Raquel Aloyz
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 0.5%
- Developmental Neuroscience top 0.5%
- Oncology top 5%
- Cancer Research top 5%
- Co-authors
- Freda D. MillerShernaz X. BamjiLawrence PanasciCarrie G. CausingJ. Paul FawcettMarta MajdanChristine D. PozniakLilian Amrein
- Topics
- DNA Repair Mechanisms (21 papers)Chronic Lymphocytic Leukemia Research (21 papers)Neuropeptides and Animal Physiology (10 papers)
- Partner nations
- CanadaUnited StatesArgentina
In The Last Decade
Raquel Aloyz
71 papers receiving 4.0k citations
Hit Papers
Peers
Comparison fields: 5 of 114
- Molecular Biology 2.1k
- Cellular and Molecular Neuroscience 1.9k
- Developmental Neuroscience 892
- Oncology 739
- Cancer Research 397
Countries citing papers authored by Raquel Aloyz
This map shows the geographic impact of Raquel Aloyz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raquel Aloyz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raquel Aloyz more than expected).
Fields of papers citing papers by Raquel Aloyz
This network shows the impact of papers produced by Raquel Aloyz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raquel Aloyz. The network helps show where Raquel Aloyz may publish in the future.
Co-authorship network of co-authors of Raquel Aloyz
This figure shows the co-authorship network connecting the top 25 collaborators of Raquel Aloyz. A scholar is included among the top collaborators of Raquel Aloyz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raquel Aloyz. Raquel Aloyz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 6 | |
| 4 | 9 | |
| 5 | 27 | |
| 6 | 24 | |
| 7 | 36 | |
| 8 | 18 | |
| 9 | 49 | |
| 10 | 18 | |
| 11 | 74 | |
| 12 | 13 | |
| 13 | 4 | |
| 14 | 13 | |
| 15 | 49 | |
| 16 | Dasatinib sensitizes chronic lymphocytic leukemia lymphocytes to chemotherapeutic agents | 1 |
| 17 | 12 | |
| 18 | 57 | |
| 19 | 43 | |
| 20 | 2 |
About Raquel Aloyz
Raquel Aloyz is a scholar working on Genetics, Developmental Neuroscience and Cellular and Molecular Neuroscience, having authored 74 papers that have together received 4.1k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (21 papers), Chronic Lymphocytic Leukemia Research (21 papers) and Neuropeptides and Animal Physiology (10 papers). The work is most often cited by research in Developmental Neuroscience (892 citations), Cellular and Molecular Neuroscience (1.9k citations) and Biological Psychiatry (158 citations). Raquel Aloyz has collaborated with scholars based in Canada, United States and Argentina. Frequent co-authors include Freda D. Miller, Shernaz X. Bamji, Lawrence Panasci, Carrie G. Causing, J. Paul Fawcett, Marta Majdan, Christine D. Pozniak, Lilian Amrein, Jean G. Toma and Daniel J. Belliveau. Their work appears in journals such as Journal of Biological Chemistry, Neuron and Journal of Clinical Oncology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.