Matthew D. Smith

4.9k total citations · 2 hit papers
85 papers, 2.9k citations indexed

About

Matthew D. Smith is a scholar working on Molecular Biology, Neurology and Pathology and Forensic Medicine. According to data from OpenAlex, Matthew D. Smith has authored 85 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 26 papers in Neurology and 25 papers in Pathology and Forensic Medicine. Recurrent topics in Matthew D. Smith's work include Neuroinflammation and Neurodegeneration Mechanisms (25 papers), Multiple Sclerosis Research Studies (20 papers) and Monoclonal and Polyclonal Antibodies Research (15 papers). Matthew D. Smith is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (25 papers), Multiple Sclerosis Research Studies (20 papers) and Monoclonal and Polyclonal Antibodies Research (15 papers). Matthew D. Smith collaborates with scholars based in United States, United Kingdom and Germany. Matthew D. Smith's co-authors include Peter A. Calabresi, Jing Jin, Kathryn C. Fitzgerald, Pavan Bhargava, Peter M. Tessier, Daniel S. Reich, Marjan Gharagozloo, Ellen M. Mowry, Kyle Martin and Thomas Garton and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Matthew D. Smith

83 papers receiving 2.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A lymphocyte–microglia–astrocyte axis in chronic active multiple sclerosis

2021 445 citations Martina Absinta, Dragan Maric et al. Nature profile →
Oligodendrocyte precursor cells present antigen and are cytotoxic targets in inflammatory demyelination

2019 278 citations Leslie A. Kirby, Jing Jin et al. Nature Communications profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew D. Smith United States	31	1.1k	762	698	654	469	85	2.9k
Christopher Lock United States	19	1.4k 1.3×	493 0.6×	823 1.2×	1.6k 2.5×	185 0.4×	33	4.0k
Veerle Somers Belgium	38	719 0.7×	355 0.5×	824 1.2×	1.6k 2.4×	72 0.2×	100	3.4k
Ruth Lyck Switzerland	36	1.5k 1.4×	1.2k 1.5×	243 0.3×	937 1.4×	111 0.2×	54	3.7k
Salvatore Monaco Italy	42	2.2k 2.0×	1.3k 1.7×	1.2k 1.7×	390 0.6×	195 0.4×	205	5.8k
J E Merrill United States	19	719 0.7×	1.2k 1.6×	697 1.0×	1.5k 2.4×	301 0.6×	28	3.6k
Bo Höjeberg Sweden	31	605 0.5×	406 0.5×	748 1.1×	1.2k 1.8×	106 0.2×	52	2.8k
Michael A. Klein Switzerland	34	2.9k 2.6×	1.5k 2.0×	126 0.2×	425 0.6×	167 0.4×	83	4.3k
Antonio Iglesias Germany	33	1.9k 1.7×	334 0.4×	440 0.6×	2.5k 3.8×	85 0.2×	74	5.3k
Marc Fontaine France	38	1.1k 1.0×	913 1.2×	150 0.2×	1.9k 2.8×	127 0.3×	94	3.7k
Peter Adamson United Kingdom	43	2.9k 2.6×	762 1.0×	159 0.2×	946 1.4×	66 0.1×	99	5.8k

Countries citing papers authored by Matthew D. Smith

Since Specialization

Citations

This map shows the geographic impact of Matthew D. Smith's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew D. Smith with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew D. Smith more than expected).

Fields of papers citing papers by Matthew D. Smith

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew D. Smith. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew D. Smith. The network helps show where Matthew D. Smith may publish in the future.

Co-authorship network of co-authors of Matthew D. Smith

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew D. Smith. A scholar is included among the top collaborators of Matthew D. Smith based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew D. Smith. Matthew D. Smith is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Desai, Alec A., Jennifer M. Zupancic, Hanna Trzeciakiewicz, et al.. (2025). Facile generation of drug-like conformational antibodies specific for amyloid fibrils. Nature Chemical Biology. 21(6). 916–925. 1 indexed citations

Gharibani, Payam, Matthew D. Smith, Judy Lee, et al.. (2025). The protein kinase C modulator bryostatin-1 therapeutically targets microglia to attenuate neuroinflammation and promote remyelination. Science Translational Medicine. 17(780). eadk3434–eadk3434. 4 indexed citations

Anderson, Mary Ann, Renata Walewska, Arnon P. Kater, et al.. (2024). Venetoclax Initiation in Chronic Lymphocytic Leukemia: International Insights and Innovative Approaches for Optimal Patient Care. Cancers. 16(5). 980–980. 2 indexed citations

Smith, Matthew D., Thomas Garton, Alexis Allot, et al.. (2024). Ependymal cells undergo astrocyte‐like reactivity in response to neuroinflammation. Journal of Neurochemistry. 168(10). 3449–3466. 5 indexed citations

Pedrini, Edoardo, Maria Reyes‐Mantilla, Matthew D. Smith, et al.. (2024). Metabolomics of Multiple Sclerosis Lesions Demonstrates Lipid Changes Linked to Alterations in Transcriptomics-Based Cellular Profiles. Neurology Neuroimmunology & Neuroinflammation. 11(3). e200219–e200219. 11 indexed citations

Zhang, Yulei, Matthew D. Smith, Alec A. Desai, et al.. (2024). Human antibody polyreactivity is governed primarily by the heavy-chain complementarity-determining regions. Cell Reports. 43(10). 114801–114801. 4 indexed citations

Zhu, Xiaolei, Shinji Sakamoto, Chiharu Ishii, et al.. (2023). Dectin-1 signaling on colonic γδ T cells promotes psychosocial stress responses. Nature Immunology. 24(4). 625–636. 58 indexed citations

Harrington, Colleen T., et al.. (2023). MHC class I and MHC class II reporter mice enable analysis of immune oligodendroglia in mouse models of multiple sclerosis. eLife. 12. 17 indexed citations

Smith, Matthew D., Xitiz Chamling, Alexander J. Gill, et al.. (2022). Reactive Astrocytes Derived From Human Induced Pluripotent Stem Cells Suppress Oligodendrocyte Precursor Cell Differentiation. Frontiers in Molecular Neuroscience. 15. 874299–874299. 14 indexed citations

10.

Tan, Xiaotian, Jennifer M. Zupancic, John S. Schardt, et al.. (2022). Rapid and Quantitative In Vitro Evaluation of SARS-CoV-2 Neutralizing Antibodies and Nanobodies. Analytical Chemistry. 94(10). 4504–4512. 3 indexed citations

11.

Zupancic, Jennifer M., John S. Schardt, Alec A. Desai, et al.. (2021). Engineered Multivalent Nanobodies Potently and Broadly Neutralize SARS‐CoV‐2 Variants. Advanced Therapeutics. 4(8). 2100099–2100099. 30 indexed citations

12.

Absinta, Martina, Dragan Maric, Marjan Gharagozloo, et al.. (2021). A lymphocyte–microglia–astrocyte axis in chronic active multiple sclerosis. Nature. 597(7878). 709–714. 445 indexed citations breakdown →

13.

Parikh, Sameer A., Sara J. Achenbach, Timothy G. Call, et al.. (2020). The impact of dose modification and temporary interruption of ibrutinib on outcomes of chronic lymphocytic leukemia patients in routine clinical practice. Cancer Medicine. 9(10). 3390–3399. 39 indexed citations

14.

Kirby, Leslie A., Jing Jin, Jaime Gonzalez Cardona, et al.. (2019). Oligodendrocyte precursor cells present antigen and are cytotoxic targets in inflammatory demyelination. Nature Communications. 10(1). 3887–3887. 278 indexed citations breakdown →

15.

Hollinger, Kristen R., Matthew D. Smith, Leslie A. Kirby, et al.. (2019). Glutamine antagonism attenuates physical and cognitive deficits in a model of MS. Neurology Neuroimmunology & Neuroinflammation. 6(6). 17 indexed citations

16.

Cardona, Jaime Gonzalez, Matthew D. Smith, Leslie A. Kirby, et al.. (2019). Quetiapine has an additive effect to triiodothyronine in inducing differentiation of oligodendrocyte precursor cells through induction of cholesterol biosynthesis. PLoS ONE. 14(9). e0221747–e0221747. 11 indexed citations

17.

Gharagozloo, Marjan, Shaimaa Mahmoud, Camille Simard, et al.. (2019). NLRX1 inhibits the early stages of CNS inflammation and prevents the onset of spontaneous autoimmunity. PLoS Biology. 17(9). e3000451–e3000451. 25 indexed citations

18.

Smith, Matthew D., et al.. (2019). Directed evolution methods for overcoming trade‐offs between protein activity and stability. AIChE Journal. 66(3). 39 indexed citations

19.

Baxi, Emily G., Inna V. Grishkan, Matthew D. Smith, et al.. (2015). Transfer of Myelin-Reactive Th17 Cells Impairs Endogenous Remyelination in the Central Nervous System of Cuprizone-Fed Mice. Journal of Neuroscience. 35(22). 8626–8639. 81 indexed citations

20.

Patel, Mira C., Matthew D. Smith, Anup Dey, et al.. (2013). BRD4 Coordinates Recruitment of Pause Release Factor P-TEFb and the Pausing Complex NELF/DSIF To Regulate Transcription Elongation of Interferon-Stimulated Genes. Molecular and Cellular Biology. 33(12). 2497–2507. 136 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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