Kah‐Leong Lim

29.8k total citations · 1 hit paper
145 papers, 8.9k citations indexed

About

Kah‐Leong Lim is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Kah‐Leong Lim has authored 145 papers receiving a total of 8.9k indexed citations (citations by other indexed papers that have themselves been cited), including 75 papers in Molecular Biology, 75 papers in Neurology and 37 papers in Cellular and Molecular Neuroscience. Recurrent topics in Kah‐Leong Lim's work include Parkinson's Disease Mechanisms and Treatments (73 papers), Autophagy in Disease and Therapy (31 papers) and Nuclear Receptors and Signaling (22 papers). Kah‐Leong Lim is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (73 papers), Autophagy in Disease and Therapy (31 papers) and Nuclear Receptors and Signaling (22 papers). Kah‐Leong Lim collaborates with scholars based in Singapore, United States and China. Kah‐Leong Lim's co-authors include Ted M. Dawson, Valina L. Dawson, Chengwu Zhang, Jeanne M.M. Tan, Kenny K. K. Chung, Tso‐Pang Yao, Esther Wong, Chou Chai, Lin Li and Catherine J. Pallen and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Angewandte Chemie International Edition.

In The Last Decade

Kah‐Leong Lim

142 papers receiving 8.8k citations

Hit Papers

1 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Parkin ubiquitinates the α-synuclein–interacting protein, synphilin-1: implications for Lewy-body formation in Parkinson disease

2001 599 citations Kenny K. K. Chung, Kah‐Leong Lim et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kah‐Leong Lim Singapore	54	4.5k	3.5k	2.1k	2.0k	1.5k	145	8.9k
Lian Li United States	50	5.6k 1.2×	1.7k 0.5×	1.9k 0.9×	1.1k 0.6×	1.4k 0.9×	199	9.2k
Lisa Ellerby United States	51	9.3k 2.0×	1.6k 0.5×	4.4k 2.1×	1.3k 0.7×	1.4k 1.0×	113	13.5k
Daniel Offen Israel	59	5.4k 1.2×	2.5k 0.7×	3.1k 1.5×	585 0.3×	1.4k 0.9×	211	11.5k
Han Seok Ko United States	33	4.0k 0.9×	4.2k 1.2×	2.2k 1.0×	2.1k 1.1×	2.0k 1.4×	58	8.5k
Veerle Baekelandt Belgium	58	5.3k 1.2×	4.7k 1.4×	3.4k 1.6×	735 0.4×	2.7k 1.9×	218	11.7k
Dimitri Krainc United States	57	8.3k 1.8×	5.0k 1.4×	4.8k 2.3×	2.2k 1.1×	4.5k 3.1×	135	15.5k
Ruth S. Slack Canada	67	8.4k 1.9×	1.9k 0.6×	2.5k 1.2×	1.4k 0.7×	1.7k 1.1×	145	12.6k
Jean‐Christophe Rochet United States	37	4.0k 0.9×	5.4k 1.5×	2.5k 1.2×	743 0.4×	4.0k 2.7×	108	10.3k
Andrew B. West United States	55	5.0k 1.1×	6.9k 2.0×	2.9k 1.4×	914 0.5×	2.6k 1.7×	129	11.9k
Jochen Herms Germany	62	6.0k 1.3×	1.5k 0.4×	2.7k 1.3×	653 0.3×	4.5k 3.1×	243	14.0k

Countries citing papers authored by Kah‐Leong Lim

Since Specialization

Citations

This map shows the geographic impact of Kah‐Leong Lim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kah‐Leong Lim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kah‐Leong Lim more than expected).

Fields of papers citing papers by Kah‐Leong Lim

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kah‐Leong Lim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kah‐Leong Lim. The network helps show where Kah‐Leong Lim may publish in the future.

Co-authorship network of co-authors of Kah‐Leong Lim

This figure shows the co-authorship network connecting the top 25 collaborators of Kah‐Leong Lim. A scholar is included among the top collaborators of Kah‐Leong Lim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kah‐Leong Lim. Kah‐Leong Lim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wang, Ziyin, Yanmei Liu, Richard M. Y. Tang, et al.. (2025). Ergothioneine Treatment Ameliorates the Pathological Phenotypes of Parkinson's Disease Models. Journal of Neurochemistry. 169(7). e70168–e70168.

Jeon, Ji-Hyeon, et al.. (2025). Restoration of intestinal barrier function by fexuprazan, a potassium-competitive acid blocker, in Caco-2 cells and its higher gastrointestinal distribution in rats. Biomedicine & Pharmacotherapy. 188. 118185–118185. 1 indexed citations

Guo, Lixia, Y. Thomas Hou, Zheng Li, et al.. (2025). In situ dual-activated NIRF/PA carrier-free nanoprobe for diagnosis and treatment of Parkinson's disease. Biosensors and Bioelectronics. 282. 117473–117473.

Guo, Lixia, Bin Wang, Sufang Ma, et al.. (2024). Double-locked probe for NIRF/PA imaging mitochondrial H2O2 and viscosity in Parkinson's disease. Sensors and Actuators B Chemical. 426. 137104–137104. 2 indexed citations

Ma, Sufang, et al.. (2024). Role of lipid droplets in neurodegenerative diseases: From pathogenesis to therapeutics. Neuroscience & Biobehavioral Reviews. 165. 105867–105867. 9 indexed citations

Cui, Wenli, Hong Chen, Wenru Wang, et al.. (2024). Seipin Deficiency Leads to Energy Dyshomeostasis via Inducing Hypothalamic Neuroinflammation and Aberrant Expression of Neuropeptides. NeuroMolecular Medicine. 26(1). 18–18.

Wu, Yue, Naidi Yang, Chenqi Xin, et al.. (2023). Vitamin B12 Ameliorates the Pathological Phenotypes of Multiple Parkinson’s Disease Models by Alleviating Oxidative Stress. Antioxidants. 12(1). 153–153. 10 indexed citations

Lim, Kah‐Leong, et al.. (2023). From 2D to 3D: Development of Monolayer Dopaminergic Neuronal and Midbrain Organoid Cultures for Parkinson’s Disease Modeling and Regenerative Therapy. International Journal of Molecular Sciences. 24(3). 2523–2523. 13 indexed citations

Zhou, Zhiqiang, Jingyan Ge, Naidi Yang, et al.. (2023). Design and Synthesis of a Mitochondrial‐Targeted JNK Inhibitor and Its Protective Effect on Parkinson's Disease Phenotypes. ChemBioChem. 24(13). e202200748–e202200748. 3 indexed citations

10.

Lin, Chao‐Chieh, Jin Yan, Meghan Kapur, et al.. (2022). Parkin coordinates mitochondrial lipid remodeling to execute mitophagy. EMBO Reports. 23(12). e55191–e55191. 14 indexed citations

11.

Jo, Junghyun, Weonjin Yu, Alfred Xuyang Sun, et al.. (2021). Lewy Body–like Inclusions in Human Midbrain Organoids Carrying Glucocerebrosidase and α‐Synuclein Mutations. Annals of Neurology. 90(3). 490–505. 58 indexed citations

12.

Ng, Felicia, Chou Chai, Jeanne M.M. Tan, et al.. (2012). Parkin Pathway Activation Mitigates Glioma Cell Proliferation and Predicts Patient Survival. Cancer Research. 72(10). 2543–2553. 74 indexed citations

13.

Chew, Katherine C. M., Eng‐Tat Ang, Yee Kit Tai, et al.. (2011). Enhanced Autophagy from Chronic Toxicity of Iron and Mutant A53T α-Synuclein. Journal of Biological Chemistry. 286(38). 33380–33389. 73 indexed citations

14.

Ng, Chee H., Xuezhi Ouyang, Marc Fivaz, et al.. (2009). Parkin Protects against LRRK2 G2019S Mutant-Induced Dopaminergic Neurodegeneration in Drosophila. Journal of Neuroscience. 29(36). 11257–11262. 166 indexed citations

15.

Wang, Cheng, Ruifeng Lu, Xuezhi Ouyang, et al.. (2007). DrosophilaOverexpressing Parkin R275W Mutant Exhibits Dopaminergic Neuron Degeneration and Mitochondrial Abnormalities. Journal of Neuroscience. 27(32). 8563–8570. 97 indexed citations

16.

Lim, Kah‐Leong. (2007). Ubiquitin–proteasome system dysfunction in Parkinson’s disease: current evidence and controversies. Expert Review of Proteomics. 4(6). 769–781. 63 indexed citations

17.

Coelln, Rainer von, Bobby Thomas, Shaida A. Andrabi, et al.. (2006). Inclusion Body Formation and Neurodegeneration Are Parkin Independent in a Mouse Model of α-Synucleinopathy. Journal of Neuroscience. 26(14). 3685–3696. 63 indexed citations

18.

Wang, Cheng, Jeanne M.M. Tan, Norazean Zaiden, et al.. (2005). Alterations in the solubility and intracellular localization of parkin by several familial Parkinson's disease‐linked point mutations. Journal of Neurochemistry. 93(2). 422–431. 98 indexed citations

19.

Wang, Cheng, Han Seok Ko, Bobby Thomas, et al.. (2005). Stress-induced alterations in parkin solubility promote parkin aggregation and compromise parkin's protective function. Human Molecular Genetics. 14(24). 3885–3897. 169 indexed citations

20.

Lim, Kah‐Leong, Katherine C. M. Chew, Jeanne M.M. Tan, et al.. (2005). Parkin Mediates Nonclassical, Proteasomal-Independent Ubiquitination of Synphilin-1: Implications for Lewy Body Formation. Journal of Neuroscience. 25(8). 2002–2009. 454 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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