Jami Willette‐Brown

1.7k total citations
34 papers, 1.3k citations indexed

About

Jami Willette‐Brown is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, Jami Willette‐Brown has authored 34 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Immunology, 13 papers in Molecular Biology and 8 papers in Cancer Research. Recurrent topics in Jami Willette‐Brown's work include Immune Cell Function and Interaction (15 papers), T-cell and B-cell Immunology (15 papers) and NF-κB Signaling Pathways (7 papers). Jami Willette‐Brown is often cited by papers focused on Immune Cell Function and Interaction (15 papers), T-cell and B-cell Immunology (15 papers) and NF-κB Signaling Pathways (7 papers). Jami Willette‐Brown collaborates with scholars based in United States, China and France. Jami Willette‐Brown's co-authors include John R. Ortaldo, Llewellyn H. Mason, Daniel W. McVicar, Stephen K. Anderson, Pierre Gosselin, Yinling Hu, Arthur A. Hurwitz, Scott K. Durum, Lynn S. Taylor and Salvatore Sechi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Jami Willette‐Brown

34 papers receiving 1.3k citations

Peers

Jami Willette‐Brown
Lihe Su United States
John F. MacMaster United States
Xincheng Zheng United States
Janet Jackman United States
Abhishek Aphale United States
Danielle L. Gilvary United States
Donna Butcher United States
Lihe Su United States
Jami Willette‐Brown
Citations per year, relative to Jami Willette‐Brown Jami Willette‐Brown (= 1×) peers Lihe Su

Countries citing papers authored by Jami Willette‐Brown

Since Specialization
Citations

This map shows the geographic impact of Jami Willette‐Brown's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jami Willette‐Brown with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jami Willette‐Brown more than expected).

Fields of papers citing papers by Jami Willette‐Brown

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jami Willette‐Brown. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jami Willette‐Brown. The network helps show where Jami Willette‐Brown may publish in the future.

Co-authorship network of co-authors of Jami Willette‐Brown

This figure shows the co-authorship network connecting the top 25 collaborators of Jami Willette‐Brown. A scholar is included among the top collaborators of Jami Willette‐Brown based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jami Willette‐Brown. Jami Willette‐Brown is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Song, Na‐Young, Xin Li, Buyong Ma, et al.. (2022). IKKα-deficient lung adenocarcinomas generate an immunosuppressive microenvironment by overproducing Treg-inducing cytokines. Proceedings of the National Academy of Sciences. 119(6). 13 indexed citations
2.
Xiao, Zuoxiang, Sichuan Xi, Amit Kumar Singh, et al.. (2022). A TNFR1–UBCH10 axis drives lung squamous cell carcinoma dedifferentiation and metastasis through a cell-autonomous signaling loop. Cell Death and Disease. 13(10). 885–885. 5 indexed citations
3.
Zhu, Feng, Jami Willette‐Brown, Jian Zhang, et al.. (2020). NLRP3 Inhibition Ameliorates Severe Cutaneous Autoimmune Manifestations in a Mouse Model of Autoimmune Polyendocrinopathy-Candidiasis-Ectodermal Dystrophy–Like Disease. Journal of Investigative Dermatology. 141(6). 1404–1415. 8 indexed citations
4.
Sun, Qiao, Dandan Sun, Jami Willette‐Brown, et al.. (2020). C-CBL is required for inhibition of angiogenesis through modulating JAK2/STAT3 activity in ROP development. Biomedicine & Pharmacotherapy. 132. 110856–110856. 9 indexed citations
5.
Wang, Xin, Jami Willette‐Brown, Feng Zhu, et al.. (2018). Macrophage inducible nitric oxide synthase circulates inflammation and promotes lung carcinogenesis. Cell Death Discovery. 4(1). 46–46. 39 indexed citations
6.
Marien, Eyra, Michael Meister, Thomas Muley, et al.. (2016). Phospholipid profiling identifies acyl chain elongation as a ubiquitous trait and potential target for the treatment of lung squamous cell carcinoma. Oncotarget. 7(11). 12582–12597. 54 indexed citations
7.
Martin, Bradley N., Chenhui Wang, Jami Willette‐Brown, et al.. (2014). IKKα negatively regulates ASC-dependent inflammasome activation. Nature Communications. 5(1). 4977–4977. 88 indexed citations
8.
Xia, Xiaojun, Shuang Liu, Zuoxiang Xiao, et al.. (2013). An IKKα-Nucleophosmin Axis Utilizes Inflammatory Signaling to Promote Genome Integrity. Cell Reports. 5(5). 1243–1255. 19 indexed citations
9.
Willette‐Brown, Jami, et al.. (2011). IKKα represses a network of inflammation and proliferation pathways and elevates c-Myc antagonists and differentiation in a dose-dependent manner in the skin. Cell Death and Differentiation. 18(12). 1854–1864. 26 indexed citations
10.
Xia, Xiaojun, Eunmi Park, Bigang Liu, et al.. (2010). Reduction of IKKα Expression Promotes Chronic Ultraviolet B Exposure-Induced Skin Inflammation and Carcinogenesis. American Journal Of Pathology. 176(5). 2500–2508. 24 indexed citations
11.
Mazzucchelli, Renata, Julie A. Hixon, Rosanne Spolski, et al.. (2008). Development of regulatory T cells requires IL-7Rα stimulation by IL-7 or TSLP. Blood. 112(8). 3283–3292. 102 indexed citations
12.
Ortaldo, John R., Anna Mason, Jami Willette‐Brown, et al.. (2007). Modulation of lymphocyte function with inhibitory CD2: Loss of NK and NKT cells. Cellular Immunology. 249(1). 8–19. 2 indexed citations
13.
Mason, Llewellyn H., Jami Willette‐Brown, Lynn S. Taylor, & Daniel W. McVicar. (2006). Regulation of Ly49D/DAP12 Signal Transduction by Src-Family Kinases and CD45. The Journal of Immunology. 176(11). 6615–6623. 44 indexed citations
14.
Ortaldo, John R., Robin Winkler-Pickett, Jami Willette‐Brown, et al.. (1999). Structure/Function Relationship of Activating Ly-49D and Inhibitory Ly-49G2 NK Receptors. The Journal of Immunology. 163(10). 5269–5277. 35 indexed citations
15.
Mason, Llewellyn H., Jami Willette‐Brown, Stephen K. Anderson, et al.. (1998). Cutting Edge: Characterization of an Associated 16-kDa Tyrosine Phosphoprotein Required for Ly-49D Signal Transduction. The Journal of Immunology. 160(9). 4148–4152. 69 indexed citations
16.
McVicar, Daniel W., Lynn S. Taylor, Pierre Gosselin, et al.. (1998). DAP12-mediated Signal Transduction in Natural Killer Cells. Journal of Biological Chemistry. 273(49). 32934–32942. 183 indexed citations
17.
Sechi, Salvatore, Peter P. Roller, Jami Willette‐Brown, & Jean Pierre Kinet. (1996). A Conformational Rearrangement upon Binding of IgE to Its High Affinity Receptor. Journal of Biological Chemistry. 271(32). 19256–19263. 31 indexed citations
18.
Letourneur, Odile, Salvatore Sechi, Jami Willette‐Brown, Michael W. Robertson, & Jean‐Pierre Kinet. (1995). Glycosylation of Human Truncated Fc∊RI α Chain Is Necessary for Efficient Folding in the Endoplasmic Reticulum. Journal of Biological Chemistry. 270(14). 8249–8256. 88 indexed citations
19.
Ferris, Douglas K., Jami Willette‐Brown, Todd M. Martensen, & William L. Farrar. (1989). Interleukin 3 and phorbol ester stimulate tyrosine phosphorylation of overlapping substrate proteins. FEBS Letters. 246(1-2). 153–158. 10 indexed citations
20.
Harel‐Bellan, Annick, Zohair Mishal, Jami Willette‐Brown, & William L. Farrar. (1989). Detection of low and high affinity binding sites with fluoresceinated human recombinant interleukin-2. Journal of Immunological Methods. 119(1). 127–133. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026