Benjamin T. Kile

14.1k citations

137 papers · 8.3k indexed · 4 hit papers · h-index 50

Molecular Biology top 1%
Immunology top 0.5%
Hematology top 0.5%
Oncology top 2%
Cancer Research top 2%

Co-authors: Warren S. Alexander Donald Metcalf Douglas J. Hilton David C.S. Huang Kate McArthur M. J. D. White Nicos A. Nicola Emma C. Josefsson
Topics: Platelet Disorders and Treatments (41 papers)Cell death mechanisms and regulation (19 papers)interferon and immune responses (16 papers)
Cited by: Hematology Immunology Molecular Biology
Journals: Nature Cell Proceedings of the National Academy of Sciences
Partner nations: Australia United States United Kingdom

In The Last Decade

Benjamin T. Kile

137 papers receiving 8.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Programmed Anuclear Cell Death Delimits Platelet Life Span

2007 767 citations Kylie D. Mason, Marina R. Carpinelli et al. Cell profile →
Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

2014 741 citations M. J. D. White, Kate McArthur et al. Cell profile →
The conserved SOCS box motif in suppressors of cytokine signaling binds to elongins B and C and may couple bound proteins to proteasomal degradation

1999 530 citations Benjamin T. Kile, Warren S. Alexander et al. profile →
TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells

2020 325 citations Katherine R. Balka, Cynthia Louis et al. Cell Reports profile →

Peers

Countries citing papers authored by Benjamin T. Kile

Since Specialization

Citations

This map shows the geographic impact of Benjamin T. Kile's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin T. Kile with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin T. Kile more than expected).

Fields of papers citing papers by Benjamin T. Kile

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin T. Kile. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin T. Kile. The network helps show where Benjamin T. Kile may publish in the future.

Co-authorship network of co-authors of Benjamin T. Kile

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin T. Kile. A scholar is included among the top collaborators of Benjamin T. Kile based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin T. Kile. Benjamin T. Kile is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Megakaryocytes possess a STING pathway that is transferred to platelets to potentiate activation 2023 ·Life Science Alliance ·(unknown),(unknown),(unknown),(unknown),(unknown),(unknown),Georges Jourdi,(unknown),Michael P. Gantier,Justin R. Hamilton,Benjamin T. Kile,Marie Lordkipanidzé,Geneviève Pépin	6
2	Discordance in STING-Induced Activation and Cell Death Between Mouse and Human Dendritic Cell Populations 2022 ·Frontiers in Immunology ·Ee Shan Pang,(unknown),Katherine R. Balka,Dominic De Nardo,Christophe Macri,Hubertus Hochrein,Kelly‐Anne Masterman,Peck Szee Tan,(unknown),(unknown),(unknown),(unknown),Yifan Zhan,Benjamin T. Kile,Kate E. Lawlor,Kristen J. Radford,Mark D. Wright,Meredith O’Keeffe	17
3	Apoptotic Ablation of Platelets Reduces Atherosclerosis in Mice With Diabetes 2021 ·Arteriosclerosis Thrombosis and Vascular Biology ·Man K.S. Lee,Michael J. Kraakman,Dragana Dragoljevic,Nordin M.J. Hanssen,Michelle Flynn,Annas Al‐Sharea,Gopalkrishna Sreejit,Camilla Bertuzzo Veiga,(unknown),(unknown),(unknown),Mark E. Cooper,Emma C. Josefsson,Benjamin T. Kile,Prabhakara R. Nagareddy,Andrew Murphy	15
4	Acute myeloid leukemia maturation lineage influences residual disease and relapse following differentiation therapy 2021 ·Nature Communications ·(unknown),(unknown),Margherita Ghisi,(unknown),Mark McKenzie,H. L. Mitchell,Peter Kanellakis,Olivia Susanto,Michael J. Hickey,Andrew C. Perkins,Benjamin T. Kile,Ross A. Dickins	8
5	COVID‐19 patients exhibit reduced procoagulant platelet responses 2020 ·Journal of Thrombosis and Haemostasis ·Frederik Denorme,Bhanu Kanth Manne,Irina Portier,Aaron C. Petrey,Elizabeth A. Middleton,Benjamin T. Kile,Matthew T. Rondina,Robert A. Campbell	47
6	TBK1 and IKK epsilon Act Redundantly to Mediate STING-Induced NF-kappa B Responses in Myeloid Cells 2020 ·Cell Reports ·Katherine R. Balka,Cynthia Louis,Tahnee L. Saunders,Amber M. Smith,Dale J. Calleja,Damian B. D’Silva,Fiona Moghaddas,Maximilien Tailler,Kate E. Lawlor,Yifan Zhan,Christopher J. Burns,Ian P. Wicks,Jonathan J. Miner,Benjamin T. Kile,Seth L. Masters,Dominic De Nardo	10
7	Shared roles for Scl and Lyl1 in murine platelet production and function 2019 ·Blood ·(unknown),(unknown),(unknown),(unknown),Sarah Ellis,Benjamin T. Kile,Yizhou Huang,Diego Chacon Fajardo,John E. Pimanda,Dominik Beck,Justin R. Hamilton,Cédric S. Tremblay,David J. Curtis	8
8	Apoptotic Caspases: Multiple or Mistaken Identities? 2018 ·Trends in Cell Biology ·Kate McArthur,Benjamin T. Kile	119
9	A mouse model of hereditary coproporphyria identified in an ENU mutagenesis screen 2017 ·Disease Models & Mechanisms ·(unknown),Fiona C. Brown,Robert O. Fullinfaw,Benjamin T. Kile,Stephen M. Jane,David J. Curtis	6
10	Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β 2015 ·The Journal of Experimental Medicine ·(unknown),Jae Jin Chae,Yong Hwan Park,Dominic De Nardo,Roslynn A. Stirzaker,Hyun‐Ja Ko,Hazel Tye,Louise H. Cengia,Ladina DiRago,Donald Metcalf,Andrew W. Roberts,Daniel L. Kastner,Andrew M. Lew,Dena Lyras,Benjamin T. Kile,Ben A. Croker,Seth L. Masters	107
11	Mice Haploinsufficient for Ets1 and Fli1 Display Middle Ear Abnormalities and Model Aspects of Jacobsen Syndrome 2015 ·American Journal Of Pathology ·Marina R. Carpinelli,Elizabeth Kruse,Benedicta D. Arhatari,Marlyse A. Debrincat,(unknown),Raphaël Borie,Benjamin T. Kile,Rachel Burt	10
12	Aging platelets stimulate TPO production 2015 ·Nature Medicine ·Benjamin T. Kile	6
13	Platelet production proceeds independently of the intrinsic and extrinsic apoptosis pathways 2014 ·Nature Communications ·Emma C. Josefsson,Deborah L. Burnett,Marion Lebois,Marlyse A. Debrincat,M. J. D. White,Katya J. Henley,Rachael M. Lane,Diane Moujalled,Simon Preston,Lorraine A. O’Reilly,Marc Pellegrini,Donald Metcalf,Andreas Strasser,Benjamin T. Kile	63
14	Correction: Two ENU-Induced Alleles of Atp2b2 Cause Deafness in Mice 2013 ·PLoS ONE ·Marina R. Carpinelli,(unknown),Benjamin T. Kile,Rachel Burt	4
15	Two ENU-Induced Alleles of Atp2b2 Cause Deafness in Mice 2013 ·PLoS ONE ·Marina R. Carpinelli,(unknown),Benjamin T. Kile,Rachel Burt	10
16	ENU mutagenesis identifies the first mouse mutants reproducing human β-thalassemia at the genomic level 2012 ·Blood Cells Molecules and Diseases ·Fiona C. Brown,Nicholas A. Scott,Gerhard Rank,Janelle E. Collinge,Jim Vadolas,Nicola Vickaryous,Nadia Whitelaw,Emma Whitelaw,Benjamin T. Kile,Stephen M. Jane,David J. Curtis	15
17	Transgenic, inducible RNAi in megakaryocytes and platelets in mice 2010 ·Journal of Thrombosis and Haemostasis ·(unknown),Chloé James,Emma C. Josefsson,Catherine Carmichael,Prem K. Premsrirut,Scott W. Lowe,Justin R. Hamilton,David C.S. Huang,Benjamin T. Kile,Ross A. Dickins	10
18	A Kinase-Dead Allele of Lyn Attenuates Autoimmune Disease Normally Associated with Lyn Deficiency 2009 ·The Journal of Immunology ·Anne M. Verhagen,Morgan E. Wallace,(unknown),Sarah A. Jones,Hayley A. Croom,Donald Metcalf,Janelle E. Collinge,Mhairi J. Maxwell,Margaret L. Hibbs,Warren S. Alexander,Douglas J. Hilton,Benjamin T. Kile,Robyn Starr	15
19	A Novel Mutation in the Nfkb2 Gene Generates an NF-κB2 “Super Repressor” 2007 ·The Journal of Immunology ·Elena J. Tucker,Kristy O’Donnell,Martina Fuchsberger,Adrienne A. Hilton,Donald Metcalf,Kylie T. Greig,Natalie A. Sims,J Quinn,Warren S. Alexander,Douglas J. Hilton,Benjamin T. Kile,David M. Tarlinton,Robyn Starr	63
20	Programmed Anuclear Cell Death Delimits Platelet Life Spanbreakdown → 2007 ·Cell ·Kylie D. Mason,Marina R. Carpinelli,Jamie I. Fletcher,Janelle E. Collinge,Adrienne A. Hilton,Sarah Ellis,Priscilla N. Kelly,Paul G. Ekert,Donald Metcalf,Andrew W. Roberts,David C.S. Huang,Benjamin T. Kile	767

About Benjamin T. Kile

Benjamin T. Kile is a scholar working on Hematology, Immunology and Genetics, having authored 137 papers that have together received 8.3k indexed citations. Recurring topics across this work include Platelet Disorders and Treatments (41 papers), Cell death mechanisms and regulation (19 papers) and interferon and immune responses (16 papers). The work is most often cited by research in Hematology (1.9k citations), Immunology (2.8k citations) and Molecular Biology (4.6k citations). Benjamin T. Kile has collaborated with scholars based in Australia, United States and United Kingdom. Frequent co-authors include Warren S. Alexander, Donald Metcalf, Douglas J. Hilton, David C.S. Huang, Kate McArthur, M. J. D. White, Nicos A. Nicola, Emma C. Josefsson, Andrew W. Roberts and Kylie D. Mason. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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