Andrew R. Marks

684 total citations
10 papers, 491 citations indexed

About

Andrew R. Marks is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Andrew R. Marks has authored 10 papers receiving a total of 491 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Cardiology and Cardiovascular Medicine and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Andrew R. Marks's work include Ion channel regulation and function (5 papers), Mitochondrial Function and Pathology (3 papers) and Cardiac electrophysiology and arrhythmias (3 papers). Andrew R. Marks is often cited by papers focused on Ion channel regulation and function (5 papers), Mitochondrial Function and Pathology (3 papers) and Cardiac electrophysiology and arrhythmias (3 papers). Andrew R. Marks collaborates with scholars based in United States, France and Italy. Andrew R. Marks's co-authors include Gaetano Santulli, Steven Reiken, Alain Lacampagne, Nathalie Saint, Renaud Bussiere, Mounia Chami, Ran Zalk, Frédéric Checler, Haikel Dridi and J Watras and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Andrew R. Marks

7 papers receiving 485 citations

Peers

Andrew R. Marks
Michael J. Higgins United States
Tanya R. Cully Australia
Cristina I. Linde United States
Alisa Umanskaya United States
Korah P. Kuruvilla United States
Alice E. Adriaenssens United Kingdom
Andrew R. Marks
Citations per year, relative to Andrew R. Marks Andrew R. Marks (= 1×) peers Yuta Kobayashi

Countries citing papers authored by Andrew R. Marks

Since Specialization
Citations

This map shows the geographic impact of Andrew R. Marks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew R. Marks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew R. Marks more than expected).

Fields of papers citing papers by Andrew R. Marks

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew R. Marks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew R. Marks. The network helps show where Andrew R. Marks may publish in the future.

Co-authorship network of co-authors of Andrew R. Marks

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew R. Marks. A scholar is included among the top collaborators of Andrew R. Marks based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew R. Marks. Andrew R. Marks is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Torre, Eleonora, Mélanie Faure, Isabelle Bidaud, et al.. (2025). L-Type Ca v 1.3 and HCN Channels Mediate Heart Rate Acceleration by Catecholamines. Circulation Research. 138(1). e327497–e327497.
2.
Reiken, Steve, Joël Rousseau, Anetta Wronska, et al.. (2025). Universal Prime Editing Therapeutic Strategy for RyR1-Related Myopathies: A Protective Mutation Rescues Leaky RyR1 Channel. International Journal of Molecular Sciences. 26(7). 2835–2835.
3.
Marx, Steven O., et al.. (2025). Targeting Calcium Regulation for Heart Failure and Arrhythmia Therapeutics: A Critical Review. Circulation. 152(13). 957–970.
4.
Dridi, Haikel, Frances M. Forrester, Alisa Umanskaya, et al.. (2022). Role of oxidation of excitation-contraction coupling machinery in age-dependent loss of muscle function in Caenorhabditis elegans. eLife. 11. 5 indexed citations
5.
Miotto, Marco, Haikel Dridi, Qi Yuan, et al.. (2022). Structural analyses of human ryanodine receptor type 2 channels reveal the mechanisms for sudden cardiac death and treatment. Science Advances. 8(29). eabo1272–eabo1272. 37 indexed citations
6.
Lacampagne, Alain, Xiaoping Liu, Steven Reiken, et al.. (2017). Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer’s disease-likepathologies and cognitive deficits. Acta Neuropathologica. 134(5). 749–767. 133 indexed citations
7.
Bussiere, Renaud, Alain Lacampagne, Steven Reiken, et al.. (2017). Amyloid β production is regulated by β2-adrenergic signaling-mediated post-translational modifications of the ryanodine receptor. Journal of Biological Chemistry. 292(24). 10153–10168. 48 indexed citations
8.
Matecki, Stéfan, Haikel Dridi, Boris Jung, et al.. (2016). Leaky ryanodine receptors contribute to diaphragmatic weakness during mechanical ventilation. Proceedings of the National Academy of Sciences. 113(32). 9069–9074. 78 indexed citations
9.
Santulli, Gaetano & Andrew R. Marks. (2015). Essential Roles of Intracellular Calcium Release Channels in Muscle, Brain, Metabolism, and Aging. Current Molecular Pharmacology. 8(2). 206–222. 156 indexed citations
10.
Moschella, Maria C., et al.. (1995). Inositol 1,4,5-trisphosphate receptor in skeletal muscle: differential expression in myofibres. Journal of Muscle Research and Cell Motility. 16(4). 390–400. 34 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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