Maya Saleh

7.5k citations
80 papers · 6.0k indexed · h-index 41
  • Immunology top 0.5%
    • Immune Response and Inflammation 14
    • interferon and immune responses 11
    • Immune Cell Function and Interaction 9
    • IL-33, ST2, and ILC Pathways 6
    • Immune cells in cancer 6
    • Inflammasome and immune disorders 37
    • Cell death mechanisms and regulation 20
    • Heme Oxygenase-1 and Carbon Monoxide 6

Maya Saleh

79 papers receiving 5.9k citations

Peers

Maya Saleh
Comparison fields: 5 of 128
  • Immunology 2.6k
  • Molecular Biology 3.9k
  • Cancer Research 515
  • Epidemiology 918
  • Biological Psychiatry 63
Replace Joseph A. Duncan with:
Joseph A. Duncan United States
Ricardo Weinlich Brazil
Beckley K. Davis United States
Jennifer Martinez United States
Thomas Weichhart Austria
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Pinaki Datta United States
Prajwal Gurung United States
Shruti Sharma United States
Chris Haslett United Kingdom
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Citations per field
00.5×1.5×
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Citations per year

Countries citing papers authored by Maya Saleh

Since Specialization
Citations

This map shows the geographic impact of Maya Saleh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maya Saleh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maya Saleh more than expected).

Fields of papers citing papers by Maya Saleh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maya Saleh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maya Saleh. The network helps show where Maya Saleh may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Maya Saleh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Maya Saleh Line = papers co-authored together Maya Saleh links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202116
2 202146
3 202021
4 20190
5 201820
6 201749
7 201519
8 201516
9 2014132
10 201457
11 201321
12 201192
13 2011126
14 2010431
15 20104
16 200948
17 200894
18 200840
19 200674
20 2000166

About Maya Saleh

Maya Saleh is a scholar working on Immunology, Molecular Biology and Endocrinology, having authored 80 papers that have together received 6.0k indexed citations. Recurring topics across this work include Inflammasome and immune disorders (37 papers), Cell death mechanisms and regulation (20 papers), Immune Response and Inflammation (14 papers), interferon and immune responses (11 papers), Immune Cell Function and Interaction (9 papers), IL-33, ST2, and ILC Pathways (6 papers), Heme Oxygenase-1 and Carbon Monoxide (6 papers) and Immune cells in cancer (6 papers). The work is most often cited by research in Immunology (2.6k citations), Molecular Biology (3.9k citations) and Cancer Research (515 citations). Maya Saleh has collaborated with scholars based in Canada, United States and France. Frequent co-authors include Katherine Labbè, Karine Doiron, Giorgio Trinchieri, Garabet Yeretssian, Maryse Dagenais, Todd Douglas, Jeremy Dupaul-Chicoine, Philippe M. LeBlanc, Douglas R. Green and Alexander Skeldon. Their work appears in journals such as Nature, Science and Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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