François Mercier
- Molecular Biology top 10%
- Hematology top 1%
- Immunology top 5%
- Cancer Research top 5%
- Genetics top 5%
- Co-authors
- David T. ScaddenChristine RaguRuslan I. SadreyevEda YildirimJeannie T. LeeJames E. KirbyDiane E. BrownYoumna Kfoury
- Topics
- Acute Myeloid Leukemia Research (16 papers)Hematopoietic Stem Cell Transplantation (10 papers)CRISPR and Genetic Engineering (7 papers)
- Cited by
- HematologyGeneticsCancer Research
- Partner nations
- CanadaUnited StatesGermany
In The Last Decade
François Mercier
48 papers receiving 2.1k citations
Hit Papers
Peers
Comparison fields: 5 of 100
- Molecular Biology 1.1k
- Hematology 642
- Immunology 533
- Cancer Research 428
- Genetics 325
Countries citing papers authored by François Mercier
This map shows the geographic impact of François Mercier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by François Mercier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites François Mercier more than expected).
Fields of papers citing papers by François Mercier
This network shows the impact of papers produced by François Mercier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by François Mercier. The network helps show where François Mercier may publish in the future.
Co-authorship network of co-authors of François Mercier
This figure shows the co-authorship network connecting the top 25 collaborators of François Mercier. A scholar is included among the top collaborators of François Mercier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with François Mercier. François Mercier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 8 | |
| 4 | 1 | |
| 5 | 3 | |
| 6 | 7 | |
| 7 | 1 | |
| 8 | 1 | |
| 9 | 27 | |
| 10 | 4 | |
| 11 | 32 | |
| 12 | 13 | |
| 13 | A Cellular Taxonomy of the Bone Marrow Stroma in Homeostasis and Leukemiabreakdown → | 558 |
| 14 | 24 | |
| 15 | 2 | |
| 16 | 88 | |
| 17 | 174 | |
| 18 | 379 | |
| 19 | Akt/foxo signaling pathway enforces the differentiation blockade in myeloid leukemias | 1 |
| 20 | 217 |
About François Mercier
François Mercier is a scholar working on Hematology, Immunology and Genetics, having authored 51 papers that have together received 2.1k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (16 papers), Hematopoietic Stem Cell Transplantation (10 papers) and CRISPR and Genetic Engineering (7 papers). The work is most often cited by research in Hematology (642 citations), Genetics (325 citations) and Cancer Research (428 citations). François Mercier has collaborated with scholars based in Canada, United States and Germany. Frequent co-authors include David T. Scadden, Christine Ragu, Ruslan I. Sadreyev, Eda Yildirim, Jeannie T. Lee, James E. Kirby, Diane E. Brown, Youmna Kfoury, Ninib Baryawno and Ani Papazian. Their work appears in journals such as Cell, Journal of Clinical Investigation and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.