Thomas Koeglsperger
- Neurology top 0.2%
- Immunology top 2%
- Physiology top 2%
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 2%
- Co-authors
- Howard L. WeinerShaomin LiDennis J. SelkoeMing JinMark P. JedrychowskiRichard M. RansohoffRon CialicGalina Gabriely
- Topics
- Parkinson's Disease Mechanisms and Treatments (15 papers)Neurological disorders and treatments (10 papers)Alzheimer's disease research and treatments (5 papers)
- Partner nations
- GermanyUnited StatesAustria
In The Last Decade
Thomas Koeglsperger
27 papers receiving 3.4k citations
Hit Papers
Peers
Comparison fields: 5 of 108
- Neurology 1.8k
- Immunology 1.0k
- Physiology 981
- Molecular Biology 845
- Cellular and Molecular Neuroscience 817
Countries citing papers authored by Thomas Koeglsperger
This map shows the geographic impact of Thomas Koeglsperger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Koeglsperger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Koeglsperger more than expected).
Fields of papers citing papers by Thomas Koeglsperger
This network shows the impact of papers produced by Thomas Koeglsperger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Koeglsperger. The network helps show where Thomas Koeglsperger may publish in the future.
Co-authorship network of co-authors of Thomas Koeglsperger
This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Koeglsperger. A scholar is included among the top collaborators of Thomas Koeglsperger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Koeglsperger. Thomas Koeglsperger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 0 | |
| 5 | 2 | |
| 6 | 6 | |
| 7 | 61 | |
| 8 | 4 | |
| 9 | 64 | |
| 10 | 0 | |
| 11 | 6 | |
| 12 | 14 | |
| 13 | 14 | |
| 14 | 137 | |
| 15 | 47 | |
| 16 | 137 | |
| 17 | 18 | |
| 18 | 122 | |
| 19 | 145 | |
| 20 | 84 |
About Thomas Koeglsperger
Thomas Koeglsperger is a scholar working on Neurology, Cellular and Molecular Neuroscience and Neurology, having authored 30 papers that have together received 3.5k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (15 papers), Neurological disorders and treatments (10 papers) and Alzheimer's disease research and treatments (5 papers). The work is most often cited by research in Neurology (1.8k citations), Biological Psychiatry (260 citations) and Developmental Neuroscience (305 citations). Thomas Koeglsperger has collaborated with scholars based in Germany, United States and Austria. Frequent co-authors include Howard L. Weiner, Shaomin Li, Dennis J. Selkoe, Ming Jin, Mark P. Jedrychowski, Richard M. Ransohoff, Ron Cialic, Galina Gabriely, Craig S. Moore and Jeffrey D. Rothstein. Their work appears in journals such as Angewandte Chemie International Edition, Neuron and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.