Matteo E. Mangoni

6.3k total citations
90 papers, 4.4k citations indexed

About

Matteo E. Mangoni is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Matteo E. Mangoni has authored 90 papers receiving a total of 4.4k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Cardiology and Cardiovascular Medicine, 59 papers in Molecular Biology and 32 papers in Cellular and Molecular Neuroscience. Recurrent topics in Matteo E. Mangoni's work include Cardiac electrophysiology and arrhythmias (66 papers), Ion channel regulation and function (50 papers) and Neuroscience and Neural Engineering (19 papers). Matteo E. Mangoni is often cited by papers focused on Cardiac electrophysiology and arrhythmias (66 papers), Ion channel regulation and function (50 papers) and Neuroscience and Neural Engineering (19 papers). Matteo E. Mangoni collaborates with scholars based in France, United States and Italy. Matteo E. Mangoni's co-authors include Joël Nargeot, Pietro Mesirca, Dario DiFrancesco, Brigitte Couette, Jörg Striessnig, Angelo G. Torrente, Emmanuel Bourinet, Gianmaria Maccaferri, Laurine Marger and Josef Platzer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Matteo E. Mangoni

85 papers receiving 4.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matteo E. Mangoni France	33	3.1k	2.6k	1.4k	292	280	90	4.4k
Thomas V. McDonald United States	33	2.6k 0.8×	1.6k 0.6×	939 0.7×	158 0.5×	342 1.2×	88	3.5k
Hemin Chin United States	24	2.1k 0.7×	941 0.4×	1.2k 0.9×	434 1.5×	105 0.4×	45	3.1k
K. George Chandy United States	25	1.7k 0.6×	558 0.2×	647 0.5×	194 0.7×	182 0.7×	67	2.6k
Pompeo Volpe Italy	42	5.3k 1.7×	2.2k 0.8×	2.1k 1.5×	711 2.4×	586 2.1×	132	6.8k
Nili Avidan Israel	25	1.4k 0.5×	617 0.2×	295 0.2×	156 0.5×	404 1.4×	40	2.9k
Martin F. Schneider United States	38	4.1k 1.3×	1.5k 0.6×	2.3k 1.7×	590 2.0×	246 0.9×	113	4.9k
Manjula Mahata United States	36	2.4k 0.8×	256 0.1×	1.8k 1.3×	472 1.6×	59 0.2×	104	4.1k
Mark S. Shapiro United States	40	3.7k 1.2×	1.4k 0.5×	2.3k 1.7×	440 1.5×	477 1.7×	100	4.8k
Peter J. Hanley Germany	31	1.4k 0.5×	425 0.2×	401 0.3×	265 0.9×	405 1.4×	62	3.1k
Isabelle Marty France	36	2.8k 0.9×	1.3k 0.5×	643 0.5×	509 1.7×	219 0.8×	106	3.5k

Countries citing papers authored by Matteo E. Mangoni

Since Specialization

Citations

This map shows the geographic impact of Matteo E. Mangoni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matteo E. Mangoni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matteo E. Mangoni more than expected).

Fields of papers citing papers by Matteo E. Mangoni

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matteo E. Mangoni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matteo E. Mangoni. The network helps show where Matteo E. Mangoni may publish in the future.

Co-authorship network of co-authors of Matteo E. Mangoni

This figure shows the co-authorship network connecting the top 25 collaborators of Matteo E. Mangoni. A scholar is included among the top collaborators of Matteo E. Mangoni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matteo E. Mangoni. Matteo E. Mangoni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Torre, Eleonora, Mélanie Faure, Isabelle Bidaud, et al.. (2025). L-Type Ca _v 1.3 and HCN Channels Mediate Heart Rate Acceleration by Catecholamines. Circulation Research. 138(1). e327497–e327497.

Boyett, Mark R., Gwilym M. Morris, Aneil Malhotra, et al.. (2024). Symptomatic bradyarrhythmias in the athlete—Underlying mechanisms and treatments. Heart Rhythm. 21(8). 1415–1427. 4 indexed citations

Bartolucci, Chiara, et al.. (2024). Computational modelling of mouse atrio ventricular node action potential and automaticity. The Journal of Physiology. 602(19). 4821–4847. 2 indexed citations

Wallace, Michael B., Nathaniel P. Murphy, Mei Han, et al.. (2023). Impact of stress on cardiac phenotypes in mice harboring an ankyrin-B disease variant. Journal of Biological Chemistry. 299(6). 104818–104818. 1 indexed citations

Odening, Katja E., Ana M. Gómez, Dobromir Dobrev, et al.. (2021). ESC working group on cardiac cellular electrophysiology position paper: relevance, opportunities, and limitations of experimental models for cardiac electrophysiology research. EP Europace. 23(11). 1795–1814. 30 indexed citations

Torrente, Angelo G., et al.. (2020). Hypercalcemia impairs sino-atrial automaticity through excessive Cav1.2-mediated Ca2+ influx. Archives of Cardiovascular Diseases Supplements. 12(2-4). 255–255. 2 indexed citations

Bidaud, Isabelle, et al.. (2020). Cholinergic regulation of heart rate: Functional importance of l-type cav1.3 channels. Archives of Cardiovascular Diseases Supplements. 12(2-4). 255–255.

Bidaud, Isabelle, Alicia D’Souza, Angelo G. Torrente, et al.. (2020). Genetic ablation of G protein-gated inwardly rectifying K+ (Girk)4 channels prevents heart rate reduction induced by intensive exercise training. Archives of Cardiovascular Diseases Supplements. 12(2-4). 253–254. 1 indexed citations

Saponaro, Andrea, Francesca Cantini, Alessandro Porro, et al.. (2019). Developing Synthetic Peptides to Regulate Native HCN Channels. Biophysical Journal. 116(3). 302a–302a. 1 indexed citations

10.

Saponaro, Andrea, Francesca Cantini, Alessandro Porro, et al.. (2018). A synthetic peptide that prevents cAMP regulation in mammalian hyperpolarization-activated cyclic nucleotide-gated (HCN) channels. eLife. 7. 38 indexed citations

11.

Mesirca, Pietro, Elena Marqués‐Sulé, Alexandra Zahradníková, et al.. (2017). RyR2R420Q catecholaminergic polymorphic ventricular tachycardia mutation induces bradycardia by disturbing the coupled clock pacemaker mechanism. JCI Insight. 2(8). 24 indexed citations

12.

Torrente, Angelo G., Pietro Mesirca, Patricia Ñeco, et al.. (2016). L-type Ca_v1.3 channels regulate ryanodine receptor-dependent Ca²⁺release during sino-atrial node pacemaker activity. Cardiovascular Research. 109(3). 451–461. 75 indexed citations

13.

Sah, Rajan, Pietro Mesirca, Jonathan N. Rosen, et al.. (2013). Ion channel-kinase TRPM 7 is required for maintaining cardiac automaticity. Proceedings of the National Academy of Sciences. 110(32). E3037–46. 84 indexed citations

14.

Ñeco, Patricia, Angelo G. Torrente, Pietro Mesirca, et al.. (2012). Paradoxical Effect of Increased Diastolic Ca ²⁺ Release and Decreased Sinoatrial Node Activity in a Mouse Model of Catecholaminergic Polymorphic Ventricular Tachycardia. Circulation. 126(4). 392–401. 64 indexed citations

15.

Marger, Laurine, Pietro Mesirca, Angelo G. Torrente, et al.. (2011). Functional roles of Ca_v1.3, Ca_v3.1 and HCN channels in automaticity of mouse atrioventricular cells. Channels. 5(3). 251–261. 62 indexed citations

16.

Marger, Laurine, et al.. (2010). Pacemaker Cells of the Atrioventricular Node are CaV1.3 Dependent Oscillators. Biophysical Journal. 98(3). 339a–339a. 1 indexed citations

17.

Marger, Laurine, et al.. (2009). Control of heart rate by cAMP sensitivity of HCN channels. Proceedings of the National Academy of Sciences. 106(29). 12189–12194. 93 indexed citations

18.

Mangoni, Matteo E., Anne‐Laure Léoni, Brigitte Couette, et al.. (2006). Bradycardia and Slowing of the Atrioventricular Conduction in Mice Lacking Ca _V 3.1/α _1G T-Type Calcium Channels. Circulation Research. 98(11). 1422–1430. 228 indexed citations

19.

Marger, Laurine, Kenneth W. Hewett, T. Jarry‐Guichard, et al.. (2006). Nkx2.5 cell-autonomous gene function is required for the postnatal formation of the peripheral ventricular conduction system. Developmental Biology. 303(2). 740–753. 61 indexed citations

20.

Clark, Robert B., et al.. (2004). A rapidly activating delayed rectifier K⁺ current regulates pacemaker activity in adult mouse sinoatrial node cells. American Journal of Physiology-Heart and Circulatory Physiology. 286(5). H1757–H1766. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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