Edimara S. Reis
- Immunology top 0.5%
- Complement system in diseases 48
- Immunotherapy and Immune Responses 6
- Nephrology top 1%
- Renal Diseases and Glomerulopathies 12
- Hematology top 1%
- Blood groups and transfusion 11
- Platelet Disorders and Treatments 5
- Transplantation top 2%
- Periodontics top 1%
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- Erythrocyte Function and Pathophysiology 5
- Adenosine and Purinergic Signaling 4
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- Coagulation, Bradykinin, Polyphosphates, and Angioedema 4
- Co-authors
- John D. LambrisDaniel RicklinDimitrios C. MastellosGeorge HajishengallisJörg KöhlRahasson R. AgerAndrea J. TennerAndreas Klos
- Cited by
- ImmunologyNephrologyHematology
- Partner nations
- United StatesGreeceBrazil
In The Last Decade
Edimara S. Reis
59 papers receiving 4.4k citations
Hit Papers
Peers
Comparison fields: 5 of 117
- Immunology 2.9k
- Nephrology 667
- Hematology 740
- Transplantation 133
- Periodontics 224
Countries citing papers authored by Edimara S. Reis
This map shows the geographic impact of Edimara S. Reis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edimara S. Reis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edimara S. Reis more than expected).
Fields of papers citing papers by Edimara S. Reis
This network shows the impact of papers produced by Edimara S. Reis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edimara S. Reis. The network helps show where Edimara S. Reis may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Edimara S. Reis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 0 | |
| 2 | 2021 | 38 | |
| 3 | 2021 | 51 | |
| 4 | 2019 | 17 | |
| 5 | 2018 | 86 | |
| 6 | 2017 | 31 | |
| 7 | 2017 | 350 | |
| 8 | 2017 | 310 | |
| 9 | 2016 | 34 | |
| 10 | 2016 | 23 | |
| 11 | 2016 | 14 | |
| 12 | 2016 | 3 | |
| 13 | 2015 | 47 | |
| 14 | 2014 | 50 | |
| 15 | 2013 | 76 | |
| 16 | 2012 | 46 | |
| 17 | 2009 | 106 | |
| 18 | 2009 | 11 | |
| 19 | 2007 | 24 | |
| 20 | A stop codon in exon 13 causes the complete lack of human complement component C3 deficiency | 2001 | 1 |
About Edimara S. Reis
Edimara S. Reis is a scholar working on Immunology, Nephrology and Hematology, having authored 61 papers that have together received 4.5k indexed citations. Recurring topics across this work include Complement system in diseases (48 papers), Renal Diseases and Glomerulopathies (12 papers), Blood groups and transfusion (11 papers), Immunotherapy and Immune Responses (6 papers), Erythrocyte Function and Pathophysiology (5 papers), Platelet Disorders and Treatments (5 papers), Adenosine and Purinergic Signaling (4 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (4 papers). The work is most often cited by research in Immunology (2.9k citations), Nephrology (667 citations) and Hematology (740 citations). Edimara S. Reis has collaborated with scholars based in United States, Greece and Brazil. Frequent co-authors include John D. Lambris, Daniel Ricklin, Dimitrios C. Mastellos, George Hajishengallis, Jörg Köhl, Rahasson R. Ager, Andrea J. Tenner, Andreas Klos, Lourdes Isaac and Piet Gros. Their work appears in journals such as Circulation, Nature Communications and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.