Klaus‐Armin Nave
- Molecular Biology top 0.1%
- Cellular and Molecular Neuroscience top 0.02%
- Developmental Neuroscience top 0.01%
- Neurology top 0.02%
- Physiology top 0.5%
- Co-authors
- Bruce D. TrappSandra GoebbelsHauke WernerWiebke MöbiusMikael SimonsMarkus H. SchwabIan R. GriffithsMichael W. Sereda
- Topics
- Neurogenesis and neuroplasticity mechanisms (111 papers)Hereditary Neurological Disorders (62 papers)Neuroinflammation and Neurodegeneration Mechanisms (58 papers)
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Klaus‐Armin Nave
293 papers receiving 31.8k citations
Hit Papers
Peers
Comparison fields: 5 of 176
- Molecular Biology 15.0k
- Cellular and Molecular Neuroscience 11.4k
- Developmental Neuroscience 9.9k
- Neurology 6.3k
- Physiology 3.4k
Countries citing papers authored by Klaus‐Armin Nave
This map shows the geographic impact of Klaus‐Armin Nave's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus‐Armin Nave with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus‐Armin Nave more than expected).
Fields of papers citing papers by Klaus‐Armin Nave
This network shows the impact of papers produced by Klaus‐Armin Nave. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus‐Armin Nave. The network helps show where Klaus‐Armin Nave may publish in the future.
Co-authorship network of co-authors of Klaus‐Armin Nave
This figure shows the co-authorship network connecting the top 25 collaborators of Klaus‐Armin Nave. A scholar is included among the top collaborators of Klaus‐Armin Nave based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klaus‐Armin Nave. Klaus‐Armin Nave is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 5 | |
| 3 | 1 | |
| 4 | 16 | |
| 5 | 8 | |
| 6 | 6 | |
| 7 | 55 | |
| 8 | 35 | |
| 9 | 11 | |
| 10 | 14 | |
| 11 | 16 | |
| 12 | 19 | |
| 13 | 79 | |
| 14 | 25 | |
| 15 | 106 | |
| 16 | 249 | |
| 17 | 175 | |
| 18 | 34 | |
| 19 | 87 | |
| 20 | 89 |
About Klaus‐Armin Nave
Klaus‐Armin Nave is a scholar working on Developmental Neuroscience, Neurology and Cellular and Molecular Neuroscience, having authored 296 papers that have together received 32.1k indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (111 papers), Hereditary Neurological Disorders (62 papers) and Neuroinflammation and Neurodegeneration Mechanisms (58 papers). The work is most often cited by research in Developmental Neuroscience (9.9k citations), Neurology (6.3k citations) and Cellular and Molecular Neuroscience (11.4k citations). Klaus‐Armin Nave has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Bruce D. Trapp, Sandra Goebbels, Hauke Werner, Wiebke Möbius, Mikael Simons, Markus H. Schwab, Ian R. Griffiths, Michael W. Sereda, Aiman S. Saab and Carmen Birchmeier. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.