W. Michael Kavanaugh

2.7k total citations · 1 hit paper
29 papers, 2.3k citations indexed

About

W. Michael Kavanaugh is a scholar working on Molecular Biology, Radiology, Nuclear Medicine and Imaging and Oncology. According to data from OpenAlex, W. Michael Kavanaugh has authored 29 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 13 papers in Radiology, Nuclear Medicine and Imaging and 11 papers in Oncology. Recurrent topics in W. Michael Kavanaugh's work include Monoclonal and Polyclonal Antibodies Research (13 papers), Protein Kinase Regulation and GTPase Signaling (8 papers) and Cell Adhesion Molecules Research (6 papers). W. Michael Kavanaugh is often cited by papers focused on Monoclonal and Polyclonal Antibodies Research (13 papers), Protein Kinase Regulation and GTPase Signaling (8 papers) and Cell Adhesion Molecules Research (6 papers). W. Michael Kavanaugh collaborates with scholars based in United States, Japan and United Kingdom. W. Michael Kavanaugh's co-authors include Lewis T. Williams, Jaime A. Escobedo, L T Williams, Victor A. Fried, Sutip Navankasattusas, Dale Milfay, Anke Klippel, Gerald Apell, Christoph W. Turck and Christoph Reinhard and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

W. Michael Kavanaugh

28 papers receiving 2.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

cDNA cloning of a Novel 85 kd protein that has SH2 domains and regulates binding of PI3-kinase to the PDGF β-receptor

1991 528 citations Jaime A. Escobedo, Sutip Navankasattusas et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
W. Michael Kavanaugh United States	17	1.7k	489	339	336	272	29	2.3k
J A Escobedo United States	20	1.8k 1.1×	455 0.9×	361 1.1×	358 1.1×	265 1.0×	28	2.6k
I. Hiles United Kingdom	12	2.1k 1.2×	482 1.0×	399 1.2×	585 1.7×	221 0.8×	12	2.6k
Bao Hoang United States	26	1.2k 0.7×	534 1.1×	294 0.9×	242 0.7×	200 0.7×	40	2.0k
Gibbes R. Johnson United States	26	1.2k 0.7×	737 1.5×	307 0.9×	190 0.6×	240 0.9×	42	1.9k
Neetu Gupta France	19	1.7k 1.0×	298 0.6×	504 1.5×	471 1.4×	103 0.4×	33	2.6k
Joseph P. Gardner United States	6	1.2k 0.7×	483 1.0×	616 1.8×	293 0.9×	123 0.5×	8	2.1k
Bruce E. Elliott Canada	26	1.1k 0.7×	584 1.2×	272 0.8×	298 0.9×	102 0.4×	62	1.9k
Roy Katso United Kingdom	13	1.9k 1.1×	409 0.8×	375 1.1×	466 1.4×	69 0.3×	15	2.6k
A MacAuley United States	14	1.1k 0.6×	327 0.7×	285 0.8×	336 1.0×	153 0.6×	19	1.6k
Scott R. Frank United States	17	2.5k 1.4×	622 1.3×	244 0.7×	646 1.9×	105 0.4×	20	3.0k

Countries citing papers authored by W. Michael Kavanaugh

Since Specialization

Citations

This map shows the geographic impact of W. Michael Kavanaugh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Michael Kavanaugh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Michael Kavanaugh more than expected).

Fields of papers citing papers by W. Michael Kavanaugh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Michael Kavanaugh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Michael Kavanaugh. The network helps show where W. Michael Kavanaugh may publish in the future.

Co-authorship network of co-authors of W. Michael Kavanaugh

This figure shows the co-authorship network connecting the top 25 collaborators of W. Michael Kavanaugh. A scholar is included among the top collaborators of W. Michael Kavanaugh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. Michael Kavanaugh. W. Michael Kavanaugh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Boustany, Leila M., Sherry L. LaPorte, Joel Shen, et al.. (2022). A Probody T Cell–Engaging Bispecific Antibody Targeting EGFR and CD3 Inhibits Colon Cancer Growth with Limited Toxicity. Cancer Research. 82(22). 4288–4298. 35 indexed citations

Vasiljeva, Olga, et al.. (2020). Monitoring protease activity in biological tissues using antibody prodrugs as sensing probes. Scientific Reports. 10(1). 5894–5894. 26 indexed citations

Sallee, Nathan A, et al.. (2020). A Pilot Screen of a Novel Peptide Hormone Library Identified Candidate GPR83 Ligands. SLAS DISCOVERY. 25(9). 1047–1063. 7 indexed citations

Stroh, Mark, Michelle Green, Joshua F. Apgar, et al.. (2020). Model‐Informed Drug Development of the Masked Anti‑PD‑L1 Antibody CX‐072. Clinical Pharmacology & Therapeutics. 109(2). 383–393. 10 indexed citations

Boustany, Leila M., Linnea Diep, Yuanhui Huang, et al.. (2018). Abstract A164: EGFR-CD3 bispecific Probody™ therapeutic induces tumor regressions and increases maximum tolerated dose >60-fold in preclinical studies. Molecular Cancer Therapeutics. 17(1_Supplement). A164–A164. 25 indexed citations

Tipton, Kimberly, Kenneth Wong, Jennifer Richardson, et al.. (2016). Abstract 3211: PD-1-targeted Probody therapeutics provide anti-tumor efficacy and a 10-fold dose protection against systemic autoimmunity in preclinical studies. Cancer Research. 76(14_Supplement). 3211–3211. 3 indexed citations

Tolcher, Anthony W., Kyriakos P. Papadopoulos, Amita Patnaik, et al.. (2015). A phase I, first in human study of FP-1039 (GSK3052230), a novel FGF ligand trap, in patients with advanced solid tumors. Annals of Oncology. 27(3). 526–532. 73 indexed citations

Wong, Kenneth, et al.. (2015). In vivo imaging of protease activity by Probody therapeutic activation. Biochimie. 122. 62–67. 25 indexed citations

LaPorte, Sherry L., Daniel R. Hostetter, Linnea Diep, et al.. (2015). Abstract A203: CD3-EGFR bispecific Probody™ therapeutics induced tumor regressions and increased therapeutic window in preclinical studies. Molecular Cancer Therapeutics. 14(12_Supplement_2). A203–A203. 1 indexed citations

10.

Sallee, Nathan A, Ernestine Lee, Andrew L. Rankin, et al.. (2014). Discovery of novel immune checkpoint regulators in a comprehensive library of human extracellular proteins. Journal for ImmunoTherapy of Cancer. 2(S3).

11.

Harding, Thomas C., Li Long, Servando Palencia, et al.. (2013). Blockade of Nonhormonal Fibroblast Growth Factors by FP-1039 Inhibits Growth of Multiple Types of Cancer. Science Translational Medicine. 5(178). 178ra39–178ra39. 104 indexed citations

12.

Vollenweider, Péter, Martin Clodi, Stuart S. Martin, et al.. (1999). An SH2 Domain-Containing 5′ Inositolphosphatase Inhibits Insulin-Induced GLUT4 Translocation and Growth Factor-Induced Actin Filament Rearrangement. Molecular and Cellular Biology. 19(2). 1081–1091. 71 indexed citations

13.

Shyamala, Venkatakrishna, et al.. (1999). High-Throughput Screening for Ligand-Induced c-fos mRNA Expression by Branched DNA Assay in Chinese Hamster Ovary Cells. Analytical Biochemistry. 266(1). 140–147. 8 indexed citations

14.

Senderowicz, Lionel, et al.. (1997). 3-Phosphohistidine Cannot Replace Phosphotyrosine in High-Affinity Binding to Phosphotyrosine Binding or Src Homology 2 Domains. Biochemistry. 36(34). 10538–10544. 7 indexed citations

15.

Klippel, Anke, et al.. (1996). Membrane Localization of Phosphatidylinositol 3-Kinase Is Sufficient To Activate Multiple Signal-Transducing Kinase Pathways. Molecular and Cellular Biology. 16(8). 4117–4127. 395 indexed citations

16.

Kavanaugh, W. Michael & Lewis T. Williams. (1994). An Alternative to SH2 Domains for Binding Tyrosine-Phosphorylated Proteins. Science. 266(5192). 1862–1865. 425 indexed citations

17.

Kavanaugh, W. Michael, Anke Klippel, Jaime A. Escobedo, & Lewis T. Williams. (1992). Modification of the 85-Kilodalton Subunit of Phosphatidylinositol-3 Kinase in Platelet-Derived Growth Factor-Stimulated Cells. Molecular and Cellular Biology. 12(8). 3415–3424. 20 indexed citations

18.

Escobedo, Jaime A., Sutip Navankasattusas, W. Michael Kavanaugh, et al.. (1991). cDNA cloning of a Novel 85 kd protein that has SH2 domains and regulates binding of PI3-kinase to the PDGF β-receptor. Cell. 65(1). 75–82. 528 indexed citations breakdown →

19.

Kavanaugh, W. Michael, Lewis T. Williams, Harlan E. Ives, & Shaun R. Coughlin. (1988). Serotonin-Induced Deoxyribonucleic Acid Synthesis in Vascular Smooth Muscle Cells Involves a Novel, Pertussis Toxin-Sensitive Pathway. Molecular Endocrinology. 2(7). 599–605. 46 indexed citations

20.

Spicer, Eleanor K., W. Michael Kavanaugh, W S Dallas, et al.. (1981). Sequence homologies between A subunits of Escherichia coli and Vibrio cholerae enterotoxins.. Proceedings of the National Academy of Sciences. 78(1). 50–54. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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