Ricardo Escalante
- Molecular Biology top 10%
- Cell Biology top 1%
- Epidemiology top 5%
- Physiology top 10%
- Genetics top 10%
- Co-authors
- Leandro SastreWilliam F. LoomisJuan Jesus VicenteJavier Calvo‐GarridoGad ShaulskyMaría Galardi‐CastillaSergio Carilla-LatorreLuis Carlos Tábara
- Topics
- Autophagy in Disease and Therapy (22 papers)Cellular Mechanics and Interactions (21 papers)Endoplasmic Reticulum Stress and Disease (19 papers)
- Cited by
- Cell BiologyAgingEndocrinology
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryThe EMBO Journal
- Partner nations
- SpainUnited StatesUnited Kingdom
In The Last Decade
Ricardo Escalante
77 papers receiving 2.2k citations
Peers
Comparison fields: 5 of 117
- Molecular Biology 1.1k
- Cell Biology 867
- Epidemiology 491
- Physiology 219
- Genetics 190
Countries citing papers authored by Ricardo Escalante
This map shows the geographic impact of Ricardo Escalante's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ricardo Escalante with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ricardo Escalante more than expected).
Fields of papers citing papers by Ricardo Escalante
This network shows the impact of papers produced by Ricardo Escalante. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ricardo Escalante. The network helps show where Ricardo Escalante may publish in the future.
Co-authorship network of co-authors of Ricardo Escalante
This figure shows the co-authorship network connecting the top 25 collaborators of Ricardo Escalante. A scholar is included among the top collaborators of Ricardo Escalante based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ricardo Escalante. Ricardo Escalante is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 4 | |
| 3 | 3 | |
| 4 | 8 | |
| 5 | 13 | |
| 6 | 18 | |
| 7 | 36 | |
| 8 | 72 | |
| 9 | 17 | |
| 10 | 41 | |
| 11 | 13 | |
| 12 | 122 | |
| 13 | 58 | |
| 14 | 19 | |
| 15 | 9 | |
| 16 | 13 | |
| 17 | 29 | |
| 18 | 54 | |
| 19 | 19 | |
| 20 | 6 |
About Ricardo Escalante
Ricardo Escalante is a scholar working on Cell Biology, Aging and Endocrinology, having authored 79 papers that have together received 2.2k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (22 papers), Cellular Mechanics and Interactions (21 papers) and Endoplasmic Reticulum Stress and Disease (19 papers). The work is most often cited by research in Cell Biology (867 citations), Aging (76 citations) and Endocrinology (162 citations). Ricardo Escalante has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Leandro Sastre, William F. Loomis, Juan Jesus Vicente, Javier Calvo‐Garrido, Gad Shaulsky, María Galardi‐Castilla, Sergio Carilla-Latorre, Luis Carlos Tábara, Olivier Vincent and Rosa Calvo. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.