Tae‐In Kam

7.8k total citations · 3 hit papers
38 papers, 3.4k citations indexed

About

Tae‐In Kam is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Tae‐In Kam has authored 38 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Physiology, 15 papers in Molecular Biology and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Tae‐In Kam's work include Alzheimer's disease research and treatments (18 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). Tae‐In Kam is often cited by papers focused on Alzheimer's disease research and treatments (18 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). Tae‐In Kam collaborates with scholars based in United States, South Korea and Japan. Tae‐In Kam's co-authors include Valina L. Dawson, Ted M. Dawson, Yong‐Keun Jung, Jihoon Nah, Sunmin Jung, Senthilkumar S. Karuppagounder, Seung-Min Yoo, Jared T. Hinkle, Nikhil Panicker and Hye-Hyun Ahn and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

Tae‐In Kam

37 papers receiving 3.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Transneuronal Propagation of Pathologic α-Synuclein from the Gut to the Brain Models Parkinson’s Disease

2019 961 citations Sangjune Kim, Seung‐Hwan Kwon et al. Neuron profile →
Overexpression of Atg5 in mice activates autophagy and extends lifespan

2013 545 citations Jihoon Nah, Tae‐In Kam et al. profile →
Neuronal NLRP3 is a parkin substrate that drives neurodegeneration in Parkinson’s disease

2022 139 citations Nikhil Panicker, Tae‐In Kam et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Tae‐In Kam United States	24	1.3k	1.1k	1.1k	788	657	38	3.4k
Senthilkumar S. Karuppagounder United States	32	1.8k 1.4×	1.8k 1.6×	1.0k 1.0×	643 0.8×	1.1k 1.7×	58	4.5k
Itender Singh United States	19	1.2k 0.9×	1.1k 1.0×	1.4k 1.4×	1.9k 2.3×	940 1.4×	24	4.8k
Carla L. Busceti Italy	42	1.7k 1.3×	1.1k 1.0×	881 0.8×	615 0.8×	2.0k 3.0×	141	4.6k
Xingshun Xu China	36	2.0k 1.6×	505 0.5×	566 0.5×	557 0.7×	836 1.3×	105	4.0k
Francesca Biagioni Italy	39	1.6k 1.2×	676 0.6×	624 0.6×	490 0.6×	1.5k 2.3×	137	4.0k
M. Zameel Cader United Kingdom	29	2.0k 1.6×	495 0.4×	1.2k 1.1×	778 1.0×	880 1.3×	80	4.7k
Melissa K. McCoy United States	15	1.0k 0.8×	852 0.8×	483 0.5×	958 1.2×	731 1.1×	22	2.7k
Yazhou Li United States	36	1.7k 1.3×	1.4k 1.3×	1.1k 1.1×	748 0.9×	1.2k 1.8×	74	5.3k
Anne Grünewald Germany	33	2.0k 1.5×	1.9k 1.7×	752 0.7×	821 1.0×	1.1k 1.7×	77	4.1k
Gundars Goldsteins Finland	34	1.5k 1.2×	914 0.8×	1.3k 1.2×	1.8k 2.2×	1.1k 1.7×	50	4.6k

Countries citing papers authored by Tae‐In Kam

Since Specialization

Citations

This map shows the geographic impact of Tae‐In Kam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tae‐In Kam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tae‐In Kam more than expected).

Fields of papers citing papers by Tae‐In Kam

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tae‐In Kam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tae‐In Kam. The network helps show where Tae‐In Kam may publish in the future.

Co-authorship network of co-authors of Tae‐In Kam

This figure shows the co-authorship network connecting the top 25 collaborators of Tae‐In Kam. A scholar is included among the top collaborators of Tae‐In Kam based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tae‐In Kam. Tae‐In Kam is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Mills, Kelly A., Yong Du, Jennifer M. Coughlin, et al.. (2025). Exploring [11C]CPPC as a CSF1R-targeted PET imaging marker for early Parkinson’s disease severity. Journal of Clinical Investigation. 135(12). 2 indexed citations

Shin, Jisu, et al.. (2025). In‐Depth Cell‐Type‐Specific Proteome Landscape of the Brain from Human Amyloid‐β Overexpression Mouse Model. Advanced Science. 12(20). e2409318–e2409318.

Kang, Hojin, Areum Jo, Xiaobo Mao, et al.. (2023). PARIS undergoes liquid–liquid phase separation and poly( ADP ‐ribose)‐mediated solidification. EMBO Reports. 24(11). e56166–e56166. 6 indexed citations

Chun, Yoon S., Mi‐Yeon Kim, Sun‐Young Lee, et al.. (2022). MEK1/2 inhibition rescues neurodegeneration by TFEB-mediated activation of autophagic lysosomal function in a model of Alzheimer’s Disease. Molecular Psychiatry. 27(11). 4770–4780. 28 indexed citations

Panicker, Nikhil, Tae‐In Kam, Hu Wang, et al.. (2022). Neuronal NLRP3 is a parkin substrate that drives neurodegeneration in Parkinson’s disease. Neuron. 110(15). 2422–2437.e9. 139 indexed citations breakdown →

Park, Hyejin, Youngwon Kim, Jae‐Hoon Lee, et al.. (2022). Pathogenic Role of RAGE in Tau Transmission and Memory Deficits. Biological Psychiatry. 93(9). 829–841. 18 indexed citations

Park, Jong Sung, Tae‐In Kam, Saebom Lee, et al.. (2021). Blocking microglial activation of reactive astrocytes is neuroprotective in models of Alzheimer’s disease. Acta Neuropathologica Communications. 9(1). 78–78. 134 indexed citations

Aggarwal, Akanksha, et al.. (2021). Recent advances in preventing neurodegenerative diseases. PubMed. 10. 81–81. 13 indexed citations

Kam, Tae‐In, Jared T. Hinkle, Ted M. Dawson, & Valina L. Dawson. (2020). Microglia and astrocyte dysfunction in parkinson's disease. Neurobiology of Disease. 144. 105028–105028. 247 indexed citations

10.

Park, Hyejin, Tae‐In Kam, Ted M. Dawson, & Valina L. Dawson. (2020). Poly (ADP-ribose) (PAR)-dependent cell death in neurodegenerative diseases. International review of cell and molecular biology. 353. 1–29. 80 indexed citations

11.

Kim, Sangjune, Seung‐Hwan Kwon, Tae‐In Kam, et al.. (2019). Transneuronal Propagation of Pathologic α-Synuclein from the Gut to the Brain Models Parkinson’s Disease. Neuron. 103(4). 627–641.e7. 961 indexed citations breakdown →

12.

Jin, Jing, Matthew D. Smith, Calvin J. Kersbergen, et al.. (2019). Glial pathology and retinal neurotoxicity in the anterior visual pathway in experimental autoimmune encephalomyelitis. Acta Neuropathologica Communications. 7(1). 125–125. 54 indexed citations

13.

Gwon, Youngdae, Tae‐In Kam, Sungmin Song, et al.. (2018). TOM1 Regulates Neuronal Accumulation of Amyloid-β Oligomers by FcγRIIb2 Variant in Alzheimer's Disease. Journal of Neuroscience. 38(42). 9001–9018. 24 indexed citations

14.

Yun, Seung Pil, Donghoon Kim, Sangjune Kim, et al.. (2018). α-Synuclein accumulation and GBA deficiency due to L444P GBA mutation contributes to MPTP-induced parkinsonism. Molecular Neurodegeneration. 13(1). 1–1. 105 indexed citations

15.

Park, Hyejin, et al.. (2015). Essential role of POLDIP2 in Tau aggregation and neurotoxicity via autophagy/proteasome inhibition. Biochemical and Biophysical Research Communications. 462(2). 112–118. 21 indexed citations

16.

Choi, Hyunwoo, Won‐Jae Lee, Hyejin Park, et al.. (2015). Caspase-cleaved tau exhibits rapid memory impairment associated with tau oligomers in a transgenic mouse model. Neurobiology of Disease. 87. 19–28. 52 indexed citations

17.

Kam, Tae‐In, Youngdae Gwon, & Yong‐Keun Jung. (2014). Amyloid beta receptors responsible for neurotoxicity and cellular defects in Alzheimer’s disease. Cellular and Molecular Life Sciences. 71(24). 4803–4813. 47 indexed citations

18.

Park, Hyejin, Tae‐In Kam, Young-Doo Kim, et al.. (2012). Neuropathogenic role of adenylate kinase-1 in Aβ-mediated tau phosphorylation via AMPK and GSK3β. Human Molecular Genetics. 21(12). 2725–2737. 71 indexed citations

19.

Song, Sungmin, Huikyong Lee, Tae‐In Kam, et al.. (2008). E2-25K/Hip-2 regulates caspase-12 in ER stress–mediated Aβ neurotoxicity. The Journal of Cell Biology. 182(4). 675–684. 79 indexed citations

20.

Xie, Zhong, et al.. (1991). Studies on substances that induce long-term potentiation in guinea-pig hippocampal slices. Neuroscience. 43(1). 11–20. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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