Anne Grünewald

7.0k total citations · 3 hit papers
77 papers, 4.1k citations indexed

About

Anne Grünewald is a scholar working on Neurology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Anne Grünewald has authored 77 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Neurology, 38 papers in Molecular Biology and 22 papers in Cellular and Molecular Neuroscience. Recurrent topics in Anne Grünewald's work include Parkinson's Disease Mechanisms and Treatments (45 papers), Mitochondrial Function and Pathology (19 papers) and Autophagy in Disease and Therapy (14 papers). Anne Grünewald is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (45 papers), Mitochondrial Function and Pathology (19 papers) and Autophagy in Disease and Therapy (14 papers). Anne Grünewald collaborates with scholars based in Germany, Luxembourg and United Kingdom. Anne Grünewald's co-authors include Christine Klein, Carolyn M. Sue, Kishore R. Kumar, Aleksandar Raković, Sandro L. Pereira, Philip Seibler, Katja Lohmann, Semra Smajić, Alexander Skupin and Karolina A. Rygiel and has published in prestigious journals such as Science, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Anne Grünewald

75 papers receiving 4.1k citations

Hit Papers

Single-cell sequencing of human midbrain reveals glial... 2018 2026 2020 2023 2021 2018 2021 100 200 300

Peers

Anne Grünewald
Olga Corti France
Clemens R. Scherzer United States
Laurie H. Sanders United States
Amy K. Reeve United Kingdom
Olga Corti France
Anne Grünewald
Citations per year, relative to Anne Grünewald Anne Grünewald (= 1×) peers Olga Corti

Countries citing papers authored by Anne Grünewald

Since Specialization
Citations

This map shows the geographic impact of Anne Grünewald's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anne Grünewald with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anne Grünewald more than expected).

Fields of papers citing papers by Anne Grünewald

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anne Grünewald. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anne Grünewald. The network helps show where Anne Grünewald may publish in the future.

Co-authorship network of co-authors of Anne Grünewald

This figure shows the co-authorship network connecting the top 25 collaborators of Anne Grünewald. A scholar is included among the top collaborators of Anne Grünewald based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anne Grünewald. Anne Grünewald is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Žagare, Alise, Pierre Garcia, Jochen Ohnmacht, et al.. (2025). Parkinson’s disease mutant Miro1 causes mitochondrial dysfunction and dopaminergic neuron loss. Brain. 148(10). 3607–3622. 5 indexed citations
2.
Seibler, Philip, et al.. (2025). Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease. Journal of Neuroinflammation. 22(1). 136–136. 9 indexed citations
3.
Neumann, Katrin, Nathalie Legrave, François Bernardin, et al.. (2024). Metformin impacts the differentiation of mouse bone marrow cells into macrophages affecting tumour immunity. Heliyon. 10(18). e37792–e37792. 5 indexed citations
4.
Mulica, Patrycja, Carmen Venegas, Zied Landoulsi, et al.. (2023). Comparison of two protocols for the generation of iPSC-derived human astrocytes. Biological Procedures Online. 25(1). 26–26. 4 indexed citations
6.
König, Inke R., et al.. (2023). Lifestyle factors and clinical severity of Parkinson’s disease. Scientific Reports. 13(1). 9537–9537. 8 indexed citations
7.
Brunelli, F., Liliana Torosantucci, Vania Gelmetti, et al.. (2022). PINK1 Protects against Staurosporine-Induced Apoptosis by Interacting with Beclin1 and Impairing Its Pro-Apoptotic Cleavage. Cells. 11(4). 678–678. 16 indexed citations
8.
Smajić, Semra, Cesar A. Prada‐Medina, Zied Landoulsi, et al.. (2021). Single-cell sequencing of human midbrain reveals glial activation and a Parkinson-specific neuronal state. Brain. 145(3). 964–978. 306 indexed citations breakdown →
9.
Wasner, Kobi, Christine Klein, Susen Schaake, et al.. (2021). Nanopore Single-Molecule Sequencing for Mitochondrial DNA Methylation Analysis: Investigating Parkin-Associated Parkinsonism as a Proof of Concept. Frontiers in Aging Neuroscience. 13. 713084–713084. 18 indexed citations
10.
Delcambre, Sylvie, Jenny Ghelfi, Léa Grandmougin, et al.. (2020). Mitochondrial Mechanisms of LRRK2 G2019S Penetrance. Frontiers in Neurology. 11. 881–881. 22 indexed citations
11.
Großmann, Dajana, David Scheibner, François Massart, et al.. (2019). Mutations in RHOT1 Disrupt Endoplasmic Reticulum–Mitochondria Contact Sites Interfering with Calcium Homeostasis and Mitochondrial Dynamics in Parkinson's Disease. Antioxidants and Redox Signaling. 31(16). 1213–1234. 66 indexed citations
12.
Morais, Vanessa A., Dominik Haddad, Katleen Craessaerts, et al.. (2014). PINK1 Loss-of-Function Mutations Affect Mitochondrial Complex I Activity via NdufA10 Ubiquinone Uncoupling. Science. 344(6180). 203–207. 278 indexed citations
13.
Seibler, Philip, Diana Braunholz, Reinhard Depping, et al.. (2014). THAP1, the gene mutated in DYT6 dystonia, autoregulates its own expression. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1839(11). 1196–1204. 18 indexed citations
14.
Grünewald, Anne, et al.. (2013). Does Uncoupling Protein 2 Expression Qualify as Marker of Disease Status in LRRK2 -Associated Parkinson's Disease?. Antioxidants and Redox Signaling. 20(13). 1955–1960. 32 indexed citations
15.
Kumar, Kishore R., Nicholas Blair, Himesha Vandebona, et al.. (2013). Targeted next generation sequencing in SPAST-negative hereditary spastic paraplegia. Journal of Neurology. 260(10). 2516–2522. 33 indexed citations
16.
Raković, Aleksandar, Philip Seibler, Anne Grünewald, et al.. (2012). Phosphatase and Tensin Homolog (PTEN)-induced Putative Kinase 1 (PINK1)-dependent Ubiquitination of Endogenous Parkin Attenuates Mitophagy. Journal of Biological Chemistry. 288(4). 2223–2237. 175 indexed citations
17.
Grünewald, Anne, Philip Seibler, Aleksandar Raković, et al.. (2012). ATP13A2 mutations impair mitochondrial function in fibroblasts from patients with Kufor-Rakeb syndrome. Neurobiology of Aging. 33(8). 1843.e1–1843.e7. 113 indexed citations
18.
Raković, Aleksandar, Anne Grünewald, Jan Kottwitz, et al.. (2011). Mutations in PINK1 and Parkin Impair Ubiquitination of Mitofusins in Human Fibroblasts. PLoS ONE. 6(3). e16746–e16746. 189 indexed citations
19.
Grünewald, Anne, Aleksandar Raković, Meike Kasten, et al.. (2010). Mutant Parkin Impairs Mitochondrial Function and Morphology in Human Fibroblasts. PLoS ONE. 5(9). e12962–e12962. 122 indexed citations
20.
Grünewald, Anne, M. Gloor, Wolfgang Gehring, & Peter Kleesz. (1995). Damage to the skin by repetitive washing. Contact Dermatitis. 32(4). 225–232. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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