Dirk Isbrandt

4.7k total citations
72 papers, 3.3k citations indexed

About

Dirk Isbrandt is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cell Biology. According to data from OpenAlex, Dirk Isbrandt has authored 72 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 25 papers in Cardiology and Cardiovascular Medicine and 23 papers in Cell Biology. Recurrent topics in Dirk Isbrandt's work include Ion channel regulation and function (25 papers), Cardiac electrophysiology and arrhythmias (22 papers) and Muscle metabolism and nutrition (19 papers). Dirk Isbrandt is often cited by papers focused on Ion channel regulation and function (25 papers), Cardiac electrophysiology and arrhythmias (22 papers) and Muscle metabolism and nutrition (19 papers). Dirk Isbrandt collaborates with scholars based in Germany, France and United Kingdom. Dirk Isbrandt's co-authors include Olaf Pongs, Axel Neu, Eric Schulze‐Bahr, Günter Breithardt, Patrick Friederich, Hua Hu, Henning Peters, Johan F. Storm, Robert Bähring and Kurt Von Figura and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Dirk Isbrandt

70 papers receiving 3.3k citations

Peers

Dirk Isbrandt
Allen Kaasik Estonia
Keith J. Buckler United Kingdom
Axel Neu Germany
Caryl E. Hill Australia
Jürgen Daut Germany
G. Campbell United Kingdom
Maureen W. McEnery United States
Allen Kaasik Estonia
Dirk Isbrandt
Citations per year, relative to Dirk Isbrandt Dirk Isbrandt (= 1×) peers Allen Kaasik

Countries citing papers authored by Dirk Isbrandt

Since Specialization
Citations

This map shows the geographic impact of Dirk Isbrandt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dirk Isbrandt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dirk Isbrandt more than expected).

Fields of papers citing papers by Dirk Isbrandt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dirk Isbrandt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dirk Isbrandt. The network helps show where Dirk Isbrandt may publish in the future.

Co-authorship network of co-authors of Dirk Isbrandt

This figure shows the co-authorship network connecting the top 25 collaborators of Dirk Isbrandt. A scholar is included among the top collaborators of Dirk Isbrandt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dirk Isbrandt. Dirk Isbrandt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Torre, Eleonora, Mélanie Faure, Isabelle Bidaud, et al.. (2025). L-Type Ca v 1.3 and HCN Channels Mediate Heart Rate Acceleration by Catecholamines. Circulation Research. 138(1). e327497–e327497.
2.
Tellkamp, Frederik, Simon Heß, Milad Mohammadi, et al.. (2022). Autophagy regulates neuronal excitability by controlling cAMP /protein kinase A signaling at the synapse. The EMBO Journal. 41(22). e110963–e110963. 31 indexed citations
3.
Buss, Eric W., Félix Leroy, Tobias Bock, et al.. (2022). Seizures, behavioral deficits, and adverse drug responses in two new genetic mouse models of HCN1 epileptic encephalopathy. eLife. 11. 12 indexed citations
4.
Vay, Sabine Ulrike, Rafael Campos-Martín, Marta Florio, et al.. (2021). Developmental HCN channelopathy results in decreased neural progenitor proliferation and microcephaly in mice. Proceedings of the National Academy of Sciences. 118(35). 7 indexed citations
5.
Zhang, Chuanqiang, Stephan Lawrence Marguet, Tanja Herrmann, et al.. (2021). A limited role of NKCC1 in telencephalic glutamatergic neurons for developing hippocampal network dynamics and behavior. Proceedings of the National Academy of Sciences. 118(14). 18 indexed citations
6.
Muhia, Mary, Sergio Castro‐Gomez, Ute Süsens, et al.. (2019). Disturbed Prefrontal Cortex Activity in the Absence of Schizophrenia-Like Behavioral Dysfunction inArc/Arg3.1Deficient Mice. Journal of Neuroscience. 39(41). 8149–8163. 11 indexed citations
7.
Hornig, Sönke, Erik Hanff, Rainer H. Böger, et al.. (2019). Muscle phenotype of AGAT- and GAMT-deficient mice after simvastatin exposure. Amino Acids. 52(1). 73–85. 10 indexed citations
8.
Castro‐Gomez, Sergio, Sabine Graf, Ute Süsens, et al.. (2018). Arc/Arg3.1 mediates a critical period for spatial learning and hippocampal networks. Proceedings of the National Academy of Sciences. 115(49). 12531–12536. 36 indexed citations
9.
Tran, Christel, Mehrdad Yazdanpanah, Lianna Kyriakopoulou, et al.. (2014). Stable isotope dilution microquantification of creatine metabolites in plasma, whole blood and dried blood spots for pharmacological studies in mouse models of creatine deficiency. Clinica Chimica Acta. 436. 160–168. 12 indexed citations
10.
Marger, Laurine, Pietro Mesirca, Angelo G. Torrente, et al.. (2011). Functional roles of Cav1.3, Cav3.1 and HCN channels in automaticity of mouse atrioventricular cells. Channels. 5(3). 251–261. 62 indexed citations
11.
Neu, Axel, Matthias Paul, Kathrin Sauter, et al.. (2010). A homozygous SCN5A mutation in a severe, recessive type of cardiac conduction disease. Human Mutation. 31(8). E1609–E1621. 24 indexed citations
12.
Marger, Laurine, et al.. (2009). Control of heart rate by cAMP sensitivity of HCN channels. Proceedings of the National Academy of Sciences. 106(29). 12189–12194. 93 indexed citations
13.
Kan, Hermien E., et al.. (2007). Creatine uptake in brain and skeletal muscle of mice lacking guanidinoacetate methyltransferase assessed by magnetic resonance spectroscopy. Journal of Applied Physiology. 102(6). 2121–2127. 22 indexed citations
14.
Isbrandt, Dirk, et al.. (2005). Contribution of N‐ and C‐terminal channel domains to Kv channel interacting proteins in a mammalian cell line. The Journal of Physiology. 568(2). 397–412. 53 indexed citations
15.
Peters, Henning, Hua Hu, Olaf Pongs, Johan F. Storm, & Dirk Isbrandt. (2004). Conditional transgenic suppression of M channels in mouse brain reveals functions in neuronal excitability, resonance and behavior. Nature Neuroscience. 8(1). 51–60. 338 indexed citations
16.
Kan, Hermien E., et al.. (2004). Phosphorylated guanidinoacetate partly compensates for the lack of phosphocreatine in skeletal muscle of mice lacking guanidinoacetate methyltransferase. The Journal of Physiology. 560(1). 219–229. 34 indexed citations
17.
Friederich, Pascal, et al.. (2003). Local anaesthetic sensitivities of cloned HERG channels from human heart: comparison with HERG/MiRP1 and HERG/MiRP1 T8A. British Journal of Anaesthesia. 92(1). 93–101. 32 indexed citations
18.
Schulze‐Bahr, Eric, Axel Neu, Patrick Friederich, et al.. (2003). Pacemaker channel dysfunction in a patient with sinus node disease. Journal of Clinical Investigation. 111(10). 1537–1545. 236 indexed citations
19.
Schmidt, Andreas, Jack J.A. van Asten, Frank Oerlemans, et al.. (2003). MR spectroscopy of muscle and brain in guanidinoacetate methyltransferase (GAMT)‐deficient mice: Validation of an animal model to study creatine deficiency. Magnetic Resonance in Medicine. 50(5). 936–943. 45 indexed citations
20.
Das, Anibh M., Kurt Ullrich, & Dirk Isbrandt. (2000). Upregulation of respiratory chain enzymes in guanidinoacetate methyltransferase deficiency. Journal of Inherited Metabolic Disease. 23(4). 375–377. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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