Eleonora Torre

576 total citations
22 papers, 322 citations indexed

About

Eleonora Torre is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Eleonora Torre has authored 22 papers receiving a total of 322 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Cardiology and Cardiovascular Medicine, 14 papers in Molecular Biology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Eleonora Torre's work include Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (11 papers) and Cardiovascular Function and Risk Factors (4 papers). Eleonora Torre is often cited by papers focused on Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (11 papers) and Cardiovascular Function and Risk Factors (4 papers). Eleonora Torre collaborates with scholars based in Italy, France and Taiwan. Eleonora Torre's co-authors include Marcella Rocchetti, Antonio Zaza, Pietro Mesirca, Claudia Altomare, Matteo E. Mangoni, Lia Crotti, Gaspare Mostacciuolo, Luca Sala, Manuela Mura and Isabelle Bidaud and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Circulation Research.

In The Last Decade

Eleonora Torre

20 papers receiving 320 citations

Peers

Eleonora Torre
Samantha C. Salvage United Kingdom
Sophie Schobesberger United Kingdom
Hannah M. Campbell United States
Lidiya Sul United States
Shawn M. Crump United States
Ravi Vaidyanathan United States
Eleonora Torre
Citations per year, relative to Eleonora Torre Eleonora Torre (= 1×) peers Sören Brandenburg

Countries citing papers authored by Eleonora Torre

Since Specialization
Citations

This map shows the geographic impact of Eleonora Torre's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eleonora Torre with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eleonora Torre more than expected).

Fields of papers citing papers by Eleonora Torre

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eleonora Torre. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eleonora Torre. The network helps show where Eleonora Torre may publish in the future.

Co-authorship network of co-authors of Eleonora Torre

This figure shows the co-authorship network connecting the top 25 collaborators of Eleonora Torre. A scholar is included among the top collaborators of Eleonora Torre based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eleonora Torre. Eleonora Torre is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Torre, Eleonora, Mélanie Faure, Isabelle Bidaud, et al.. (2025). L-Type Ca v 1.3 and HCN Channels Mediate Heart Rate Acceleration by Catecholamines. Circulation Research. 138(1). e327497–e327497.
2.
Hsu, Shih-Che, Mara Ferrandi, Paolo Barassi, et al.. (2024). Selective SERCA2a activator as a candidate for chronic heart failure therapy. Journal of Translational Medicine. 22(1). 5 indexed citations
3.
Bartolucci, Chiara, et al.. (2024). Computational modelling of mouse atrio ventricular node action potential and automaticity. The Journal of Physiology. 602(19). 4821–4847. 2 indexed citations
4.
Mesirca, Pietro, Jean Chemin, Christian Barrère, et al.. (2024). Selective blockade of Cav1.2 (α1C) versus Cav1.3 (α1D) L-type calcium channels by the black mamba toxin calciseptine. Nature Communications. 15(1). 54–54. 10 indexed citations
5.
Torre, Eleonora, Matteo E. Mangoni, Alain Lacampagne, Albano C. Méli, & Pietro Mesirca. (2024). State-of-the-Art Differentiation Protocols for Patient-Derived Cardiac Pacemaker Cells. International Journal of Molecular Sciences. 25(6). 3387–3387. 1 indexed citations
6.
Torre, Eleonora, Colin H. Peters, W. Garrett Nichols, et al.. (2024). Identifying sex similarities and differences in structure and function of the sinoatrial node in the mouse heart. Frontiers in Medicine. 11. 1488478–1488478. 3 indexed citations
7.
DiFrancesco, Mattia L., Eleonora Torre, Pietro Mesirca, et al.. (2023). Characterization of sinoatrial automaticity in Microcebus murinus to study the effect of aging on cardiac activity and the correlation with longevity. Scientific Reports. 13(1). 3054–3054. 1 indexed citations
8.
Zorn‐Pauly, Klaus, Pietro Mesirca, Chintan N. Koyani, et al.. (2023). Lipopolysaccharide-induced sepsis impairs M2R-GIRK signaling in the mouse sinoatrial node. Proceedings of the National Academy of Sciences. 120(28). e2210152120–e2210152120. 5 indexed citations
9.
Ferrandi, Mara, Paolo Barassi, Shih-Che Hsu, et al.. (2022). Istaroxime Metabolite PST3093 Selectively Stimulates SERCA2a and Reverses Disease-Induced Changes in Cardiac Function. Journal of Pharmacology and Experimental Therapeutics. 384(1). 231–244. 13 indexed citations
10.
Luraghi, Andrea, Mara Ferrandi, Paolo Barassi, et al.. (2022). Highly Selective SERCA2a Activators: Preclinical Development of a Congeneric Group of First-in-Class Drug Leads against Heart Failure. Journal of Medicinal Chemistry. 65(10). 7324–7333. 14 indexed citations
11.
Torre, Eleonora, Isabelle Bidaud, Ned Lamb, et al.. (2022). L-Type Cav1.3 Calcium Channels Are Required for Beta-Adrenergic Triggered Automaticity in Dormant Mouse Sinoatrial Pacemaker Cells. Cells. 11(7). 1114–1114. 23 indexed citations
12.
Altomare, Claudia, Giuseppina Milano, Vanessa Biemmi, et al.. (2021). Structural and Electrophysiological Changes in a Model of Cardiotoxicity Induced by Anthracycline Combined With Trastuzumab. Frontiers in Physiology. 12. 658790–658790. 14 indexed citations
13.
Torre, Eleonora, Mara Ferrandi, Paolo Barassi, et al.. (2021). SERCA2a stimulation by istaroxime improves intracellular Ca2+ handling and diastolic dysfunction in a model of diabetic cardiomyopathy. Cardiovascular Research. 118(4). 1020–1032. 37 indexed citations
14.
Bidaud, Isabelle, Alicia D’Souza, Gabriella Forte, et al.. (2021). Genetic Ablation of G Protein-Gated Inwardly Rectifying K+ Channels Prevents Training-Induced Sinus Bradycardia. Frontiers in Physiology. 11. 519382–519382. 12 indexed citations
16.
Sala, Luca, Manuela Mura, Marcella Rocchetti, et al.. (2020). MTMR4 SNVs modulate ion channel degradation and clinical severity in congenital long QT syndrome: insights in the mechanism of action of protective modifier genes. Cardiovascular Research. 117(3). 767–779. 32 indexed citations
17.
Torre, Eleonora, Isabelle Bidaud, Angelo G. Torrente, et al.. (2020). Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity. Scientific Reports. 10(1). 18906–18906. 34 indexed citations
18.
Torre, Eleonora, Paolo Barassi, Mara Ferrandi, et al.. (2019). Istaroxime improves diabetic diastolic dysfunction through SERCA stimulation. Archives of Cardiovascular Diseases Supplements. 11(2). 234–235. 3 indexed citations
19.
Ronchi, Carlotta, et al.. (2017). Late sodium current and intracellular ionic homeostasis in acute ischemia. Basic Research in Cardiology. 112(2). 12–12. 22 indexed citations
20.
Rocchetti, Marcella, Luca Sala, Lisa Dreizehnter, et al.. (2017). Elucidating arrhythmogenic mechanisms of long-QT syndrome CALM1-F142L mutation in patient-specific induced pluripotent stem cell-derived cardiomyocytes. Cardiovascular Research. 113(5). 531–541. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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