Ellen Freed
- Cell Biology top 2%
- Microtubule and mitosis dynamics 2
- Oncology top 5%
- Immunology and Allergy top 5%
- Molecular Biology top 5%
- Ubiquitin and proteasome pathways 3
- Protein Kinase Regulation and GTPase Signaling 2
- DNA Repair Mechanisms 1
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- Chronic Lymphocytic Leukemia Research 2
- Digestive system and related health 2
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- Eosinophilic Disorders and Syndromes 2
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- Chronic Myeloid Leukemia Treatments 2
- Co-authors
- Peter K. JacksonJerry Y. HsuJulie D.R. ReimannBrett K. KaiserAdam G. EldridgeFrank McCormickMarc SymonsRosamaria Ruggieri
- Partner nations
- United StatesAustriaRussia
In The Last Decade
Ellen Freed
14 papers receiving 2.1k citations
Hit Papers
Peers
Comparison fields: 5 of 85
- Cell Biology 543
- Oncology 659
- Immunology and Allergy 143
- Molecular Biology 1.5k
- Pathology and Forensic Medicine 260
Countries citing papers authored by Ellen Freed
This map shows the geographic impact of Ellen Freed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ellen Freed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ellen Freed more than expected).
Fields of papers citing papers by Ellen Freed
This network shows the impact of papers produced by Ellen Freed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ellen Freed. The network helps show where Ellen Freed may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Ellen Freed, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 25 | |
| 2 | 2010 | 59 | |
| 3 | 2009 | 240 | |
| 4 | 2009 | 147 | |
| 5 | 2008 | 79 | |
| 6 | 2001 | 313 | |
| 7 | The lore of the RINGs: substrate recognition and catalysis by ubiquitin ligasesbreakdown → | 2000 | 532 |
| 8 | 1999 | 172 | |
| 9 | 1994 | 357 | |
| 10 | Platelet-derived growth factor-induced p21ras-mediated signaling is independent of platelet-derived growth factor receptor interaction with GTPase-activating protein or phosphatidylinositol-3-kinase. | 1994 | 12 |
| 11 | 1992 | 2 | |
| 12 | 1992 | 11 | |
| 13 | 1989 | 151 | |
| 14 | 1988 | 29 |
About Ellen Freed
Ellen Freed is a scholar working on Genetics, Cell Biology and Hematology, having authored 14 papers that have together received 2.1k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (3 papers), Chronic Lymphocytic Leukemia Research (2 papers), Eosinophilic Disorders and Syndromes (2 papers), Microtubule and mitosis dynamics (2 papers), Chronic Myeloid Leukemia Treatments (2 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Digestive system and related health (2 papers) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Cell Biology (543 citations), Oncology (659 citations) and Immunology and Allergy (143 citations). Ellen Freed has collaborated with scholars based in United States, Austria and Russia. Frequent co-authors include Peter K. Jackson, Jerry Y. Hsu, Julie D.R. Reimann, Brett K. Kaiser, Adam G. Eldridge, Frank McCormick, Marc Symons, Rosamaria Ruggieri, Susan G. Macdonald and Edgar R. Kramer. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.