Enrique Samper
- Aging top 0.2%
- Physiology top 0.5%
- Telomeres, Telomerase, and Senescence 16
- Molecular Biology top 1%
- DNA Repair Mechanisms 11
- Mitochondrial Function and Pathology 4
- RNA Interference and Gene Delivery 4
- CRISPR and Genetic Engineering 4
- Genetics top 2%
- Mesenchymal stem cell research 3
- Developmental Neuroscience top 5%
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- Cancer Research and Treatments 3
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- Cancer-related Molecular Pathways 3
- Co-authors
- Marı́a A. BlascoM. Prakash HandePeter M. LansdorpHan‐Woong LeeCarol W. GreiderRonald A. DePinhoSimon MelovJuana M. Flores
- Cited by
- AgingPhysiologyMolecular Biology
- Journals
- PLoS Genetics (3 papers)The Journal of Cell Biology (2 papers)Free Radical Biology and Medicine (2 papers)
- Partner nations
- SpainUnited StatesCanada
In The Last Decade
Enrique Samper
32 papers receiving 6.1k citations
Hit Papers
Peers
Comparison fields: 5 of 117
- Aging 854
- Physiology 3.4k
- Molecular Biology 4.1k
- Genetics 578
- Developmental Neuroscience 173
Countries citing papers authored by Enrique Samper
This map shows the geographic impact of Enrique Samper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Enrique Samper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Enrique Samper more than expected).
Fields of papers citing papers by Enrique Samper
This network shows the impact of papers produced by Enrique Samper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Enrique Samper. The network helps show where Enrique Samper may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Enrique Samper, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 9 | |
| 2 | 2017 | 2 | |
| 3 | Impaired cell reprogramming in non-homologous end joining deficient cells | 2013 | 1 |
| 4 | 2013 | 14 | |
| 5 | 2013 | 184 | |
| 6 | 2012 | 26 | |
| 7 | 2011 | 226 | |
| 8 | 2009 | 31 | |
| 9 | 2008 | 37 | |
| 10 | 2007 | 45 | |
| 11 | 2006 | 63 | |
| 12 | 2005 | 10 | |
| 13 | 2004 | 65 | |
| 14 | 2003 | 87 | |
| 15 | 2001 | 214 | |
| 16 | 2000 | 284 | |
| 17 | 2000 | 264 | |
| 18 | 1999 | 287 | |
| 19 | Role of the NF-ATc transcription factor in morphogenesis of cardiac valves and septumbreakdown → | 1998 | 517 |
| 20 | Telomere Shortening and Tumor Formation by Mouse Cells Lacking Telomerase RNAbreakdown → | 1997 | 1728 |
About Enrique Samper
Enrique Samper is a scholar working on Neuropsychology and Physiological Psychology, Physiology, Aging, Biotechnology and Molecular Biology, having authored 32 papers that have together received 6.2k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (16 papers), DNA Repair Mechanisms (11 papers), Mitochondrial Function and Pathology (4 papers), RNA Interference and Gene Delivery (4 papers), CRISPR and Genetic Engineering (4 papers), Cancer Research and Treatments (3 papers), Mesenchymal stem cell research (3 papers) and Cancer-related Molecular Pathways (3 papers). The work is most often cited by research in Aging (854 citations), Physiology (3.4k citations), Molecular Biology (4.1k citations), Genetics (578 citations) and Developmental Neuroscience (173 citations). Enrique Samper has collaborated with scholars based in Spain, United States and Canada. Frequent co-authors include Marı́a A. Blasco, M. Prakash Hande, Peter M. Lansdorp, Han‐Woong Lee, Carol W. Greider, Ronald A. DePinho, Simon Melov, Juana M. Flores, Ana Krtolica and Joshua C. Goldstein. Their work appears in journals such as PLoS Genetics, The Journal of Cell Biology, Free Radical Biology and Medicine, EMBO Reports and Aging Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.