Philip J. Horner

7.9k total citations · 2 hit papers
96 papers, 6.4k citations indexed

About

Philip J. Horner is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Pathology and Forensic Medicine. According to data from OpenAlex, Philip J. Horner has authored 96 papers receiving a total of 6.4k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Molecular Biology, 30 papers in Cellular and Molecular Neuroscience and 27 papers in Pathology and Forensic Medicine. Recurrent topics in Philip J. Horner's work include Spinal Cord Injury Research (27 papers), Nerve injury and regeneration (20 papers) and Neurogenesis and neuroplasticity mechanisms (18 papers). Philip J. Horner is often cited by papers focused on Spinal Cord Injury Research (27 papers), Nerve injury and regeneration (20 papers) and Neurogenesis and neuroplasticity mechanisms (18 papers). Philip J. Horner collaborates with scholars based in United States, Italy and Canada. Philip J. Horner's co-authors include Fred H. Gage, Denise M. Inman, David J. Calkins, Suzie H. Pun, Monica L. Vetter, Michael R. Steele, Drew L. Sellers, Samuel D. Crish, Rebecca M. Sappington and Jūratė Lašienė and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Neuroscience.

In The Last Decade

Philip J. Horner

94 papers receiving 6.3k citations

Hit Papers

Regenerating the damaged central nervous system 2000 2026 2008 2017 2000 2000 200 400 600

Peers

Philip J. Horner
Comparison fields: 5 of 132
  • Molecular Biology 2.9k
  • Cellular and Molecular Neuroscience 2.0k
  • Ophthalmology 1.7k
  • Developmental Neuroscience 1.4k
  • Pathology and Forensic Medicine 1.2k
Replace Solon Thanos with:
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Citations per field, relative to Philip J. Horner
Philip J. Horner · 1×
Citations per year, relative to Philip J. Horner
Philip J. Horner · 1×

Countries citing papers authored by Philip J. Horner

Since Specialization
Citations

This map shows the geographic impact of Philip J. Horner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip J. Horner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip J. Horner more than expected).

Fields of papers citing papers by Philip J. Horner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip J. Horner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip J. Horner. The network helps show where Philip J. Horner may publish in the future.

Co-authorship network of co-authors of Philip J. Horner

This figure shows the co-authorship network connecting the top 25 collaborators of Philip J. Horner. A scholar is included among the top collaborators of Philip J. Horner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip J. Horner. Philip J. Horner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 1
2 2
3 4
4 0
5 5
6 1
7 4
8 4
9 17
10 46
11
NFkB signaling in retinal glia mediates progressive neural degeneration and vision decline in glaucoma
2
12 11
13
Potential Role of Retinal Microglia in RGC Synapse Removal in DBA/2J Mice
1
14
High IOP Decreases Compound Action Potential Amplitude and Leads to Preferential Loss of Physiologically Identified Small Caliber Axons in DBA/2 Optic Nerve
1
15 65
16
Dietary Lipoic Acid Attenuates Oxidative Stress and Retinal Ganglion Cell Loss in the DBA/2J Mouse Model of Glaucoma
2
17 173
18 211
19
Whole Retinal Microarray Analysis of DBA/2J Mice: A Model for Glaucoma
2
20
Retinal Vessel Volume and Pericyte Number Decrease Over Time in the DBA/2 Mouse Model of Glaucoma
4

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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