Emma T. Bowden
- Molecular Biology top 10%
- Cell Biology top 2%
- Oncology top 10%
- Immunology and Allergy top 2%
- Cancer Research top 5%
- Co-authors
- Susette C. MuellerAnton WellsteinGerald E. StoicaPeter J. CoopmanD. A. ThomasRobert I. GlazerAnna T. RiegelAngera H. Kuo
- Topics
- Fibroblast Growth Factor Research (5 papers)Cell Adhesion Molecules Research (5 papers)Proteoglycans and glycosaminoglycans research (5 papers)
- Partner nations
- United StatesFranceCanada
In The Last Decade
Emma T. Bowden
22 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 92
- Molecular Biology 1.1k
- Cell Biology 619
- Oncology 407
- Immunology and Allergy 330
- Cancer Research 319
Countries citing papers authored by Emma T. Bowden
This map shows the geographic impact of Emma T. Bowden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emma T. Bowden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emma T. Bowden more than expected).
Fields of papers citing papers by Emma T. Bowden
This network shows the impact of papers produced by Emma T. Bowden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emma T. Bowden. The network helps show where Emma T. Bowden may publish in the future.
Co-authorship network of co-authors of Emma T. Bowden
This figure shows the co-authorship network connecting the top 25 collaborators of Emma T. Bowden. A scholar is included among the top collaborators of Emma T. Bowden based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emma T. Bowden. Emma T. Bowden is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 5 | |
| 3 | 0 | |
| 4 | 2 | |
| 5 | 19 | |
| 6 | 187 | |
| 7 | 152 | |
| 8 | 39 | |
| 9 | 19 | |
| 10 | 33 | |
| 11 | 37 | |
| 12 | 0 | |
| 13 | 85 | |
| 14 | 271 | |
| 15 | 70 | |
| 16 | 112 | |
| 17 | 3 | |
| 18 | 271 | |
| 19 | 307 | |
| 20 | Tyrosine phosphorylation mediates ConA-induced membrane type 1-matrix metalloproteinase expression and matrix metalloproteinase-2 activation in MDA-MB-231 human breast carcinoma cells. | 17 |
About Emma T. Bowden
Emma T. Bowden is a scholar working on Immunology and Allergy, Cell Biology and Cancer Research, having authored 25 papers that have together received 1.8k indexed citations. Recurring topics across this work include Fibroblast Growth Factor Research (5 papers), Cell Adhesion Molecules Research (5 papers) and Proteoglycans and glycosaminoglycans research (5 papers). The work is most often cited by research in Immunology and Allergy (330 citations), Cell Biology (619 citations) and Cancer Research (319 citations). Emma T. Bowden has collaborated with scholars based in United States, France and Canada. Frequent co-authors include Susette C. Mueller, Anton Wellstein, Gerald E. Stoica, Peter J. Coopman, D. A. Thomas, Robert I. Glazer, Anna T. Riegel, Angera H. Kuo, Peter D. Burbelo and Michael Tri H.. Their work appears in journals such as Nature, Journal of Biological Chemistry and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.