Kristina B. Emdal

1.0k total citations
15 papers, 606 citations indexed

About

Kristina B. Emdal is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Kristina B. Emdal has authored 15 papers receiving a total of 606 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Kristina B. Emdal's work include Cancer, Hypoxia, and Metabolism (3 papers), Advanced Proteomics Techniques and Applications (2 papers) and Neuroblastoma Research and Treatments (2 papers). Kristina B. Emdal is often cited by papers focused on Cancer, Hypoxia, and Metabolism (3 papers), Advanced Proteomics Techniques and Applications (2 papers) and Neuroblastoma Research and Treatments (2 papers). Kristina B. Emdal collaborates with scholars based in Denmark, United Kingdom and United States. Kristina B. Emdal's co-authors include Jesper V. Olsen, Dorte B. Bekker‐Jensen, Chiara Francavilla, Anne E. Lykkesfeldt, Christina W. Yde, Bárbara Guerra, Erik Vernet, Gianni Carraro, Blagoy Blagoev and Anna-Kathrine Pedersen and has published in prestigious journals such as Nature, Cell and Molecular Cell.

In The Last Decade

Kristina B. Emdal

15 papers receiving 605 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kristina B. Emdal Denmark 13 469 127 99 85 67 15 606
Katja Parapatics Austria 14 762 1.6× 195 1.5× 86 0.9× 33 0.4× 49 0.7× 18 962
Eugene Goufman Russia 10 695 1.5× 353 2.8× 129 1.3× 96 1.1× 48 0.7× 21 878
Yasuharu Hayashida Japan 6 255 0.5× 125 1.0× 72 0.7× 84 1.0× 61 0.9× 9 469
Elmar Bucher United States 10 514 1.1× 183 1.4× 205 2.1× 38 0.4× 79 1.2× 18 746
Joanne Berry United Kingdom 4 415 0.9× 100 0.8× 99 1.0× 103 1.2× 34 0.5× 4 516
Indranil Paul India 9 582 1.2× 165 1.3× 125 1.3× 42 0.5× 42 0.6× 12 679
Margaret Soucheray United States 6 491 1.0× 140 1.1× 57 0.6× 100 1.2× 94 1.4× 9 650
Linda Baskcomb United Kingdom 5 693 1.5× 208 1.6× 111 1.1× 24 0.3× 30 0.4× 5 772
Shih-Ya Wang United States 8 666 1.4× 203 1.6× 136 1.4× 24 0.3× 47 0.7× 9 751
Constance M. Cultraro United States 14 381 0.8× 166 1.3× 65 0.7× 24 0.3× 109 1.6× 30 510

Countries citing papers authored by Kristina B. Emdal

Since Specialization
Citations

This map shows the geographic impact of Kristina B. Emdal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kristina B. Emdal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kristina B. Emdal more than expected).

Fields of papers citing papers by Kristina B. Emdal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kristina B. Emdal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kristina B. Emdal. The network helps show where Kristina B. Emdal may publish in the future.

Co-authorship network of co-authors of Kristina B. Emdal

This figure shows the co-authorship network connecting the top 25 collaborators of Kristina B. Emdal. A scholar is included among the top collaborators of Kristina B. Emdal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kristina B. Emdal. Kristina B. Emdal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Emdal, Kristina B., Anna-Kathrine Pedersen, Lene Nygaard Axelsen, et al.. (2024). Multi-layered proteomics identifies insulin-induced upregulation of the EphA2 receptor via the ERK pathway which is dependent on low IGF1R level. Scientific Reports. 14(1). 28856–28856. 2 indexed citations
2.
Kverneland, Anders, Ole Østergaard, Kristina B. Emdal, Inge Marie Svane, & Jesper V. Olsen. (2022). Differential ultracentrifugation enables deep plasma proteomics through enrichment of extracellular vesicles. PROTEOMICS. 23(7-8). e2200039–e2200039. 19 indexed citations
3.
Emdal, Kristina B., Nicolàs Palacio‐Escat, Caroline Wigerup, et al.. (2022). Phosphoproteomics of primary AML patient samples reveals rationale for AKT combination therapy and p53 context to overcome selinexor resistance. Cell Reports. 40(6). 111177–111177. 21 indexed citations
4.
Dugourd, Aurélien, Christoph Kuppe, Marco Sciacovelli, et al.. (2021). Causal integration of multi‐omics data with prior knowledge to generate mechanistic hypotheses. Molecular Systems Biology. 17(1). e9730–e9730. 90 indexed citations
5.
Lundby, Alicia, Giulia Franciosa, Kristina B. Emdal, et al.. (2019). Oncogenic Mutations Rewire Signaling Pathways by Switching Protein Recruitment to Phosphotyrosine Sites. Cell. 179(2). 543–560.e26. 62 indexed citations
6.
Hamilton, William B., Yaron Mosesson, Kristina B. Emdal, et al.. (2019). Dynamic lineage priming is driven via direct enhancer regulation by ERK. Nature. 575(7782). 355–360. 49 indexed citations
7.
Emdal, Kristina B., Anna-Kathrine Pedersen, Dorte B. Bekker‐Jensen, et al.. (2018). Integrated proximal proteomics reveals IRS2 as a determinant of cell survival in ALK-driven neuroblastoma. Science Signaling. 11(557). 32 indexed citations
8.
Emdal, Kristina B., et al.. (2017). Characterization of In Vivo Resistance to Osimertinib and JNJ-61186372, an EGFR/Met Bispecific Antibody, Reveals Unique and Consensus Mechanisms of Resistance. Molecular Cancer Therapeutics. 16(11). 2572–2585. 23 indexed citations
9.
Liu, Hui, Charles J. Murphy, Florian A. Karreth, et al.. (2017). Identifying and Targeting Sporadic Oncogenic Genetic Aberrations in Mouse Models of Triple-Negative Breast Cancer. Cancer Discovery. 8(3). 354–369. 49 indexed citations
10.
Emdal, Kristina B., et al.. (2017). Characterization of in vivo resistance to osimertinib and JNJ-61186372, an EGFR/Met bi-specific antibody, reveals unique and consensus mechanisms of resistance. PMC. 1 indexed citations
11.
Radic-Sarikas, Branka, Kalliopi P. Tsafou, Kristina B. Emdal, et al.. (2016). Combinatorial Drug Screening Identifies Ewing Sarcoma–specific Sensitivities. Molecular Cancer Therapeutics. 16(1). 88–101. 16 indexed citations
12.
Emdal, Kristina B., Anna-Kathrine Pedersen, Dorte B. Bekker‐Jensen, et al.. (2015). Temporal proteomics of NGF-TrkA signaling identifies an inhibitory role for the E3 ligase Cbl-b in neuroblastoma cell differentiation. Science Signaling. 8(374). ra40–ra40. 56 indexed citations
13.
Francavilla, Chiara, Kristoffer Rigbolt, Kristina B. Emdal, et al.. (2013). Functional Proteomics Defines the Molecular Switch Underlying FGF Receptor Trafficking and Cellular Outputs. Molecular Cell. 51(6). 707–722. 111 indexed citations
14.
Yde, Christina W., Kristina B. Emdal, Bárbara Guerra, & Anne E. Lykkesfeldt. (2012). NFκB signaling is important for growth of antiestrogen resistant breast cancer cells. Breast Cancer Research and Treatment. 135(1). 67–78. 48 indexed citations
15.
Emdal, Kristina B., et al.. (2009). Breast cancer cells can switch between estrogen receptor α and ErbB signaling and combined treatment against both signaling pathways postpones development of resistance. Breast Cancer Research and Treatment. 121(3). 601–613. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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