Okiru Komine

2.6k total citations
35 papers, 1.7k citations indexed

About

Okiru Komine is a scholar working on Neurology, Neurology and Molecular Biology. According to data from OpenAlex, Okiru Komine has authored 35 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Neurology, 17 papers in Neurology and 12 papers in Molecular Biology. Recurrent topics in Okiru Komine's work include Amyotrophic Lateral Sclerosis Research (17 papers), Neuroinflammation and Neurodegeneration Mechanisms (16 papers) and Neurogenetic and Muscular Disorders Research (8 papers). Okiru Komine is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (17 papers), Neuroinflammation and Neurodegeneration Mechanisms (16 papers) and Neurogenetic and Muscular Disorders Research (8 papers). Okiru Komine collaborates with scholars based in Japan, United States and Switzerland. Okiru Komine's co-authors include Koji Yamanaka, Fumito Endo, Seiji Watanabe, Akira Sobue, Shijie Jin, Hiromi Tamada, Hiroshi Kiyama, Noriko Fujimori-Tonou, Masahisa Katsuno and Tomoo Ogi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and The EMBO Journal.

In The Last Decade

Okiru Komine

34 papers receiving 1.7k citations

Peers

Okiru Komine
Ikuko Mizuta Japan
Mark E. Hester United States
Ping-Wu Zhang United States
Karen Jansen‐West United States
Elize D. Haasdijk Netherlands
Silvia Coco Italy
Lydie Morel United States
Stefania Fasano Italy
Flavia Trettel Italy
Xian Lin China
Ikuko Mizuta Japan
Okiru Komine
Citations per year, relative to Okiru Komine Okiru Komine (= 1×) peers Ikuko Mizuta

Countries citing papers authored by Okiru Komine

Since Specialization
Citations

This map shows the geographic impact of Okiru Komine's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Okiru Komine with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Okiru Komine more than expected).

Fields of papers citing papers by Okiru Komine

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Okiru Komine. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Okiru Komine. The network helps show where Okiru Komine may publish in the future.

Co-authorship network of co-authors of Okiru Komine

This figure shows the co-authorship network connecting the top 25 collaborators of Okiru Komine. A scholar is included among the top collaborators of Okiru Komine based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Okiru Komine. Okiru Komine is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sobue, Akira, Okiru Komine, Yuko Saito, et al.. (2025). Long-term systemic androgen deprivation partially modulates neuroinflammation in male AppNL−G−F/NL−G−F mice. Scientific Reports. 15(1). 14702–14702.
2.
Fujisawa, Haruki, Takashi Watanabe, Okiru Komine, et al.. (2024). Prolonged extracellular low sodium concentrations and subsequent their rapid correction modulate nitric oxide production dependent on NFAT5 in microglia. Free Radical Biology and Medicine. 223. 458–472. 1 indexed citations
3.
Watanabe, Seiji, Okiru Komine, Eiki Takahashi, et al.. (2024). ALS-linked mutant TDP-43 in oligodendrocytes induces oligodendrocyte damage and exacerbates motor dysfunction in mice. Acta Neuropathologica Communications. 12(1). 184–184. 4 indexed citations
4.
Komine, Okiru, Kunihiko Hinohara, Yuichiro Hara, et al.. (2024). Genetic background variation impacts microglial heterogeneity and disease progression in amyotrophic lateral sclerosis model mice. iScience. 27(2). 108872–108872. 7 indexed citations
5.
Matsudaira, Tatsuyuki, Yusuke Konishi, Shimpei Kawamoto, et al.. (2023). Cellular senescence in white matter microglia is induced during ageing in mice and exacerbates the neuroinflammatory phenotype. Communications Biology. 6(1). 665–665. 43 indexed citations
6.
Sobue, Akira, Okiru Komine, & Koji Yamanaka. (2023). Neuroinflammation in Alzheimer’s disease: microglial signature and their relevance to disease. Inflammation and Regeneration. 43(1). 26–26. 64 indexed citations
7.
8.
Sobue, Akira, Okiru Komine, Yuichiro Hara, et al.. (2021). Microglial gene signature reveals loss of homeostatic microglia associated with neurodegeneration of Alzheimer’s disease. Acta Neuropathologica Communications. 9(1). 1–1. 151 indexed citations
9.
Konishi, Hiroyuki, Takayuki Okamoto, Yuichiro Hara, et al.. (2020). Astrocytic phagocytosis is a compensatory mechanism for microglial dysfunction. The EMBO Journal. 39(22). e104464–e104464. 131 indexed citations
10.
11.
Watanabe, Seiji, Okiru Komine, Akira Sobue, et al.. (2020). ALS-linked TDP-43M337V knock-in mice exhibit splicing deregulation without neurodegeneration. Molecular Brain. 13(1). 8–8. 42 indexed citations
12.
Watanabe, Seiji, Okiru Komine, Akira Sobue, et al.. (2020). Aggresome formation and liquid–liquid phase separation independently induce cytoplasmic aggregation of TAR DNA-binding protein 43. Cell Death and Disease. 11(10). 909–909. 41 indexed citations
13.
Sugiyama, Mariko, Ryoichi Banno, Akira Mizoguchi, et al.. (2020). Hypothalamic glial cells isolated by MACS reveal that microglia and astrocytes induce hypothalamic inflammation via different processes under high-fat diet conditions. Neurochemistry International. 136. 104733–104733. 18 indexed citations
14.
Komine, Okiru, Hirofumi Yamashita, Noriko Fujimori-Tonou, et al.. (2018). Innate immune adaptor TRIF deficiency accelerates disease progression of ALS mice with accumulation of aberrantly activated astrocytes. Cell Death and Differentiation. 25(12). 2130–2146. 48 indexed citations
15.
Yamanaka, Koji & Okiru Komine. (2017). The multi-dimensional roles of astrocytes in ALS. Neuroscience Research. 126. 31–38. 153 indexed citations
16.
Watanabe, Seiji, Hristelina Ilieva, Hiromi Tamada, et al.. (2016). Mitochondria‐associated membrane collapse is a common pathomechanism in SIGMAR 1 ‐ and SOD 1 ‐linked ALS. EMBO Molecular Medicine. 8(12). 1421–1437. 201 indexed citations
17.
Lašienė, Jūratė, Okiru Komine, Noriko Fujimori-Tonou, et al.. (2016). Neuregulin 1 confers neuroprotection in SOD1-linked amyotrophic lateral sclerosis mice via restoration of C-boutons of spinal motor neurons. Acta Neuropathologica Communications. 4(1). 15–15. 46 indexed citations
18.
Watanabe, Seiji, Natsumi Ageta‐Ishihara, Keizo Takao, et al.. (2014). SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system. Molecular Brain. 7(1). 62–62. 80 indexed citations
19.
Komine, Okiru, Mai Nagaoka, Yuichi Hiraoka, et al.. (2011). RBP-J promotes the maturation of neuronal progenitors. Developmental Biology. 354(1). 44–54. 9 indexed citations
20.
Takayasu, Yukihiro, Masae Iino, Wataru Kakegawa, et al.. (2005). Differential Roles of Glial and Neuronal Glutamate Transporters in Purkinje Cell Synapses. Journal of Neuroscience. 25(38). 8788–8793. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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