Elaine D. MacKenzie

4.6k total citations · 1 hit paper
15 papers, 3.6k citations indexed

About

Elaine D. MacKenzie is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Elaine D. MacKenzie has authored 15 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Cancer Research and 2 papers in Genetics. Recurrent topics in Elaine D. MacKenzie's work include Cancer, Hypoxia, and Metabolism (8 papers), Mitochondrial Function and Pathology (5 papers) and Epigenetics and DNA Methylation (3 papers). Elaine D. MacKenzie is often cited by papers focused on Cancer, Hypoxia, and Metabolism (8 papers), Mitochondrial Function and Pathology (5 papers) and Epigenetics and DNA Methylation (3 papers). Elaine D. MacKenzie collaborates with scholars based in United Kingdom, United States and Netherlands. Elaine D. MacKenzie's co-authors include Eyal Gottlieb, Mary Selak, David G. Watson, Houda Boulahbel, Craig B. Thompson, Yi Pan, Sean M. Armour, M. Celeste Simon, Kyle D. Mansfield and Liang Zheng and has published in prestigious journals such as Nature, Nature Communications and The Journal of Cell Biology.

In The Last Decade

Elaine D. MacKenzie

15 papers receiving 3.6k citations

Hit Papers

Succinate links TCA cycle dysfunction to oncogenesis by i... 2005 2026 2012 2019 2005 500 1000 1.5k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Elaine D. MacKenzie United Kingdom 13 2.5k 1.9k 399 336 296 15 3.6k
Cláudio R. Santos United Kingdom 17 2.2k 0.9× 1.4k 0.7× 411 1.0× 191 0.6× 318 1.1× 21 3.2k
William W. Wheaton United States 9 3.2k 1.3× 2.2k 1.1× 325 0.8× 293 0.9× 307 1.0× 9 4.3k
Saroj P. Mathupala United States 24 2.6k 1.0× 2.1k 1.1× 296 0.7× 214 0.6× 177 0.6× 32 3.6k
Jessica L. Yecies United States 10 2.0k 0.8× 926 0.5× 433 1.1× 333 1.0× 204 0.7× 13 3.0k
Geoffrey D. Girnun United States 25 2.3k 0.9× 1.2k 0.6× 234 0.6× 613 1.8× 200 0.7× 33 3.3k
Evgueni Daikhin United States 8 3.1k 1.2× 2.5k 1.3× 183 0.5× 606 1.8× 170 0.6× 10 4.3k
Barrie Peck United Kingdom 16 2.1k 0.8× 1.6k 0.8× 250 0.6× 240 0.7× 401 1.4× 22 3.2k
Dan Y. Gui United States 14 3.3k 1.3× 2.4k 1.3× 223 0.6× 360 1.1× 258 0.9× 25 4.5k
Houda Boulahbel United Kingdom 7 1.4k 0.6× 1.3k 0.7× 323 0.8× 215 0.6× 142 0.5× 7 2.1k
Karim Bensaad United Kingdom 14 2.6k 1.0× 2.0k 1.0× 242 0.6× 270 0.8× 201 0.7× 18 3.7k

Countries citing papers authored by Elaine D. MacKenzie

Since Specialization
Citations

This map shows the geographic impact of Elaine D. MacKenzie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elaine D. MacKenzie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elaine D. MacKenzie more than expected).

Fields of papers citing papers by Elaine D. MacKenzie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elaine D. MacKenzie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elaine D. MacKenzie. The network helps show where Elaine D. MacKenzie may publish in the future.

Co-authorship network of co-authors of Elaine D. MacKenzie

This figure shows the co-authorship network connecting the top 25 collaborators of Elaine D. MacKenzie. A scholar is included among the top collaborators of Elaine D. MacKenzie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elaine D. MacKenzie. Elaine D. MacKenzie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Cardaci, Simone, Liang Zheng, Gillian Mackay, et al.. (2015). Pyruvate carboxylation enables growth of SDH-deficient cells by supporting aspartate biosynthesis. Nature Cell Biology. 17(10). 1317–1326. 211 indexed citations
2.
Zheng, Liang, Simone Cardaci, Livnat Jerby, et al.. (2015). Fumarate induces redox-dependent senescence by modifying glutathione metabolism. Nature Communications. 6(1). 6001–6001. 205 indexed citations
3.
Zheng, Liang, Elaine D. MacKenzie, Saadia A. Karim, et al.. (2013). Reversed argininosuccinate lyase activity in fumarate hydratase-deficient cancer cells. Cancer & Metabolism. 1(1). 12–12. 85 indexed citations
4.
Durán, Raúl V., Elaine D. MacKenzie, Houda Boulahbel, et al.. (2012). HIF-independent role of prolyl hydroxylases in the cellular response to amino acids. Oncogene. 32(38). 4549–4556. 103 indexed citations
5.
Frezza, Christian, Liang Zheng, Kartik N. Rajagopalan, et al.. (2011). Haem oxygenase is synthetically lethal with the tumour suppressor fumarate hydratase. Nature. 477(7363). 225–228. 377 indexed citations
6.
Tennant, Daniel A., Christian Frezza, Elaine D. MacKenzie, et al.. (2009). Reactivating HIF prolyl hydroxylases under hypoxia results in metabolic catastrophe and cell death. Oncogene. 28(45). 4009–4021. 103 indexed citations
7.
Gonzalvez, François, Zachary T. Schug, Riekelt H. Houtkooper, et al.. (2008). Cardiolipin provides an essential activating platform for caspase-8 on mitochondria. The Journal of Cell Biology. 183(4). 681–696. 233 indexed citations
8.
MacKenzie, Elaine D., Mary Selak, Daniel A. Tennant, et al.. (2007). Cell-Permeating α -Ketoglutarate Derivatives Alleviate Pseudohypoxia in Succinate Dehydrogenase-Deficient Cells. Molecular and Cellular Biology. 27(9). 3282–3289. 295 indexed citations
9.
Selak, Mary, Sean M. Armour, Elaine D. MacKenzie, et al.. (2005). Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-α prolyl hydroxylase. Cancer Cell. 7(1). 77–85. 1632 indexed citations breakdown →
10.
Coutts, Amanda S., Elaine D. MacKenzie, Elen Griffith, & Donald M. Black. (2003). TES is a novel focal adhesion protein with a role in cell spreading. Journal of Cell Science. 116(5). 897–906. 86 indexed citations
11.
Tobias, Edward S., Adam Hurlstone, Elaine D. MacKenzie, Robert McFarlane, & Donald M. Black. (2001). The TES gene at 7q31.1 is methylated in tumours and encodes a novel growth-suppressing LIM domain protein. Oncogene. 20(22). 2844–2853. 83 indexed citations
12.
Lamb, Richard F., Robert F. Hennigan, Kostas D. Katsanakis, et al.. (1997). AP-1-Mediated Invasion Requires Increased Expression of the Hyaluronan Receptor CD44. Molecular and Cellular Biology. 17(2). 963–976. 150 indexed citations
13.
MacKenzie, Elaine D., et al.. (1993). ABL-BCR mRNAs transcribed from chromosome 9q+ in Philadelphia-chromosome-positive chronic myeloid leukaemia.. PubMed. 7(5). 702–6. 9 indexed citations
14.
Benn, Peter, et al.. (1990). bcr breakpoint and prognosis of chronic phase chronic myeloid leukemia [letter; comment]. Blood. 76(12). 2637–2639. 5 indexed citations
15.
Mills, Ken, et al.. (1989). Further evidence that the site of the breakpoint in the major breakpoint cluster region (M-bcr) may be a prognostic indicator.. PubMed. 3(12). 837–40. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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