Michael Karin
- Cancer Research top 0.01%
- NF-κB Signaling Pathways 201
- Immunology top 0.01%
- Immune Response and Inflammation 119
- interferon and immune responses 48
- Oncology top 0.01%
- Cytokine Signaling Pathways and Interactions 101
- Molecular Biology top 0.01%
- Melanoma and MAPK Pathways 58
- Protein Kinase Regulation and GTPase Signaling 50
- Ubiquitin and proteasome pathways 41
- Cell Biology top 0.01%
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- Trace Elements in Health 49
- Co-authors
- Florian R. GretenSergei I. GrivennikovLufen ChangPeter AngelYinon Ben‐NeriahEbrahim ZandiAnning LinPeter J. Barnes
- Cited by
- Cancer ResearchImmunologyOncology
- Journals
- Proceedings of the National Academy of Sciences (54 papers)Cell (50 papers)Molecular and Cellular Biology (48 papers)
- Partner nations
- United StatesJapanGermany
In The Last Decade
Michael Karin
648 papers receiving 208.8k citations
Hit Papers
Peers
Comparison fields: 5 of 203
- Cancer Research 56.6k
- Immunology 60.2k
- Oncology 45.8k
- Molecular Biology 113.1k
- Cell Biology 13.5k
Countries citing papers authored by Michael Karin
This map shows the geographic impact of Michael Karin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Karin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Karin more than expected).
Fields of papers citing papers by Michael Karin
This network shows the impact of papers produced by Michael Karin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Karin. The network helps show where Michael Karin may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Michael Karin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 10 | |
| 2 | 2023 | 1 | |
| 3 | An AMPK–caspase-6 axis controls liver damage in nonalcoholic steatohepatitisbreakdown → | 2020 | 236 |
| 4 | 2019 | 22 | |
| 5 | 2018 | 75 | |
| 6 | 2014 | 174 | |
| 7 | 2014 | 36 | |
| 8 | 2014 | 95 | |
| 9 | 2011 | 30 | |
| 10 | Macrophage Expression of Hypoxia-Inducible Factor-1α Suppresses T-Cell Function and Promotes Tumor Progressionbreakdown → | 2010 | 523 |
| 11 | 2010 | 476 | |
| 12 | 2010 | 264 | |
| 13 | 2009 | 67 | |
| 14 | 2008 | 179 | |
| 15 | 2008 | 157 | |
| 16 | 2008 | 79 | |
| 17 | 2007 | 105 | |
| 18 | Gender Disparity in Liver Cancer Due to Sex Differences in MyD88-Dependent IL-6 Productionbreakdown → | 2007 | 1487 |
| 19 | Nod2 Mutation in Crohn's Disease Potentiates NF-κB Activity and IL-1ß Processingbreakdown → | 2005 | 599 |
| 20 | 2004 | 336 |
About Michael Karin
Michael Karin is a scholar working on Cancer Research, Immunology and Oncology, having authored 652 papers that have together received 212.6k indexed citations. Recurring topics across this work include NF-κB Signaling Pathways (201 papers), Immune Response and Inflammation (119 papers), Cytokine Signaling Pathways and Interactions (101 papers), Melanoma and MAPK Pathways (58 papers), Protein Kinase Regulation and GTPase Signaling (50 papers), Trace Elements in Health (49 papers), interferon and immune responses (48 papers) and Ubiquitin and proteasome pathways (41 papers). The work is most often cited by research in Cancer Research (56.6k citations), Immunology (60.2k citations) and Oncology (45.8k citations). Michael Karin has collaborated with scholars based in United States, Japan and Germany. Frequent co-authors include Florian R. Greten, Sergei I. Grivennikov, Lufen Chang, Peter Angel, Yinon Ben‐Neriah, Ebrahim Zandi, Anning Lin, Peter J. Barnes, Eitan Shaulian and Tod Smeal. Their work appears in journals such as Proceedings of the National Academy of Sciences, Cell, Molecular and Cellular Biology, Nature and Science.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.