John W. Kappler

51.2k citations
383 papers · 44.3k · 16 hit papers · h-index 111

Impact in

  • Immunology top 0.01%
    • T-cell and B-cell Immunology
    • Immune Cell Function and Interaction
    • Immunotherapy and Immune Responses
    • Immune Response and Inflammation
    • Monoclonal and Polyclonal Antibodies Research

Papers in

    • T-cell and B-cell Immunology 242
    • Immune Cell Function and Interaction 224
    • Immunotherapy and Immune Responses 118
    • Immune Response and Inflammation 43
    • Monoclonal and Polyclonal Antibodies Research 84

John W. Kappler

380 papers receiving 42.3k citations

John W. Kappler's Hit Papers

Toll-like receptor 7 (TLR7)–driven accumulation of a novel CD11c+ B-cell population is important for the development of autoimmunity 2011 · 585 citations
5850+14+28Years since publication50010001.5k

Peers

John W. Kappler
Comparison fields: 5 of 173
  • Immunology 32.9k
  • Radiology, Nuclear Medicine and Imaging 7.1k
  • Immunology and Allergy 1.5k
  • Oncology 5.5k
  • Virology 921
Replace Philippa Marrack with:
Philippa Marrack United States
Emil R. Unanue United States
William E. Paul United States
Thomas A. Waldmann United States
Max D. Cooper United States
Hans Wigzell Sweden
Richard S. Blumberg United States
Ellis L. Reinherz United States
Hidde L. Ploegh United States
Hergen Spits Netherlands
John W. Kappler relative to Philippa Marrack United States Philippa Marrack's profile →
Citations per field
00.5×1.5×
Philippa Marrack · 1×
Citations per year

Countries citing papers authored by John W. Kappler

Since Specialization
Citations

This map shows the geographic impact of John W. Kappler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Kappler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Kappler more than expected).

Fields of papers citing papers by John W. Kappler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Kappler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Kappler. The network helps show where John W. Kappler may publish in the future.

Co-authors

The 25 scholars most cited alongside John W. Kappler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John W. Kappler Line = papers co-authored together John W. Kappler links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 383 papers — load more, or switch the sort, to bring in the rest.

#Work
1
T cell tolerance by clonal elimination in the thymus
Hit paper breakdown →
19871910
2
The Vβ-specific superantigen staphylococcal enterotoxin B: Stimulation of mature T cells and clonal deletion in neonatal mice
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1989980
3
Characterization of the Murine Antigenic Determinant, Designated L3T4a, Recognized by Monoclonal Antibody GK 1.5: Expression of L3T4a by Functional T Cell Clones Appears to Correlate Primarily with Class II MHC Antigen‐Reactivity
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1983976
4
Self-tolerance eliminates T cells specific for Mls-modified products of the major histocompatibility complex
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1988933
5
Normal Development of Mice Deficient in β 2 M, MCClass I Proteins, and CD8 + T Cells
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1990808
6
The major histocompatibility complex-restricted antigen receptor on T cells. I. Isolation with a monoclonal antibody.
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1983777
7
Control of Homeostasis of CD8 + Memory T Cells by Opposing Cytokines
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2000724
8
Characterization of a monoclonal antibody which detects all murine alpha beta T cell receptors.
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1989723
9
Type I Interferons Keep Activated T Cells Alive
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1999643
10
Antigen recognition by H-2-restricted T cells. I. Cell-free antigen processing.
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1983624
11
Toll-like receptor 7 (TLR7)–driven accumulation of a novel CD11c+ B-cell population is important for the development of autoimmunity
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2011585
12
Evidence for the Effects of a Superantigen in Rheumatoid Arthritis
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1991580
13
An animal model of hemophagocytic lymphohistiocytosis (HLH): CD8+ T cells and interferon gamma are essential for the disorder
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2004577
14
Superantigens and Their Potential Role in Human Disease
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1993542
15
The Role of the T Cell Receptor in Positive and Negative Selection of Developing T Cells
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1990512
16 1987487
17 2001474
18 2002473
19 1988451
20 1999424

About John W. Kappler

John W. Kappler is a scholar working on Immunology, Radiology, Nuclear Medicine and Imaging, Oncology, Molecular Biology and Genetics, having authored 383 papers that have together received 44.3k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (242 papers), Immune Cell Function and Interaction (224 papers), Immunotherapy and Immune Responses (118 papers), Monoclonal and Polyclonal Antibodies Research (84 papers), Immune Response and Inflammation (43 papers), CAR-T cell therapy research (42 papers), Diabetes and associated disorders (23 papers) and Glycosylation and Glycoproteins Research (12 papers). The work is most often cited by research in Immunology (32.9k citations), Radiology, Nuclear Medicine and Imaging (7.1k citations), Immunology and Allergy (1.5k citations), Oncology (5.5k citations) and Virology (921 citations). John W. Kappler has collaborated with scholars based in United States, China and Germany. Frequent co-authors include Philippa Marrack, Neal W. Roehm, Ralph T. Kubo, David A. Hildeman, Janice White, Frances Crawford, Brian L. Kotzin, Tom Mitchell, Kathryn Haskins and Ann M. Pullen. Their work appears in journals such as The Journal of Immunology, Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine, Cell and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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