João B. Relvas
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Developmental Neuroscience top 0.2%
- Neurology top 1%
- Cell Biology top 2%
- Co-authors
- Charles ffrench‐ConstantUeli SuterRenato SocodatoCamila C. PortugalHelena S. DominguesCord BrakebuschDino P. LeoneReinhard Fässler
- Topics
- Neurogenesis and neuroplasticity mechanisms (31 papers)Neuroinflammation and Neurodegeneration Mechanisms (30 papers)Nerve injury and regeneration (20 papers)
- Partner nations
- PortugalUnited KingdomSwitzerland
In The Last Decade
João B. Relvas
100 papers receiving 4.4k citations
Hit Papers
Peers
Comparison fields: 5 of 135
- Molecular Biology 2.0k
- Cellular and Molecular Neuroscience 1.5k
- Developmental Neuroscience 1.3k
- Neurology 785
- Cell Biology 721
Countries citing papers authored by João B. Relvas
This map shows the geographic impact of João B. Relvas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by João B. Relvas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites João B. Relvas more than expected).
Fields of papers citing papers by João B. Relvas
This network shows the impact of papers produced by João B. Relvas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by João B. Relvas. The network helps show where João B. Relvas may publish in the future.
Co-authorship network of co-authors of João B. Relvas
This figure shows the co-authorship network connecting the top 25 collaborators of João B. Relvas. A scholar is included among the top collaborators of João B. Relvas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with João B. Relvas. João B. Relvas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 10 | |
| 3 | 0 | |
| 4 | 5 | |
| 5 | 0 | |
| 6 | 1 | |
| 7 | 5 | |
| 8 | 7 | |
| 9 | 4 | |
| 10 | 13 | |
| 11 | 4 | |
| 12 | 4 | |
| 13 | 8 | |
| 14 | 8 | |
| 15 | 6 | |
| 16 | 27 | |
| 17 | 26 | |
| 18 | 28 | |
| 19 | 58 | |
| 20 | 141 |
About João B. Relvas
João B. Relvas is a scholar working on Developmental Neuroscience, Neurology and Immunology and Allergy, having authored 104 papers that have together received 4.4k indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (31 papers), Neuroinflammation and Neurodegeneration Mechanisms (30 papers) and Nerve injury and regeneration (20 papers). The work is most often cited by research in Developmental Neuroscience (1.3k citations), Neurology (785 citations) and Cellular and Molecular Neuroscience (1.5k citations). João B. Relvas has collaborated with scholars based in Portugal, United Kingdom and Switzerland. Frequent co-authors include Charles ffrench‐Constant, Ueli Suter, Renato Socodato, Camila C. Portugal, Helena S. Domingues, Cord Brakebusch, Dino P. Leone, Reinhard Fässler, Jorge A. Pereira and Lia S. Campos. Their work appears in journals such as Science, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.