Bé Wieringa

15.6k total citations · 4 hit papers
193 papers, 12.6k citations indexed

About

Bé Wieringa is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Bé Wieringa has authored 193 papers receiving a total of 12.6k indexed citations (citations by other indexed papers that have themselves been cited), including 152 papers in Molecular Biology, 66 papers in Cellular and Molecular Neuroscience and 48 papers in Cell Biology. Recurrent topics in Bé Wieringa's work include Genetic Neurodegenerative Diseases (60 papers), Mitochondrial Function and Pathology (36 papers) and Muscle Physiology and Disorders (30 papers). Bé Wieringa is often cited by papers focused on Genetic Neurodegenerative Diseases (60 papers), Mitochondrial Function and Pathology (36 papers) and Muscle Physiology and Disorders (30 papers). Bé Wieringa collaborates with scholars based in Netherlands, United States and Germany. Bé Wieringa's co-authors include Gerard J.M. Martens, Helena Xicoy, Frank Oerlemans, Robert G. Korneluk, Gert Jansen, Wiljan Hendriks, Jack Fransen, Bernard A. van Oost, Mani S. Mahadevan and Chris T. Amemiya and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

Bé Wieringa

193 papers receiving 12.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Myotonic Dystrophy Mutation: an Unstable CTG Repeat in the 3′ Untranslated region of the Gene

1992 1.3k citations Gert Jansen, Bé Wieringa et al. profile →
An Unstable Triplet Repeat in a Gene Related to Myotonic Muscular Dystrophy

1992 1.2k citations Bé Wieringa et al. profile →
The SH-SY5Y cell line in Parkinson’s disease research: a systematic review

2017 703 citations Bé Wieringa et al. profile →
Requirement of Human Renal Water Channel Aquaporin-2 for Vasopressin-dependent Concentration of Urine

1994 698 citations Bé Wieringa et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Bé Wieringa Netherlands	55	9.3k	4.9k	1.7k	1.6k	1.4k	193	12.6k
Rune R. Frants Netherlands	73	10.3k 1.1×	3.5k 0.7×	873 0.5×	1.4k 0.8×	2.4k 1.7×	317	18.5k
Giorgio Casari Italy	55	5.6k 0.6×	2.3k 0.5×	842 0.5×	1.4k 0.8×	815 0.6×	144	10.5k
Thomas L. Schwarz United States	63	12.2k 1.3×	6.9k 1.4×	4.5k 2.7×	1.3k 0.8×	1.6k 1.2×	120	17.1k
Akira Kakizuka Japan	54	11.6k 1.2×	4.0k 0.8×	2.6k 1.5×	1.2k 0.7×	440 0.3×	146	15.7k
Hiroyoshi Hidaka Japan	59	9.3k 1.0×	2.6k 0.5×	1.7k 1.0×	688 0.4×	1.4k 1.0×	326	14.3k
Yuko Fukata Japan	54	9.5k 1.0×	3.9k 0.8×	6.0k 3.6×	1.2k 0.8×	982 0.7×	99	15.3k
Miriam H. Meisler United States	65	8.9k 1.0×	5.3k 1.1×	1.7k 1.0×	897 0.5×	1.3k 0.9×	248	14.3k
Michio Hirano United States	82	16.4k 1.8×	2.1k 0.4×	754 0.5×	1.3k 0.8×	892 0.6×	341	20.5k
Kenneth H. Fischbeck United States	66	13.1k 1.4×	9.4k 1.9×	1.8k 1.1×	4.4k 2.7×	682 0.5×	207	18.9k
P. H. Seeburg Germany	59	10.5k 1.1×	6.9k 1.4×	605 0.4×	698 0.4×	495 0.4×	102	15.9k

Countries citing papers authored by Bé Wieringa

Since Specialization

Citations

This map shows the geographic impact of Bé Wieringa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bé Wieringa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bé Wieringa more than expected).

Fields of papers citing papers by Bé Wieringa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bé Wieringa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bé Wieringa. The network helps show where Bé Wieringa may publish in the future.

Co-authorship network of co-authors of Bé Wieringa

This figure shows the co-authorship network connecting the top 25 collaborators of Bé Wieringa. A scholar is included among the top collaborators of Bé Wieringa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bé Wieringa. Bé Wieringa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Wieringa, Bé, et al.. (2019). Expanded CUG repeats in DMPK transcripts adopt diverse hairpin conformations without influencing the structure of the flanking sequences. RNA. 25(4). 481–495. 12 indexed citations

Mulders, Susan, et al.. (2013). Design and Analysis of Effects of Triplet Repeat Oligonucleotides in Cell Models for Myotonic Dystrophy. Molecular Therapy — Nucleic Acids. 2. e81–e81. 36 indexed citations

Middelbeek, Jeroen, Arthur J. Kuipers, Linda Henneman, et al.. (2012). TRPM7 Is Required for Breast Tumor Cell Metastasis. Cancer Research. 72(16). 4250–4261. 176 indexed citations

Overheul, Gijs J., et al.. (2012). Gene duplication and conversion events shaped three homologous, differentially expressed myosin regulatory light chain (MLC2) genes. European Journal of Cell Biology. 91(8). 629–639. 11 indexed citations

Valsecchi, Federica, Claire Monge, Marleen Forkink, et al.. (2012). Metabolic consequences of NDUFS4 gene deletion in immortalized mouse embryonic fibroblasts. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1817(10). 1925–1936. 57 indexed citations

Mulders, Susan, Walther J. A. A. van den Broek, Thurman M. Wheeler, et al.. (2009). Triplet-repeat oligonucleotide-mediated reversal of RNA toxicity in myotonic dystrophy. Proceedings of the National Academy of Sciences. 106(33). 13915–13920. 204 indexed citations

Sandström, Marie, et al.. (2007). Activation of glucose transport and AMP‐activated protein kinase during muscle contraction in adenylate kinase‐1 knockout mice. Acta Physiologica. 192(3). 413–420. 15 indexed citations

Wanschers, Bas F.J., et al.. (2007). Rab6 family proteins interact with the dynein light chain protein DYNLRB1. Cell Motility and the Cytoskeleton. 65(3). 183–196. 62 indexed citations

Klivènyi, Péter, Noel Y. Calingasan, Anatoly A. Starkov, et al.. (2004). Neuroprotective mechanisms of creatine occur in the absence of mitochondrial creatine kinase. Neurobiology of Disease. 15(3). 610–617. 47 indexed citations

10.

Ham, Marco van, Huib J. E. Croes, Jan Schepens, et al.. (2003). Cloning and characterization of mCRIP2, a mouse LIM‐only protein that interacts with PDZ domain IV of PTP‐BL. Genes to Cells. 8(7). 631–644. 26 indexed citations

11.

Janssen, Edwin, J. W. Kuiper, Denice M. Hodgson, et al.. (2003). Two structurally distinct and spatially compartmentalized adenylate kinases are expressed from the AK1 gene in mouse brain. Molecular and Cellular Biochemistry. 256-257(1-2). 59–72. 23 indexed citations

12.

Schaapveld, Roel Q.J., Jan Schepens, Dietmar Bächner, et al.. (1998). Developmental expression of the cell adhesion molecule-like protein tyrosine phosphatases LAR, RPTPδ and RPTPσ in the mouse. Mechanisms of Development. 77(1). 59–62. 60 indexed citations

13.

Hurk, José A. J. M. van den, Wiljan Hendriks, Dorien J.R. van de Pol, et al.. (1997). Mouse Choroideremia Gene Mutation Causes Photoreceptor Cell Degeneration and is not Transmitted through the Female Germline. Human Molecular Genetics. 6(6). 851–858. 57 indexed citations

14.

Schaapveld, Roel Q.J., Jan Schepens, Gertraud W. Robinson, et al.. (1997). Impaired Mammary Gland Development and Function in Mice Lacking LAR Receptor-like Tyrosine Phosphatase Activity. Developmental Biology. 188(1). 134–146. 114 indexed citations

15.

Jansen, Gert, Patricia J.T.A. Groenen, Dietmar Bächner, et al.. (1996). Abnormal myotonic dystrophy protein kinase levels produce only mild myopathy in mice. Nature Genetics. 13(3). 316–324. 250 indexed citations

16.

Steeghs, Karen, Frank Oerlemans, & Bé Wieringa. (1995). Mice deficient in ubiquitous mitochondrial creatine kinase are viable and fertile. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1230(3). 130–138. 40 indexed citations

17.

Deursen, Jan van & Bé Wieringa. (1994). Approaching the multifaceted nature of energy metabolism: inactivation of the cytosolic creatine kinases via homologous recombination in mouse embryonic stem cells. Molecular and Cellular Biochemistry. 133-134(1). 263–274. 7 indexed citations

18.

Mariman, Edwin C.M. & Bé Wieringa. (1991). Expression of the gene encoding human brain creatine kinase depends on sequences immediately following the transcription start point. Gene. 102(2). 205–212. 20 indexed citations

19.

Diergaarde, P.J., Bé Wieringa, E. M. Bleeker‐Wagemakers, et al.. (1989). Physical fine-mapping of a deletion spanning the Norrie gene. Human Genetics. 84(1). 22–26. 13 indexed citations

20.

Oerlemans, Frank, H.J.M. Smeets, Jan Schepens, et al.. (1989). Definition of subchromosomal intervals around the myotonic dystrophy gene region at 19q. Genomics. 4(3). 384–396. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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