and procoagulant effects of ultralarge HIT immune complexes. Blood. 138(21). 2106–2116. 24 indexed citations
9.
Schmitz, Robin, Zachary W. Fitch, Paul M. Schroder, et al.. (2021). C3 complement inhibition prevents antibody-mediated rejection and prolongs renal allograft survival in sensitized non-human primates. Nature Communications. 12(1). 5456–5456. 38 indexed citations
10.
Steinkjer, Bjørg, Bente Halvorsen, Mona Skjelland, et al.. (2019). Cholesterol Crystals Induce Coagulation Activation through Complement-Dependent Expression of Monocytic Tissue Factor. The Journal of Immunology. 203(4). 853–863. 27 indexed citations
11.
Mödinger, Yvonne, Anna E. Rapp, Verena Fischer, et al.. (2018). Reduced Terminal Complement Complex Formation in Mice Manifests in Low Bone Mass and Impaired Fracture Healing. American Journal Of Pathology. 189(1). 147–161. 11 indexed citations
12.
Kwak, Jeff, Jennifer Laskowski, Howard Li, et al.. (2017). Complement Activation via a C3a Receptor Pathway Alters CD4+ T Lymphocytes and Mediates Lung Cancer Progression. Cancer Research. 78(1). 143–156. 103 indexed citations
13.
Wang, Hongbin, Daniel Ricklin, & John D. Lambris. (2017). Complement-activation fragment C4a mediates effector functions by binding as untethered agonist to protease-activated receptors 1 and 4. Proceedings of the National Academy of Sciences. 114(41). 10948–10953. 84 indexed citations
14.
Reis, Edimara S., et al.. (2016). High-Fat Diet-Induced Complement Activation Mediates Intestinal Inflammation and Neoplasia, Independent of Obesity. Molecular Cancer Research. 14(10). 953–965. 34 indexed citations
15.
Forneris, Federico, Jin Wu, Daniel Ricklin, et al.. (2016). Regulators of complement activity mediate inhibitory mechanisms through a common C3b‐binding mode. The EMBO Journal. 35(10). 1133–1149. 88 indexed citations
16.
Mastellos, Dimitrios C., Despina Yancopoulou, Petros Kokkinos, et al.. (2015). Compstatin: a C3‐targeted complement inhibitor reaching its prime for bedside intervention. European Journal of Clinical Investigation. 45(4). 423–440. 170 indexed citations
17.
Miwa, Takashi, Brendan Hilliard, Youhai Chen, et al.. (2005). The complement inhibitory protein DAF (CD55) suppresses T cell immunity in vivo. The Journal of Experimental Medicine. 201(4). 567–577. 198 indexed citations
18.
Park, Jung‐Won, Christian Taube, Anthony Joetham, et al.. (2004). Complement Activation Is Critical to Airway Hyperresponsiveness after Acute Ozone Exposure. American Journal of Respiratory and Critical Care Medicine. 169(6). 726–732. 50 indexed citations
19.
Riedemann, Niels C., Thomas A. Neff, Ren-Feng Guo, et al.. (2003). Protective Effects of IL-6 Blockade in Sepsis Are Linked to Reduced C5a Receptor Expression. The Journal of Immunology. 170(1). 503–507. 256 indexed citations
20.
Riedemann, Niels C., Renfeng Guo, Thomas A. Neff, et al.. (2002). Increased C5a receptor expression in sepsis. Journal of Clinical Investigation. 110(1). 101–108. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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