Hilde Fure

1.5k total citations
38 papers, 1.2k citations indexed

About

Hilde Fure is a scholar working on Immunology, Hematology and Epidemiology. According to data from OpenAlex, Hilde Fure has authored 38 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Immunology, 15 papers in Hematology and 9 papers in Epidemiology. Recurrent topics in Hilde Fure's work include Complement system in diseases (17 papers), Blood Coagulation and Thrombosis Mechanisms (13 papers) and Immune Response and Inflammation (8 papers). Hilde Fure is often cited by papers focused on Complement system in diseases (17 papers), Blood Coagulation and Thrombosis Mechanisms (13 papers) and Immune Response and Inflammation (8 papers). Hilde Fure collaborates with scholars based in Norway, United States and Australia. Hilde Fure's co-authors include Tom Eirik Mollnes, Ole‐Lars Brekke, Dorte Christiansen, Michael Fung, Erik Waage Nielsen, John D. Lambris, Knut Tore Lappegård, Grethe Bergseth, Vibeke Videm and Jörg Köhl and has published in prestigious journals such as Blood, The Journal of Immunology and PLoS ONE.

In The Last Decade

Hilde Fure

38 papers receiving 1.2k citations

Peers

Hilde Fure
Dorte Christiansen Norway
Yaseelan Palarasah Denmark
Anne Pharo Norway
Scott M. Drouin United States
Umme Amara Germany
Arnab Ghosh United States
Maria C. Faber‐Krol Netherlands
Sylvie Amu Sweden
J McNicholl United States
Julie K. Lindstad Norway
Dorte Christiansen Norway
Hilde Fure
Citations per year, relative to Hilde Fure Hilde Fure (= 1×) peers Dorte Christiansen

Countries citing papers authored by Hilde Fure

Since Specialization
Citations

This map shows the geographic impact of Hilde Fure's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hilde Fure with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hilde Fure more than expected).

Fields of papers citing papers by Hilde Fure

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hilde Fure. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hilde Fure. The network helps show where Hilde Fure may publish in the future.

Co-authorship network of co-authors of Hilde Fure

This figure shows the co-authorship network connecting the top 25 collaborators of Hilde Fure. A scholar is included among the top collaborators of Hilde Fure based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hilde Fure. Hilde Fure is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Emblem, Åse, Erik Knutsen, Tor Erik Jørgensen, et al.. (2022). Blood Transcriptome Analysis of Septic Patients Reveals a Long Non-Coding Alu-RNA in the Complement C5a Receptor 1 Gene. Non-Coding RNA. 8(2). 24–24. 1 indexed citations
2.
Ludviksen, Judith Krey, Dorte Christiansen, Hilde Fure, et al.. (2022). Venous Air Embolism Activates Complement C3 Without Corresponding C5 Activation and Trigger Thromboinflammation in Pigs. Frontiers in Immunology. 13. 839632–839632. 6 indexed citations
3.
Christiansen, Dorte, Hilde Fure, Judith Krey Ludviksen, et al.. (2021). Air Bubbles Activate Complement and Trigger Hemostasis and C3-Dependent Cytokine Release Ex Vivo in Human Whole Blood. The Journal of Immunology. 207(11). 2828–2840. 10 indexed citations
4.
Lau, Corinna, Grethe Bergseth, Algirdas Grevys, et al.. (2019). NHDL, a recombinant V L /V H hybrid antibody control for IgG2/4 antibodies. mAbs. 12(1). 1686319–1686319. 3 indexed citations
5.
Pischke, Søren Erik, Harald Thidemann Johansen, Hilde Fure, et al.. (2019). Sepsis causes right ventricular myocardial inflammation independent of pulmonary hypertension in a porcine sepsis model. PLoS ONE. 14(6). e0218624–e0218624. 14 indexed citations
6.
Brekke, Ole‐Lars, Dorte Christiansen, Aymric Kisserli, et al.. (2019). Key role of the number of complement receptor 1 on erythrocytes for binding of Escherichia coli to erythrocytes and for leukocyte phagocytosis and oxidative burst in human whole blood. Molecular Immunology. 114. 139–148. 4 indexed citations
7.
Christiansen, Dorte, Hilde Fure, Judith Krey Ludviksen, et al.. (2018). Staphylococcus aureus‐induced complement activation promotes tissue factor‐mediated coagulation. Journal of Thrombosis and Haemostasis. 16(5). 905–918. 18 indexed citations
8.
9.
Fure, Hilde, Bjørg Steinkjer, Liv Ryan, et al.. (2017). Alginate microbeads are coagulation compatible, while alginate microcapsules activate coagulation secondary to complement or directly through FXII. Acta Biomaterialia. 58. 158–167. 23 indexed citations
10.
Hellerud, Bernt Christian, Knut Dybwik, Søren Erik Pischke, et al.. (2017). Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis. Journal of Intensive Care. 5(1). 21–21. 19 indexed citations
11.
Seip, Knut Fredrik, Anders Hovland, Knut Dybwik, et al.. (2016). Bradykinin-Induced Shock Increase Exhaled Nitric Oxide, Complement Activation and Cytokine Production in Pigs. BIBSYS Brage (BIBSYS (Norway)). 4(2). 2 indexed citations
12.
13.
Lau, Corinna, Ole Kristoffer Olstad, Marit Holden, et al.. (2015). Gene expression profiling of Gram-negative bacteria-induced inflammation in human whole blood: The role of complement and CD14-mediated innate immune response. Genomics Data. 5. 176–183. 6 indexed citations
14.
Nestvold, Torunn Kristin, et al.. (2014). Lifestyle Changes Followed by Bariatric Surgery Lower Inflammatory Markers and the Cardiovascular Risk Factors C3 and C4. Metabolic Syndrome and Related Disorders. 13(1). 29–35. 20 indexed citations
15.
Brekke, Ole‐Lars, Dorte Christiansen, Hilde Fure, et al.. (2012). The Effects of Selective Complement and CD14 Inhibition on the E. coli-Induced Tissue Factor mRNA Upregulation, Monocyte Tissue Factor Expression, and Tissue Factor Functional Activity in Human Whole Blood. Advances in experimental medicine and biology. 735. 123–136. 21 indexed citations
16.
Brekke, Ole‐Lars, Bernt Christian Hellerud, Dorte Christiansen, et al.. (2011). Neisseria meningitidis and Escherichia coli are protected from leukocyte phagocytosis by binding to erythrocyte complement receptor 1 in human blood. Molecular Immunology. 48(15-16). 2159–2169. 23 indexed citations
17.
Brekke, Ole‐Lars, Dorte Christiansen, Hilde Fure, et al.. (2008). Combined inhibition of complement and CD14 abolish E. coli-induced cytokine-, chemokine- and growth factor-synthesis in human whole blood. Molecular Immunology. 45(14). 3804–3813. 53 indexed citations
18.
Nielsen, Erik Waage, Hilde Fure, Ole‐Lars Brekke, et al.. (2006). Effect of supraphysiologic levels of C1-inhibitor on the classical, lectin and alternative pathways of complement. Molecular Immunology. 44(8). 1819–1826. 44 indexed citations
19.
Fure, Hilde, Erik Waage Nielsen, C. Erik Hack, & Tom Eirik Mollnes. (1997). A Neoepitope‐Based Enzyme Immunoassay for Quantification of C1‐Inhibitor in Complex with C1r and C1s. Scandinavian Journal of Immunology. 46(6). 553–557. 46 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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