Luis Carlos Tábara
- Molecular Biology
- Epidemiology
- Cell Biology top 10%
- Physiology
- Clinical Biochemistry top 5%
- Co-authors
- Julien PrudentRicardo EscalanteMayuko SegawaHanish AnandVincent PaupeLisa TilokaniAlberto OrtízRodolfo Zunino
- Topics
- Mitochondrial Function and Pathology (8 papers)Endoplasmic Reticulum Stress and Disease (6 papers)ATP Synthase and ATPases Research (5 papers)
- Journals
- ScienceCellNucleic Acids Research
- Partner nations
- United KingdomSpainUnited States
In The Last Decade
Luis Carlos Tábara
16 papers receiving 735 citations
Hit Papers
Peers
Comparison fields: 5 of 76
- Molecular Biology 467
- Epidemiology 196
- Cell Biology 168
- Physiology 96
- Clinical Biochemistry 87
Countries citing papers authored by Luis Carlos Tábara
This map shows the geographic impact of Luis Carlos Tábara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luis Carlos Tábara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luis Carlos Tábara more than expected).
Fields of papers citing papers by Luis Carlos Tábara
This network shows the impact of papers produced by Luis Carlos Tábara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luis Carlos Tábara. The network helps show where Luis Carlos Tábara may publish in the future.
Co-authorship network of co-authors of Luis Carlos Tábara
This figure shows the co-authorship network connecting the top 25 collaborators of Luis Carlos Tábara. A scholar is included among the top collaborators of Luis Carlos Tábara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Luis Carlos Tábara. Luis Carlos Tábara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | MTFP1 controls mitochondrial fusion to regulate inner membrane quality control and maintain mtDNA levelsbreakdown → | 51 |
| 2 | 2 | |
| 3 | Molecular mechanisms of mitochondrial dynamicsbreakdown → | 103 |
| 4 | 2 | |
| 5 | 3 | |
| 6 | 40 | |
| 7 | 8 | |
| 8 | 48 | |
| 9 | 173 | |
| 10 | 13 | |
| 11 | 18 | |
| 12 | 72 | |
| 13 | 61 | |
| 14 | 17 | |
| 15 | 76 | |
| 16 | 50 |
About Luis Carlos Tábara
Luis Carlos Tábara is a scholar working on Clinical Biochemistry, Cell Biology and Biochemistry, having authored 16 papers that have together received 737 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (8 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and ATP Synthase and ATPases Research (5 papers). The work is most often cited by research in Clinical Biochemistry (87 citations), Cell Biology (168 citations) and Nephrology (71 citations). Luis Carlos Tábara has collaborated with scholars based in United Kingdom, Spain and United States. Frequent co-authors include Julien Prudent, Ricardo Escalante, Mayuko Segawa, Hanish Anand, Vincent Paupe, Lisa Tilokani, Alberto Ortíz, Rodolfo Zunino, María Dolores Sánchez-Niño and Catalina Martín-Cleary. Their work appears in journals such as Science, Cell and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.