Anne Hamacher‐Brady

13.9k total citations · 2 hit papers
36 papers, 4.3k citations indexed

About

Anne Hamacher‐Brady is a scholar working on Molecular Biology, Epidemiology and Physiology. According to data from OpenAlex, Anne Hamacher‐Brady has authored 36 papers receiving a total of 4.3k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 23 papers in Epidemiology and 6 papers in Physiology. Recurrent topics in Anne Hamacher‐Brady's work include Autophagy in Disease and Therapy (23 papers), Mitochondrial Function and Pathology (9 papers) and Cell death mechanisms and regulation (9 papers). Anne Hamacher‐Brady is often cited by papers focused on Autophagy in Disease and Therapy (23 papers), Mitochondrial Function and Pathology (9 papers) and Cell death mechanisms and regulation (9 papers). Anne Hamacher‐Brady collaborates with scholars based in United States, Germany and United Kingdom. Anne Hamacher‐Brady's co-authors include Nathan Brady, Roberta A. Gottlieb, Roland Eils, John Hazin, Nils Eling, Åsa B. Gustafsson, M. Richard Sayen, Lorrie A. Kirshenbaum, Susan E. Logue and Verena Lang and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The EMBO Journal.

In The Last Decade

Anne Hamacher‐Brady

35 papers receiving 4.2k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy

2006 523 citations Anne Hamacher‐Brady, Nathan Brady et al. Cell Death and Differentiation profile →
Identification of artesunate as a specific activator of ferroptosis in pancreatic cancer cells

2015 442 citations Nils Eling, John Hazin et al. Oncoscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Anne Hamacher‐Brady United States	28	2.5k	2.2k	532	508	434	36	4.3k
Nathan Brady Germany	32	3.1k 1.3×	3.0k 1.4×	617 1.2×	786 1.5×	451 1.0×	45	5.9k
Herbert J. Zeh United States	16	2.7k 1.1×	2.3k 1.1×	1.0k 1.9×	498 1.0×	195 0.4×	23	5.5k
Marco Corazzari Italy	35	2.7k 1.1×	2.7k 1.3×	589 1.1×	1.4k 2.7×	170 0.4×	87	5.6k
Shani Bialik Israel	28	3.0k 1.2×	2.1k 1.0×	516 1.0×	859 1.7×	298 0.7×	46	4.7k
Du Feng China	31	3.4k 1.4×	2.3k 1.1×	869 1.6×	797 1.6×	185 0.4×	88	5.1k
Einat Zalckvar Israel	23	3.2k 1.3×	2.8k 1.3×	409 0.8×	854 1.7×	164 0.4×	42	5.2k
Sudharsana Rao Ande Canada	28	2.4k 1.0×	933 0.4×	620 1.2×	506 1.0×	173 0.4×	55	4.3k
Nikolai Engedal Norway	25	1.8k 0.8×	1.5k 0.7×	336 0.6×	636 1.3×	98 0.2×	52	3.5k
Sophie Pattingre France	23	3.6k 1.5×	4.9k 2.3×	604 1.1×	1.5k 2.9×	248 0.6×	28	7.0k
Susan E. Logue Ireland	29	3.0k 1.2×	1.8k 0.8×	389 0.7×	2.4k 4.8×	375 0.9×	38	5.6k

Countries citing papers authored by Anne Hamacher‐Brady

Since Specialization

Citations

This map shows the geographic impact of Anne Hamacher‐Brady's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anne Hamacher‐Brady with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anne Hamacher‐Brady more than expected).

Fields of papers citing papers by Anne Hamacher‐Brady

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anne Hamacher‐Brady. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anne Hamacher‐Brady. The network helps show where Anne Hamacher‐Brady may publish in the future.

Co-authorship network of co-authors of Anne Hamacher‐Brady

This figure shows the co-authorship network connecting the top 25 collaborators of Anne Hamacher‐Brady. A scholar is included among the top collaborators of Anne Hamacher‐Brady based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anne Hamacher‐Brady. Anne Hamacher‐Brady is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Abraham, Rachy, Ivo A. Hendriks, Sara C. Buch-Larsen, et al.. (2025). Interferon-induced PARP14-mediated ADP-ribosylation in p62 bodies requires the ubiquitin-proteasome system. The EMBO Journal. 44(10). 2741–2773. 5 indexed citations

Wohlfarter, Yvonne, et al.. (2024). Stem cell models of TAFAZZIN deficiency reveal novel tissue-specific pathologies in Barth syndrome. Human Molecular Genetics. 34(1). 101–115. 4 indexed citations

Coppens, Isabelle, et al.. (2020). Endolysosomal Targeting of Mitochondria Is Integral to BAX-Mediated Mitochondrial Permeabilization during Apoptosis Signaling. Developmental Cell. 53(6). 627–645.e7. 35 indexed citations

Coelho, Carolina, Lisa Brown, Raghav Vij, et al.. (2018). Listeria monocytogenes virulence factors, including listeriolysin O, are secreted in biologically active extracellular vesicles. Journal of Biological Chemistry. 294(4). 1202–1217. 128 indexed citations

Ridinger, Johannes, E Koeneke, Fiona R. Kolbinger, et al.. (2018). Dual role of HDAC10 in lysosomal exocytosis and DNA repair promotes neuroblastoma chemoresistance. Scientific Reports. 8(1). 10039–10039. 39 indexed citations

Rogov, Vladimir V., H. Suzuki, Mija Marinković, et al.. (2017). Phosphorylation of the mitochondrial autophagy receptor Nix enhances its interaction with LC3 proteins. Scientific Reports. 7(1). 1131–1131. 234 indexed citations

Brady, Nathan, et al.. (2017). Agent-Based Modeling of Mitochondria Links Sub-Cellular Dynamics to Cellular Homeostasis and Heterogeneity. PLoS ONE. 12(1). e0168198–e0168198. 28 indexed citations

Beese, Carsten Jörn, et al.. (2016). Time course decomposition of cell heterogeneity in TFEB signaling states reveals homeostatic mechanisms restricting the magnitude and duration of TFEB responses to mTOR activity modulation. BMC Cancer. 16(1). 355–355. 10 indexed citations

Ugolino, Janet, Yon Ju Ji, Justin Chu, et al.. (2016). Loss of C9orf72 Enhances Autophagic Activity via Deregulated mTOR and TFEB Signaling. PLoS Genetics. 12(11). e1006443–e1006443. 141 indexed citations

10.

Metz, Philippe, Abhilash I. Chiramel, Laurent Chatel‐Chaix, et al.. (2015). Dengue Virus Inhibition of Autophagic Flux and Dependency of Viral Replication on Proteasomal Degradation of the Autophagy Receptor p62. Journal of Virology. 89(15). 8026–8041. 93 indexed citations

11.

Eling, Nils, et al.. (2015). Identification of artesunate as a specific activator of ferroptosis in pancreatic cancer cells. Oncoscience. 2(5). 517–532. 442 indexed citations breakdown →

12.

Hamacher‐Brady, Anne & Nathan Brady. (2015). Mitophagy programs: mechanisms and physiological implications of mitochondrial targeting by autophagy. Cellular and Molecular Life Sciences. 73(4). 775–795. 316 indexed citations

13.

Brady, Nathan & Anne Hamacher‐Brady. (2014). 546: Quantitative analysis of autophagic flux in pancreatic ductal adenocarcinoma (PDAC) by cellular heterogeneity measurements. European Journal of Cancer. 50. S132–S132. 1 indexed citations

14.

Zhai, Zongzhao, Nati Ha, Fani Papagiannouli, et al.. (2012). Antagonistic Regulation of Apoptosis and Differentiation by the Cut Transcription Factor Represents a Tumor-Suppressing Mechanism in Drosophila. PLoS Genetics. 8(3). e1002582–e1002582. 35 indexed citations

15.

Zhu, Yanyan, Marco Terenzio, Verena Lang, et al.. (2012). Modulation of Serines 17 and 24 in the LC3-interacting Region of Bnip3 Determines Pro-survival Mitophagy versus Apoptosis. Journal of Biological Chemistry. 288(2). 1099–1113. 376 indexed citations

16.

Hamacher‐Brady, Anne, Henning Stein, Rodrígo Mora, et al.. (2010). Artesunate Activates Mitochondrial Apoptosis in Breast Cancer Cells via Iron-catalyzed Lysosomal Reactive Oxygen Species Production. Journal of Biological Chemistry. 286(8). 6587–6601. 191 indexed citations

17.

Hamacher‐Brady, Anne, Nathan Brady, Susan E. Logue, et al.. (2006). Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy. Cell Death and Differentiation. 14(1). 146–157. 523 indexed citations breakdown →

18.

Brady, Nathan, Anne Hamacher‐Brady, & Roberta A. Gottlieb. (2006). Proapoptotic BCL-2 family members and mitochondrial dysfunction during ischemia/reperfusion injury, a study employing cardiac HL-1 cells and GFP biosensors. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1757(5-6). 667–678. 94 indexed citations

19.

Hamacher‐Brady, Anne, Nathan Brady, Roberta A. Gottlieb, & Åsa B. Gustafsson. (2006). Autophagy as a Protective Response to Bnip3-Mediated Apoptotic Signaling in the Heart. Autophagy. 2(4). 307–309. 91 indexed citations

20.

Hamacher‐Brady, Anne, Nathan Brady, & Roberta A. Gottlieb. (2006). Enhancing Macroautophagy Protects against Ischemia/Reperfusion Injury in Cardiac Myocytes. Journal of Biological Chemistry. 281(40). 29776–29787. 476 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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