William E. Paul

54.3k citations
350 papers · 43.4k · 21 hit papers · h-index 100

Impact in

  • Immunology top 0.01%
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Immunotherapy and Immune Responses
    • IL-33, ST2, and ILC Pathways
    • Immune Response and Inflammation

Papers in

    • Immune Cell Function and Interaction 160
    • T-cell and B-cell Immunology 158
    • Immunotherapy and Immune Responses 68
    • Immune Response and Inflammation 38
    • IL-33, ST2, and ILC Pathways 28
    • Immunodeficiency and Autoimmune Disorders 18

William E. Paul

346 papers receiving 41.5k citations

William E. Paul's Hit Papers

S1P-dependent interorgan trafficking of group 2 innate lymphoid cells supports host defense 2018 · 427 citations
4270+11+22Years since publication50010001.5k2.0k2.5k

Peers

William E. Paul
Comparison fields: 5 of 174
  • Immunology 29.6k
  • Immunology and Allergy 2.3k
  • Virology 1.2k
  • Oncology 5.9k
  • Dermatology 1.5k
Replace Ethan M. Shevach with:
Ethan M. Shevach United States
Joost J. Oppenheim United States
Anne O’Garra United States
Robert L. Coffman United States
Warren Strober United States
Hergen Spits Netherlands
Sergio Romagnani Italy
Giorgio Trinchieri United States
Philippa Marrack United States
Charles A. Janeway United States
William E. Paul relative to Ethan M. Shevach United States Ethan M. Shevach's profile →
Citations per field
00.5×1.5×2.1×
Ethan M. Shevach · 1×
Citations per year

Countries citing papers authored by William E. Paul

Since Specialization
Citations

This map shows the geographic impact of William E. Paul's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William E. Paul with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William E. Paul more than expected).

Fields of papers citing papers by William E. Paul

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William E. Paul. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William E. Paul. The network helps show where William E. Paul may publish in the future.

Co-authors

The 25 scholars most cited alongside William E. Paul, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with William E. Paul Line = papers co-authored together William E. Paul links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 350 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Differentiation of Effector CD4 T Cell Populations
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20102592
2
Interferon-γ and B Cell Stimulatory Factor-1 Reciprocally Regulate Ig Isotype Production
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19871678
3
Lymphocyte responses and cytokines
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19941548
4
THE IL-4 RECEPTOR: Signaling Mechanisms and Biologic Functions
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19991299
5
CD4 T cells: fates, functions, and faults
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20081282
6
Mast cell lines produce lymphokines in response to cross-linkage of FcεRI or to calcium ionophores
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19891009
7
Mechanisms Underlying Lineage Commitment and Plasticity of Helper CD4 + T Cells
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2010991
8
Global Mapping of H3K4me3 and H3K27me3 Reveals Specificity and Plasticity in Lineage Fate Determination of Differentiating CD4+ T Cells
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2009909
9
Defective lymphoid development in mice lacking expression of the common cytokine receptor γ chain
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1995855
10
Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome
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2008850
11
How are TH2-type immune responses initiated and amplified?
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2010727
12
Production of a monoclonal antibody to and molecular characterization of B-cell stimulatory factor-1
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1985676
13
T-bet is rapidly induced by interferon-γ in lymphoid and myeloid cells
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2001625
14
NKT cell–mediated repression of tumor immunosurveillance by IL-13 and the IL-4R–STAT6 pathway
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2000560
15
Colocalization of X-Linked Agammaglobulinemia and X-Linked Immunodeficiency Genes
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1993545
16
Interleukin-2 Receptor γ Chain: a Functional Component of the Interleukin-4 Receptor
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1993525
17
IL-25-responsive, lineage-negative KLRG1hi cells are multipotential ‘inflammatory’ type 2 innate lymphoid cells
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2014515
18
Conditional deletion of Gata3 shows its essential function in TH1-TH2 responses
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2004500
19 2009464
20 1995448

About William E. Paul

William E. Paul is a scholar working on Immunology, Molecular Biology, Radiology, Nuclear Medicine and Imaging, Oncology and Genetics, having authored 350 papers that have together received 43.4k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (160 papers), T-cell and B-cell Immunology (158 papers), Immunotherapy and Immune Responses (68 papers), Monoclonal and Polyclonal Antibodies Research (53 papers), Immune Response and Inflammation (38 papers), IL-33, ST2, and ILC Pathways (28 papers), Cytokine Signaling Pathways and Interactions (23 papers) and Immunodeficiency and Autoimmune Disorders (18 papers). The work is most often cited by research in Immunology (29.6k citations), Immunology and Allergy (2.3k citations), Virology (1.2k citations), Oncology (5.9k citations) and Dermatology (1.5k citations). William E. Paul has collaborated with scholars based in United States, Israel and Germany. Frequent co-authors include Jinfang Zhu, Hidehiro Yamane, Clifford M. Snapper, Jane Hu‐Li, Robert A. Seder, John J. O’Shea, Baruj Benacerraf, Liying Guo, John D. Stobo and J Ohara. Their work appears in journals such as The Journal of Immunology, The Journal of Experimental Medicine, Proceedings of the National Academy of Sciences, Immunity and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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