Lisa Duvick

6.2k total citations · 1 hit paper
37 papers, 4.6k citations indexed

About

Lisa Duvick is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Lisa Duvick has authored 37 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 31 papers in Cellular and Molecular Neuroscience and 5 papers in Genetics. Recurrent topics in Lisa Duvick's work include Genetic Neurodegenerative Diseases (31 papers), Mitochondrial Function and Pathology (28 papers) and DNA Repair Mechanisms (8 papers). Lisa Duvick is often cited by papers focused on Genetic Neurodegenerative Diseases (31 papers), Mitochondrial Function and Pathology (28 papers) and DNA Repair Mechanisms (8 papers). Lisa Duvick collaborates with scholars based in United States, Poland and Russia. Lisa Duvick's co-authors include Harry T. Orr, Huda Y. Zoghbi, Antonio Servadio, Ming‐Yi Chung, Laura P.W. Ranum, Sandro Banfi, Alanna E. McCall, Thomas J. Kwiatkowski, Arthur L. Beaudet and Eric N. Burright and has published in prestigious journals such as Cell, Nucleic Acids Research and Nature Communications.

In The Last Decade

Lisa Duvick

36 papers receiving 4.5k citations

Hit Papers

Expansion of an unstable trinucleotide CAG repeat in spin... 1993 2026 2004 2015 1993 400 800 1.2k

Peers

Lisa Duvick
Alanna E. McCall United States
Lisa Duvick
Citations per year, relative to Lisa Duvick Lisa Duvick (= 1×) peers Alanna E. McCall

Countries citing papers authored by Lisa Duvick

Since Specialization
Citations

This map shows the geographic impact of Lisa Duvick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lisa Duvick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lisa Duvick more than expected).

Fields of papers citing papers by Lisa Duvick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lisa Duvick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lisa Duvick. The network helps show where Lisa Duvick may publish in the future.

Co-authorship network of co-authors of Lisa Duvick

This figure shows the co-authorship network connecting the top 25 collaborators of Lisa Duvick. A scholar is included among the top collaborators of Lisa Duvick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lisa Duvick. Lisa Duvick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Báñez-Coronel, Mónica, Tao Zu, Shu Guo, et al.. (2025). Repeat-associated non-AUG translation as a common mechanism for the polyGln ataxias. Cell Reports. 45(1). 116741–116741.
2.
Sheeler, Carrie, Lisa Duvick, Leslie M. Thompson, et al.. (2024). Expanded ATXN1 alters transcription and calcium signaling in SCA1 human motor neurons differentiated from induced pluripotent stem cells. Neurobiology of Disease. 201. 106673–106673. 2 indexed citations
3.
O’Callaghan, Brennon, Holly Kordasiewicz, Tracy Cole, et al.. (2020). Antisense Oligonucleotide Therapeutic Approach for Suppression of Ataxin-1 Expression: A Safety Assessment. Molecular Therapy — Nucleic Acids. 21. 1006–1016. 16 indexed citations
4.
Williamson, Nicholas A., Lisa Duvick, Alexander Lee, et al.. (2020). The ataxin-1 interactome reveals direct connection with multiple disrupted nuclear transport pathways. Nature Communications. 11(1). 3343–3343. 16 indexed citations
5.
Kordasiewicz, Holly, Brennon O’Callaghan, Lisa Duvick, et al.. (2018). Antisense oligonucleotide–mediated ataxin-1 reduction prolongs survival in SCA1 mice and reveals disease-associated transcriptome profiles. JCI Insight. 3(21). 87 indexed citations
6.
Ortiz, Judit Perez, Nicholas Toker, Carolyn J. Adamski, et al.. (2018). Reduction of protein kinase A-mediated phosphorylation of ATXN1-S776 in Purkinje cells delays onset of Ataxia in a SCA1 mouse model. Neurobiology of Disease. 116. 93–105. 27 indexed citations
7.
Ingram, Melissa, Justin Barnes, Lisa Duvick, et al.. (2013). Purkinje Cell Ataxin-1 Modulates Climbing Fiber Synaptic Input in Developing and Adult Mouse Cerebellum. Journal of Neuroscience. 33(13). 5806–5820. 45 indexed citations
8.
Andresen, J. Michael, et al.. (2011). Partial loss of Tip60 slows mid-stage neurodegeneration in a spinocerebellar ataxia type 1 (SCA1) mouse model. Human Molecular Genetics. 20(11). 2204–2212. 46 indexed citations
9.
Duvick, Lisa, Justin Barnes, Smita Agrawal, et al.. (2010). SCA1-like Disease in Mice Expressing Wild-Type Ataxin-1 with a Serine to Aspartic Acid Replacement at Residue 776. Neuron. 67(6). 929–935. 116 indexed citations
10.
Andresen, J. Michael, Courtney Byam, Lisa Duvick, et al.. (2009). Phosphorylation of ATXN1 at Ser776 in the cerebellum. Journal of Neurochemistry. 110(2). 675–686. 45 indexed citations
11.
Davidson, Jack D., Brien P. Riley, Eric N. Burright, et al.. (2000). Identification and characterization of an ataxin-1-interacting protein: A1Up, a ubiquitin-like nuclear protein. Human Molecular Genetics. 9(15). 2305–2312. 78 indexed citations
12.
Kaytor, Michael D., Eric N. Burright, Lisa Duvick, Huda Y. Zoghbi, & Harry T. Orr. (1997). Increased Trinucleotide Repeat Instability with Advanced Maternal Age. Human Molecular Genetics. 6(12). 2135–2139. 76 indexed citations
13.
Burright, Eric N., H. Brent Clark, Antonio Servadio, et al.. (1995). SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeat. Cell. 82(6). 937–948. 469 indexed citations
14.
Banfi, Sandro, Antonio Servadio, Ming‐Yi Chung, et al.. (1994). Identification and characterization of the gene causing type 1 spinocerebellar ataxia. Nature Genetics. 7(4). 513–520. 278 indexed citations
15.
Orr, Harry T., Ming‐Yi Chung, Sandro Banfi, et al.. (1993). Expansion of an unstable trinucleotide CAG repeat in spinocerebellar ataxia type 1. Nature Genetics. 4(3). 221–226. 1303 indexed citations breakdown →
16.
Chung, Ming‐Yi, Laura P.W. Ranum, Lisa Duvick, et al.. (1993). Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type I. Nature Genetics. 5(3). 254–258. 401 indexed citations
17.
Schmidt, Cynthia, Μαρία Αθανασίου, Lisa Duvick, et al.. (1993). Extraembryonic expression of the human MHC class I gene HLA-G in transgenic mice. Evidence for a positive regulatory region located 1 kilobase 5' to the start site of transcription.. The Journal of Immunology. 151(5). 2633–2645. 57 indexed citations
18.
Ranum, Laura P.W., Ming‐Yi Chung, Lisa Duvick, Huda Y. Zoghbi, & Harry T. Orr. (1991). Dinucleotide repeat polymorphism at the D6S109 locus. Nucleic Acids Research. 19(5). 1171–1171. 20 indexed citations
19.
Duvick, Lisa, Stephen S. Rich, & Harry T. Orr. (1990). A polymorphic DNA probe, p1–10–2, from chromosome 6. Nucleic Acids Research. 18(10). 3105–3105. 2 indexed citations
20.
Koller, Beverly H., Daniel E. Geraghty, Robert DeMars, et al.. (1989). Chromosomal organization of the human major histocompatibility complex class I gene family.. The Journal of Experimental Medicine. 169(2). 469–480. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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