James D. Vasta
- Molecular Biology
- Oncology top 10%
- Hematology top 10%
- Organic Chemistry
- Pulmonary and Respiratory Medicine
- Co-authors
- Matthew B. RobersRonald T. RainesCesear CoronaMarjeta UrhDanette L. DanielsKristin M. RichingSarah D. MahanMark G. McDougall
- Topics
- Protein Degradation and Inhibitors (12 papers)Cancer, Hypoxia, and Metabolism (5 papers)Computational Drug Discovery Methods (5 papers)
- Cited by
- HematologyMolecular BiologyOncology
- Journals
- Journal of the American Chemical SocietyNature CommunicationsSHILAP Revista de lepidopterología
- Partner nations
- United StatesGermanyUnited Kingdom
In The Last Decade
James D. Vasta
38 papers receiving 838 citations
Peers
Comparison fields: 5 of 82
- Molecular Biology 662
- Oncology 262
- Hematology 109
- Organic Chemistry 89
- Pulmonary and Respiratory Medicine 88
Countries citing papers authored by James D. Vasta
This map shows the geographic impact of James D. Vasta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James D. Vasta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James D. Vasta more than expected).
Fields of papers citing papers by James D. Vasta
This network shows the impact of papers produced by James D. Vasta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James D. Vasta. The network helps show where James D. Vasta may publish in the future.
Co-authorship network of co-authors of James D. Vasta
This figure shows the co-authorship network connecting the top 25 collaborators of James D. Vasta. A scholar is included among the top collaborators of James D. Vasta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James D. Vasta. James D. Vasta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 13 | |
| 3 | 6 | |
| 4 | 0 | |
| 5 | 6 | |
| 6 | 6 | |
| 7 | 10 | |
| 8 | 14 | |
| 9 | 14 | |
| 10 | 26 | |
| 11 | 78 | |
| 12 | 29 | |
| 13 | 39 | |
| 14 | 1 | |
| 15 | 9 | |
| 16 | 33 | |
| 17 | 7 | |
| 18 | 1 | |
| 19 | 12 | |
| 20 | 12 |
About James D. Vasta
James D. Vasta is a scholar working on Filtration and Separation, Molecular Biology and Cancer Research, having authored 42 papers that have together received 865 indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (12 papers), Cancer, Hypoxia, and Metabolism (5 papers) and Computational Drug Discovery Methods (5 papers). The work is most often cited by research in Hematology (109 citations), Molecular Biology (662 citations) and Oncology (262 citations). James D. Vasta has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Matthew B. Robers, Ronald T. Raines, Cesear Corona, Marjeta Urh, Danette L. Daniels, Kristin M. Riching, Sarah D. Mahan, Mark G. McDougall, Bernard Fried and Stefan Knapp. Their work appears in journals such as Journal of the American Chemical Society, Nature Communications and SHILAP Revista de lepidopterología.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.