Keqiang Ye
Impact in
- Biological Psychiatry top 0.5%
- Developmental Neuroscience top 0.2%
- Neurogenesis and neuroplasticity mechanisms
Papers in
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- Neurogenesis and neuroplasticity mechanisms 23
-
- Nerve injury and regeneration 38
- Nuclear Receptors and Signaling 25
- Co-authors
- Xia LiuSeong Su KangSung‐Wuk JangZhentao ZhangChi Bun ChanSolomon H. SnyderKun‐Liang GuanJoungmok Kim
- Journals
- Proceedings of the National Academy of Sciences (35 papers)Journal of Biological Chemistry (14 papers)Molecular Psychiatry (9 papers)The EMBO Journal (8 papers)Progress in Neurobiology (7 papers)
- Partner nations
- United StatesChinaSouth Korea
In The Last Decade
Keqiang Ye
237 papers receiving 17.0k citations
Hit Papers
Peers
Comparison fields: 5 of 153
- Biological Psychiatry 775
- Developmental Neuroscience 1.1k
- Cell Biology 3.8k
- Cellular and Molecular Neuroscience 3.8k
- Neurology 1.3k
Countries citing papers authored by Keqiang Ye
This map shows the geographic impact of Keqiang Ye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keqiang Ye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keqiang Ye more than expected).
Fields of papers citing papers by Keqiang Ye
This network shows the impact of papers produced by Keqiang Ye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keqiang Ye. The network helps show where Keqiang Ye may publish in the future.
Co-authors
The 25 scholars most cited alongside Keqiang Ye, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | Antiageing strategy for neurodegenerative diseases: from mechanisms to clinical advances Hit paper breakdown → | 2025 | 36 |
| 3 | Propagation of pathologic α-synuclein from kidney to brain may contribute to Parkinson’s disease Hit paper breakdown → | 2025 | 19 |
| 4 | 2025 | 1 | |
| 5 | 2024 | 14 | |
| 6 | 2024 | 2 | |
| 7 | 2023 | 1 | |
| 8 | 2022 | 18 | |
| 9 | 2021 | 14 | |
| 10 | 2021 | 24 | |
| 11 | 2020 | 48 | |
| 12 | 2019 | 54 | |
| 13 | 2019 | 12 | |
| 14 | 2019 | 27 | |
| 15 | 2016 | 136 | |
| 16 | 2016 | 135 | |
| 17 | Excess PI3K subunit synthesis and activity as a novel therapeutic target in Fragile X Syndrome | 2010 | 1 |
| 18 | 2010 | 197 | |
| 19 | 2009 | 26 | |
| 20 | 2008 | 82 |
About Keqiang Ye
Keqiang Ye is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience, Cell Biology, Physiology and Neurology, having authored 244 papers that have together received 17.1k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (59 papers), Nerve injury and regeneration (38 papers), PI3K/AKT/mTOR signaling in cancer (30 papers), Parkinson's Disease Mechanisms and Treatments (26 papers), Nuclear Receptors and Signaling (25 papers), Protein Kinase Regulation and GTPase Signaling (23 papers), Neurogenesis and neuroplasticity mechanisms (23 papers) and Peptidase Inhibition and Analysis (23 papers). The work is most often cited by research in Biological Psychiatry (775 citations), Developmental Neuroscience (1.1k citations), Cell Biology (3.8k citations), Cellular and Molecular Neuroscience (3.8k citations) and Neurology (1.3k citations). Keqiang Ye has collaborated with scholars based in United States, China and South Korea. Frequent co-authors include Xia Liu, Seong Su Kang, Sung‐Wuk Jang, Zhentao Zhang, Chi Bun Chan, Solomon H. Snyder, Kun‐Liang Guan, Joungmok Kim, Qian Yang and Georg Halder. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry, Molecular Psychiatry, The EMBO Journal and Progress in Neurobiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.