Sven Lindner

1.4k total citations
9 papers, 417 citations indexed

About

Sven Lindner is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Sven Lindner has authored 9 papers receiving a total of 417 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Neurology and 1 paper in Genetics. Recurrent topics in Sven Lindner's work include Neuroblastoma Research and Treatments (6 papers), Cell death mechanisms and regulation (2 papers) and Ubiquitin and proteasome pathways (2 papers). Sven Lindner is often cited by papers focused on Neuroblastoma Research and Treatments (6 papers), Cell death mechanisms and regulation (2 papers) and Ubiquitin and proteasome pathways (2 papers). Sven Lindner collaborates with scholars based in Germany, South Korea and Denmark. Sven Lindner's co-authors include Johannes H. Schulte, Angelika Eggert, Hedwig E. Deubzer, Andrea Odersky, Alexander Schramm, Christian Eckert, Ina Oehme, Marco Lodrini, Wilfried Roth and Olaf Witt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Human Molecular Genetics and International Journal of Cancer.

In The Last Decade

Sven Lindner

9 papers receiving 417 citations

Peers

Sven Lindner

MB

NEURO

CR

ONCOL

EPIDE

Peter J. Huwe United States

Ryo Sakasai Japan

Crystal G. Wheeler United States

Arash Nabbi Canada

Valentina Fodale Italy

Andrey V. Reshetnyak United States

Igor I. Doronin Russia

E Koeneke Germany

T. Crnogorac-Jurcevic United Kingdom

Ana Roscic Switzerland

Peter J. Huwe United States

Sven Lindner

225 ×0.7

MB

195 ×1.6

NEURO

105 ×1.2

CR

91 ×1.3

ONCOL

26 ×0.5

EPIDE

Citations per year, relative to Sven Lindner Sven Lindner (= 1×) peers Peter J. Huwe

Countries citing papers authored by Sven Lindner

Since Specialization

Citations

This map shows the geographic impact of Sven Lindner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sven Lindner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sven Lindner more than expected).

Fields of papers citing papers by Sven Lindner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sven Lindner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sven Lindner. The network helps show where Sven Lindner may publish in the future.

Co-authorship network of co-authors of Sven Lindner

This figure shows the co-authorship network connecting the top 25 collaborators of Sven Lindner. A scholar is included among the top collaborators of Sven Lindner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sven Lindner. Sven Lindner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Lindner, Sven, Yi Bei, Heathcliff Dorado García, et al.. (2019). Synergistic activity of BET inhibitor MK-8628 and PLK inhibitor Volasertib in preclinical models of medulloblastoma. Cancer Letters. 445. 24–33. 20 indexed citations

2.

Wefers, Annika K., Sven Lindner, Johannes H. Schulte, & Ulrich Schüller. (2016). Overexpression of Lin28b in Neural Stem Cells is Insufficient for Brain Tumor Formation, but Induces Pathological Lobulation of the Developing Cerebellum. The Cerebellum. 16(1). 122–131. 10 indexed citations

3.

Pajtler, Kristian W., Sandra Ackermann, Kristina Althoff, et al.. (2016). The GSK461364 PLK1 inhibitor exhibits strong antitumoral activity in preclinical neuroblastoma models. Oncotarget. 8(4). 6730–6741. 31 indexed citations

4.

Lindner, Sven, Hagen S. Bachmann, Andrea Odersky, et al.. (2015). Absence of telomerase reverse transcriptase promoter mutations in neuroblastoma. Biomedical Reports. 3(4). 443–446. 26 indexed citations

5.

Emdal, Kristina B., Anna-Kathrine Pedersen, Dorte B. Bekker‐Jensen, et al.. (2015). Temporal proteomics of NGF-TrkA signaling identifies an inhibitory role for the E3 ligase Cbl-b in neuroblastoma cell differentiation. Science Signaling. 8(374). ra40–ra40. 56 indexed citations

6.

Althoff, Kristina, Sven Lindner, Andrea Odersky, et al.. (2014). miR‐542‐3p exerts tumor suppressive functions in neuroblastoma by downregulating S urvivin. International Journal of Cancer. 136(6). 1308–1320. 68 indexed citations

7.

Dreidax, Daniel, Kai‐Oliver Henrich, Sebastian Bender, et al.. (2014). p19-INK4d inhibits neuroblastoma cell growth, induces differentiation and is hypermethylated and downregulated in MYCN-amplified neuroblastomas. Human Molecular Genetics. 23(25). 6826–6837. 20 indexed citations

8.

Pajtler, Kristian W., Andrea Odersky, Sven Lindner, et al.. (2014). Neuroblastoma in dialog with its stroma: NTRK1 is a regulator of cellular cross-talk with Schwann cells. Oncotarget. 5(22). 11180–11192. 21 indexed citations

9.

Oehme, Ina, Barbara C. Böck, Till Milde, et al.. (2013). Histone deacetylase 10 promotes autophagy-mediated cell survival. Proceedings of the National Academy of Sciences. 110(28). E2592–601. 165 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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